brain recovery / neuroplasticity
traumatic brain injury
TBI is caused by blow to the head where frontal and temporal lobes are usually injured. It causes impairments to memory, cognitive ability, social communication and emotional regulation and is consistently linked with earlier, more frequent and violent offending.
Phineas gage:
Suffered frontal lobe brain damage when a rod went through his brain while working on a railroad
He lost mostly white brain matter (11%) compared to grey matter (4%), which has the ability to regenerate and explains his quick (6 month) recovery
He became highly aggressive, agitated and impatient — disinhibited
Harlow: brain has localised function as damaged part controlled reasoning and restraint which was compromised
However, this case could be used to support holism, as he was able to function normally, despite losing brain matter
Problems with the study: small sample, misinfo and less knowledge at the time — makes this story hard to draw conclusions and generalisations for
plasticity
Plasticity is the brain’s ability to change and adapt, usually through regeneration or reorganisation, as a result of experiences, learning or damage.
After brain damage, some recovery is usually made through either:
New branches of axons and dendrites growing between neurons to areas of damage
The brain finding another way to complete a function that was originally associated with the damaged area
Ways axons can be replaced:
Increased brain stimulation
Neighbour neurons that connect with damaged neurons no longer have inputs, stimulating them can improve recovery
E.g electroconvulsive therapy
Axon sprouting
Damaged axon lose connections, they can re-sprout on neuron or neighbouring neuron in replacement
Denervation super sensitivity
Axons that do similar job can become aroused at a high level to compensate lost ones
Replaces lost function but can cause oversensitivity to messages like pain levels
Neuroplasticity studies:
Gopnik et al — 2-3 year old: 15,000 synaptic connections, 2x as many as adults
Synaptic pruning is the deletion of unused pathways and strengthening of frequent connections
Critical period of plasticity
Maguire et al — taxi drivers have more grey matter in their hippocampus than match control group
Linked to spatial awareness/navigation
Longer in the job, more pronounced difference
Draganski et al — medical students 3 months before and after exam, changes found in posterior hippocampus and parietal cortex
Mechelli et al — bilingual participants had larger parietal cortex
Localisation studies:
Dougherty et al — 44 OCD patients with cingulotomy (lesioning of the cingulate gyrus), in 32 weeks 1/3 had successful response
Suggests symptoms/behaviours of mental disorders are localised
Lashley — 10-50% of rat’s cerebral cortex removed, still able to learn a maze, proves that no area is more important
Learning is too complex to be localised, whole brain involved
However, humans have more highly developed cerebral cortex so it is difficult to draw conclusions
‘Law of equipotentiality’ that any part of brain can perform any function (holism)
functional recovery
Functional recovery is brain plasticity as a result of brain damage, by transferring damaged functions to undamaged areas of the brain.
Axon sprouting — growth of nerve endings
Reformation of blood vessels
Recruitment of homologous areas on opposite side to perform tasks
Hemisphere removal:
Danelli et al
EB, operated on at 2 ½ to remove tumour in LH
His linguistic ability was impaired
Intensive rehab = no language problems by 8
At 17, he was tested and it was found that his RH had compensated and taken over his language abilities, but he still had some problems with grammar and naming objects compared to control group
Jodie miller case study
3 years old, she suffered seizures caused by her RH, the hemisphere was removed
She was able to walk soon after surgery and lives a functional life even today
Factors that affect brain recovery:
Perseverance — dependant on whether the individual puts work and practice into recovery or decides they can’t do it (practice = plasticity)
Age — old age affects recovery due to weakening levels of plasticity over lifespan
Marquez de la plata et al: older patients had less function after brain damage than younger patients
Exhaustion/stress/alcohol — takes a lot of effort to regain functions, these factors make it harder as energy is needed for plasticity
Gender — women recover better because it is believed that their brain function isn’t as lateralised
Education on recovery — Schneider et al:
Aim — investigate if time spent in education is a factor in recovery from brain
Procedure — 769 people who suffered head injuries followed rehab programme
25% had not finished school, 50% had 12-15 years education, 25% graduated university
Results — 40% of uni students left free of disability, only 10% who left school early made full recovery
Conclusion — education is a factor in recovery, suggests that it leads to a higher cognitive reserve and using brain more effectively for compensating function
Evaluations of plasticity/functional recovery:
EB study: ease studies can be weak since they’re not representative and might not consider all the factors that could cause the recovery
Schneider study: Correlation-causation relationship instead of cause-effect relationship, cannot be proven that education was the sole factor
Using fMRIs for research is highly scientific, valid and precise
Practical application, contributes to neurorehabilitation techniques
Medina et al — prolonged use of drugs results in poorer cognitive functioning/dementia
Ramachandran and hirstein — 70% of amputees experience phantom limb syndrome
Plasticity shown to reduce with age, but still continues throughout life
Bezzola et al — 40 hours of golf training for 40-60 year olds changes neural representation of movement