Addiction
Categories of Drugs
Common Characteristics:
Stimulants: Arouse and excite activity
Narcotics: Induces narcosis/sleep/stupor
Hallucinogens: Produces hallucinations.
Drug Categories:
Cocaine: Stimulant
MDMA: Stimulant and hallucinogen
Morphine: Narcotic
Heroin: Narcotic
Amphetamines: Stimulant
Fentanyl: Narcotic
LSD: Hallucinogen
Nicotine: Stimulant
Ritalin: Stimulant
Codeine: Narcotic
Psilocybin: Hallucinogen
Mescaline: Hallucinogen.
Australian Drug Use Statistics
Concern over Methamphetamine: Increased potency despite falling overall use.
Trends Among Students (Years 10-12): General downward trend in drug use.
2022/2023 Survey Findings: Increased use of hallucinogens and Ketamine; decreased use of opioids and ecstasy.
Reasons for Drug Use: Curiosity for initiation, enjoyment of effects for continuation.
Pharmacology and Addiction
Dopamine Signal Changes: Increases initially, diminishes as rewards become predictable.
Self-Stimulation Regions: Median Forebrain Bundle, Nucleus Accumbens, Lateral Hypothalamus.
Mechanism of Addictive Drugs: Increase dopamine in Nucleus Accumbens.
Cocaine's Effect: Blocks dopamine reuptake transporter (DAT).
Amphetamines: Reverse monoamine transporters and inhibit Monoamine Oxidase.
Cannabis: THC binds to cannabinoid receptors, inhibiting GABA release in VTA.
Nicotine: Stimulates Nicotinic receptors, enhancing dopamine release.
Long Term Effects
MDMA's Impact: Short-term serotonin depletion leads to depression; repeated use risks neurotoxicity and chronic mood issues.
Substance Use Disorders
Definitions:
Tolerance: Decreased drug effectiveness with repeated use.
Withdrawal: Symptoms from cessation of chronic use (physical dependence).
Craving: Desire for drug in its absence (psychological dependence).
Incentive-Sensitization Theory: Craving becomes dominant over time.
Limitations of Dopamine Theory: Aversive stimuli also release dopamine; not all users become addicted.
Brain Regions Affected by Stimulants:
Increased activity in Nucleus Accumbens, Hippocampus, Amygdala (memory), Dorsal Striatum (habits).
Decreased activity in Prefrontal Cortex (exec. function); increased in Orbitofrontal Cortex (reward assessment). Glutamate connections regulate craving.