Layers of the Eye:
Fibrous layer
Vascular layer
Inner layer
Key Structures:
Lacrimal caruncle
Optic disc
Central artery and vein
Optic nerve
Anterior chamber (contains aqueous humor)
Cornea (transparent)
Visual (optic) axis
Lens
Pupil
Iris
Fovea centralis
Macula
Superior canaliculus
Superior lacrimal punctum
Inferior canaliculus
Lower (inferior) lid
Ciliary body
Suspensory ligament
Retina
Choroid
Sclera
Posterior chamber (contains vitreous humor)
Lacrimal gland
Lacrimal sac
Nasolacrimal duct
Opening into inferior meatus of nose
Poor vision or blindness can significantly impact activities of daily living (ADL) and independence.
Every structure of the eye is subject to change, which can range from minor, repairable issues to severe, vision-compromising conditions.
Health History:
Systemic diseases that may lead to eye manifestations.
When was the last eye test?
Medications:
Many drugs affect the eye:
Pseudoephedrine (epi): Dilated pupils.
Antihistamines, decongestants: Dry eyes.
Long-term corticosteroids: May lead to glaucoma or cataracts with long-term use.
Beta-blockers (and beta-blockers for glaucoma): Check if the patient is on other beta-blockers; monitor blood pressure.
Hydroxychloroquine for rheumatoid arthritis (RA): May result in retinal toxicity.
Surgical History:
Laser or any other invasive surgeries.
Functional Health Patterns:
Health perception-health management pattern: Consider age, gender, ethnicity.
Family history
Genetic risk: Glaucoma, macular degeneration.
Cognitive-perceptual pattern: Hearing problems may be further compromised by vision issues.
Self-perception-self-concept: Vision loss or impairment can be devastating.
Role-relationship: Impact on daily family roles.
Physical assessment:
Inspection of the eye structure using an ophthalmoscope to visualize blood vessels and the optic nerve.
Focused Assessment:
Initial observation/inspection: Note how they are dressed; if they cover their eyes from the light; how they reach to shake your hand (depth perception). These are clues to underlying visual changes.
Symmetry and normal placement on the face.
Exophthalmos: Bulging of the eyes.
Brows, lashes: Note any loss of hair.
Ptosis: Drooping.
Functional Status:
Visual acuity (Snellen chart: 20 feet away): Understanding what 20/40 vision means.
Pupil function.
Accommodation.
Conjunctiva and sclera: Color (clear) or smoothness, foreign bodies.
Cornea: Should be clear, transparent.
Iris: Not bulging, similar color.
Optical nerve: Creamy yellow; examine the size and shape. Look for any tears, detachments, or lesions (diabetes or hypertension may present small hemorrhages).
Eye movement: EOM, nystagmus.
Palpate over lacrimal glands, puncta, and nasolacrimal duct for tenderness.
Diagnostic Tests:
Ultrasound, fluorescein angiography, perimetry (visual field) testing.
Xanthelasma: Yellow growth appearing on or by the corner of the eyelids next to the nose, may indicate diabetes.
Arcus senilis: Affects the cornea.
Structural Changes:
Ptosis: Drooping upper eyelid.
Ectropion: Eyelid turns outward.
Arcus senilis.
Functional Changes:
Presbyopia
Far point of vision decreases.
General color perception changes.
Ophthalmoscopy
Other Diagnostic Assessments:
Imaging (CT, MRI, US of retina and optic nerve)
Tonometry
Fluorescein angiography: Provides information on blood flow through retinal vessels.
Refractive Errors:
Refraction is the eye’s ability to bend light rays so that they fall on the retina.
Myopia: Nearsightedness.
Hyperopia: Farsightedness.
Presbyopia: The loss of accommodation associated with age; the lens becomes firmer and less elastic.
Astigmatism: Uneven or irregular curvature of the cornea.
Most corrected by lenses, refractive surgery, or artificial lens implant.
Nursing Considerations for Older Adults:
Visual Impairment
Legal blindness
Inflammation and Infection affecting the eye, conjunctiva, and cornea.
Sty
Conjunctivitis (bacterial or viral): Hand washing to prevent spreading.
Keratitis: Inflammation of the cornea.
Dry eye disorder: May need artificial tears or ointments.
Nursing Management:
Access for ocular changes.
Note if a patient needs two different eye drops; stagger the eye drops for better absorption.
If pain, darken the room, consider cold compresses, and administer analgesics.
