Lesson 6: Potassium
Potassium Overview
- Normal serum potassium: 3.5 - 5.5 mEq/L
- Most abundant intracellular cation.
- Regulates resting membrane potential in nervous, skeletal, and cardiac muscle.
Effects of Potassium Levels
- Hypokalemia (< 3.5 mEq/L): Hyperpolarizes membranes.
- Hyperkalemia (> 5.5 mEq/L): Depolarizes membranes.
Potassium Regulation
- The kidney is the primary regulator of potassium.
- Decreased glomerular filtration (renal failure) increases serum potassium.
Causes of Hypokalemia
- Poor Intake: Dietary deficiency.
- GI Loss: Vomiting, diarrhea, nasogastric suction.
- Renal Loss: Diuretics, metabolic alkalosis, licorice effects.
- Redistribution: Insulin, beta-2 agonists, hyperventilation.
Presentation of Hypokalemia
- Skeletal muscle cramps, weakness, paralysis.
- Can worsen digoxin toxicity.
Causes of Hyperkalemia
- Increased Intake: Potassium supplements, blood products.
- Impaired Excretion: Renal failure, hypoaldosteronism, NSAIDs.
- Redistribution: Acidosis, succinylcholine, beta-blockers.
- Cellular Injury: Tumor lysis, hemolysis, burns, crush injury.
Presentation of Hyperkalemia
- Cardiac rhythm disturbances (EKG changes):
- Peaked T waves, prolonged PR and QT intervals, sine wave pattern at very high levels.
Treatment of Hyperkalemia
- Cardiac Stabilization: IV calcium.
- Redistribution: Insulin + D50, hyperventilation, bicarbonate, beta-2 agonists.
- Elimination: Diuretics, kayexalate, dialysis.
Key Points on Treatment
- Assess total body potassium: 98% stored intracellularly.
- Treat hypokalemia only if low whole body potassium (not redistribution).
- Administer potassium slowly:
- Peripheral line: 10 mEq/hr.
- Central line: 20 mEq/hr.
- Always rule out pseudohyperkalemia due to lab errors before treating hyperkalemia.