Rhodococcus Genus Study Notes
Genus Rhodococcus
Belongs to the order Actinomycetales
Contains 9 soil-associated species that produce a red pigment
Only Rhodococcus equi has been reported to cause lesions in animals
Associated conditions include:
Purulent pneumonia
Mesenteric lymphadenitis
Arthritis in foals
Lesions similar to tuberculosis in cervical lymph nodes of swine and cattle
Rhodococcus equi
Causative agent of purulent pneumonia in foals
Morphology and Staining
Large organism with pleomorphic characteristics
Forms bacillary and coccoid forms
Coccoid on solid media
Bacillary in fluid, sometimes appears in short chains
Metachromatic granules can usually be demonstrated if grown in milk
Gram-positive and variably acid-fast
Stains readily with other dyes
Non-sporeforming
Possesses a lamellar polysaccharide capsule
Cultural and Biochemical Features
Grows well on all ordinary media
Colonies characteristics:
Diameter: 1 cm
Appearance: raised, moist, translucent, regular outline
Initial color: white, changes to rose pink especially when grown on potato
Grows poorly in milk
Biochemical characteristics:
Catalase positive
Urease positive
Cytochrome c negative
Does not ferment carbohydrates (CHO)
Reduces nitrate
Does not form indole
Non-hemolytic
Produces phospholipase and cholesterol oxidase:
Reacts with phospholipase D of C. pseudotuberculosis, beta toxin of S. aureus, and hemolysin of L. monocytogenes to completely hemolyze sheep, cattle, and rabbit RBC.
Implication: Rapid presumptive test for identification of R. equi
Antigens
Polysaccharide capsule present
Divided into 4 groups and further into 14 serotypes
Epizootiology and Pathogenesis
Acquired from soil, gut contents, and feces of herbivores
Heat resistant
Killed at 60°C for 1 hour
Resists extreme pH, 0.01% sodium azide, 0.5% formaldehyde, sunlight, and desiccation
Route of infection in foals:
Enters through the respiratory tract
Pathway includes entry into alveoli, phagocytosis by macrophages, and subsequent processes leading to degeneration and necrosis:
Massive infiltration of macrophages and multinucleated giant cells into the alveolar space
Formation of foci of alveolar necrosis where bacteria-laden macrophages degenerate
Granulomatous response in the lung
Massive invasion of polymorphonuclear leukocytes (PMNs) leading to suppurative bronchopneumonia with prominent abscesses
Clinical Signs
Common in foals aged 2 to 5 months and older
Clinical manifestations include:
Anorexia
Nasal discharge
Arthritis
Diarrhea
Mortality rate: 64%
Lymphadenitis with abscessed lymph nodes
Contrast with strangles where lymph nodes of the head are seldom involved
In addition to pneumonia, internal abscesses and pleurisy can occur
In swine, infections affect submandibular and cervical lymph nodes, sometimes associated with tubercle bacilli
Immunity
Involves cell-mediated immune responses
Surface components inhibit bactericidal mechanisms of PMNs
Antibody production measurable by:
Precipitin reaction
Complement fixation
Agglutination
Indirect hemagglutination
ELISA
Antibodies present in the serum of normal mares and passively transferred to foals
Generally poor antibody responses noted
Diagnosis
Recognized based on the source isolate
Gram staining reaction
Pleomorphism
Appearance of pinkish, mucoid, spreading colonies
Catalase and urease positivity
Lack of fermentative activity on carbohydrates (CHO)
Equi factor test (Prescott confirmatory)
Transtracheal aspirates and Gram staining cultures are of great value
Selective medium containing:
Nalidixic acid
Novobiocin
Actidione (cycloheximide)
Potassium tellurite (NANAT medium)
Resulting colonies turn black
Antimicrobial Susceptibility
Sensitive to:
Penicillin G
Doxycycline
Erythromycin
Lincomycin
Gentamycin
Neomycin
Streptomycin
Effective combinations include:
Erythromycin and rifampicin
Penicillin and gentamycin
Diseases in Humans
Particularly affects individuals with:
AIDS or undergoing immunosuppressive therapy for malignant neoplasms
Conclusion
Importance of awareness of Rhodococcus equi infection in veterinary and human health.