Corneal Scars:
Abrasion: Scrape injury.
Causes: Infection (keratitis) - bacterial, viral, fungal.
Ulceration.
Corneal Transplant
Age-related (senile cataracts).
Pathophysiology:
An opacity within the lens.
May occur in one or both eyes.
The eye lens is no longer clear; it is cloudy, resulting in cloudy, distorted vision.
Light is unable to reach the retina.
Clinical Manifestations:
Decrease in vision, gradual.
Abnormal color perception.
Increased glare, especially at night (trouble driving).
Diagnosis
Decreased visual acuity.
Opacity is directly observed by ophthalmoscopic exam.
Treatment
Nonsurgical:
Change in glasses prescription.
Magnifiers or strong readers may be needed.
Increased lighting.
Lifestyle adjustment.
Surgical:
Cataract removal.
Pre-op: Outpatient basis.
Post-op: teaching
Phacoemulsification: Small incision is made in the surface of the eye in or near the cornea.
Preop: Cyclopegic medication (anticholinergic that produces paralysis of accommodation and pupillary dilatation)
Mydriatic medication (alpha adrenergic agonist that produces pupillary dilatation)
Postop: home after sedative agents have worn off. Teach safety.
Home care recommendations: avoid activities that increase the IOP (bending, stooping, coughing, and lifting).
Pay attention to head positioning.
Patient and caregiver teaching.
Retinopathy: Microvascular damage to the retina.
Retinal Detachment: Separation of the sensory retina and the underlying pigment epithelium, with fluid accumulation between the 2 layers.
Macular Degeneration: Age-related macular degeneration (AMD) is the most common cause of irreversible central vision loss in people over age 60 in the US.
Deterioration of the macula at the back of the eye.
Dry (more common): Macular cells begin to atrophy, leading to slowly progressive and painless vision loss.
Wet: Development of abnormal blood vessels in or near the macula.
Leaking of blood vessels; blurred or darkened vision; vision distortion.
Wear sunglasses, make home modifications (loss of central vision), follow-up care with eye exam.
Taking supplements (vitamin C, vitamin E, zinc) may help slow progression.
Teach patients to eat dark green leafy vegetables.
A group of disorders characterized by increased intracranial pressure and its consequences, atrophy of the optic nerve, and peripheral field loss.
Optical nerve damage; early detection is key.
Types:
Primary open-angle glaucoma (POAG) – most common (silent).
Acute angle-closure glaucoma (AACG or acute glaucoma): Immediate emergency treatment.
Normal eye: Aqueous humor flows freely through the trabecular meshwork and the canal of Schlemm.
Angle-closure glaucoma: Closure of the anterior angle due to contact between the iris and the trabecular meshwork prevents aqueous humor from exiting, which leads to increased intraocular pressure.
Open-angle glaucoma: The anterior angle remains open, but the canal of Schlemm is obstructed by tissue abnormalities (like a clogged sink).
Clinical Manifestations:
Vision loss develops slowly.
Eventual "tunnel vision"; all peripheral vision is absent.
Diagnosis:
Increased intraocular pressures (normal 10-21 mm Hg).
Measurement of peripheral and central vision.
Treatment:
The main focus is to keep the IOP low enough to prevent patients from developing optic nerve damage.
Chronic open-angle glaucoma:
Medications do not improve vision but prevent further deterioration:
Prostaglandin analogs, adrenergic agonists, beta-adrenergic blockers (watch taking other beta-blockers and contraindications for beta blockers), carbonic anhydrase inhibitors.
Surgery
Laser to lower IOP if necessary.
Nursing Considerations:
Education: Ensure patients adhere to the treatment plan.
Include caregiver.
Sensory deficit
Pain
Impaired role performance
Drug classes and effects on pupils
Glaucoma medication
Drugs used to decrease IOP:
Adrenergic agonists
Beta-adrenergic blockers
Carbonic Anhydrase Inhibitors
Prostaglandin Agonist
Promote safe, independent living for a patient with impaired vision?
Safety
Medication administration
Communication
Preparing food
ADLs
Leisure
What nursing action is most important for the patient with age-related dry macular degeneration?
B. emphasize the use of vision enhancement techniques to improve what vision is present.
2. A 60-year-old patient is being prepared for outpatient cataract surgery, when obtaining admission data from the patient what would the nurse expect to find in the patient history
C. a gradual loss of vision with abnormal color perception and glare