B cells 2 ppt

Page 1: Introduction to B Cells

  • Title: Overview of B cells

  • Contact: Emily Gwyer Findlay - e.gwyerfindlay@soton.ac.uk

Page 2: Lecture Overview

  • Key Topics Covered:

    • Overview of B cells

    • Structure and diversity of B cell receptors and antibodies

    • B cell development and activation

    • Antibody subclasses and their functions

    • Plasma cells and memory cells

Page 3: B Cell Recombination

  • Heavy Chain Recombination:

    • Crucial for B cell development

  • Light Chain Recombination:

    • Follows heavy chain recombination

  • Key Terms:

    • BONE MARROW, STROMAL CELL, IL-7, KIT, PRE BCR, ANTIGEN-DEPENDENT, IMMATURE B CELL

  • Maturation Process:

    • Progenitor B cells develop in the bone marrow.

Page 4: Function of B Cells

  • Antibody Production:

    • B cells release antibodies responsive to specific antigens.

    • Model suggests production of up to one quintillion unique antibodies.

    • Production depends on the specific antigen exposure each B cell experiences.

Page 5: B Cell Activation and Proliferation

  • Antibody Selection:

    • Processes include Affinity selection, CSR (Class Switch Recombination).

  • B Cell Types:

    • Plasma Cells: Short-lived, produce IgM.

    • Memory B Cells: Long-lived, retain memory for future responses.

Page 6: T Cell Help and Activation

  • Activation Mechanisms:

    • Thymus-Dependent: Requires T helper cells for B cell activation.

    • Thymus-Independent: Activation occurs without T helper involvement.

Page 7: Thymus-Independent Antigens

  • Characteristics:

    • Typically polysaccharides with repeating sugar sequences.

    • Stimulate B cell activation via surface IgM and IgD binding.

    • Release mainly IgM antibodies.

Page 8: Antigen-Dependent Maturation

  • Requirement for T Cell Help:

    • Process known as Linked Recognition

    • T cell assistance is vital for full B cell activation and maturation.

Page 9: The Role of T Helper Cells

  • Interaction Steps:

    1. BCR Binding: BCR binds to antigen.

    2. Antigen Presentation: Antigen internalized and presented via MHC class II.

    3. TCR Recognition: T cells recognize MHC/antigen complex.

    4. Cytokine Release: T cells stimulate B cells to produce antibodies.

Page 10: Cytokine Signaling

  • Key Cytokines in B Cell Activation:

    • IL-4: Drives B cell proliferation.

    • IL-5 & IL-6: Stimulate plasma cell development.

Page 11: Germinal Center Reaction

  • Germinal Center Functionality:

    • Site for B cell proliferation and differentiation.

    • Includes processes of somatic hypermutation, affinity maturation, and class switching.

Page 12: Somatic Hypermutation

  • Processes Involved:

    • Introduces point mutations in the V region.

    • Enhances antigen binding affinity through natural selection of B cells with improved receptors.

Page 13: Mechanism of Somatic Hypermutation

  • Molecular Mechanism:

    • AID (Activation-Induced Cytidine Deaminase): Facilitates mutations and receptor refinement.

Page 14: Affinity Maturation

  • Selection Process:

    • Continual refinement of antibody affinity through selection of improved antigen binders.

Page 15: Vaccination Impact

  • Higher Affinity Antibodies:

    • Vaccination enhances production of high-affinity antibodies against specific pathogens.

Page 16: Class Switching

  • Mechanism of Class Switching:

    • Occurs through DNA recombination following T cell help.

    • Produces IgG, IgA, or IgE antibodies, changing effector functions.

Page 17: Mechanism of Class Switching

  • Functionality Remains:

    • Variable region (VDJ) unchanged but constant region altered to modify antibody function.

Page 18: Diversity Generation

  • Antigen-Independent vs. Dependent Mechanisms:

    • Independent: Involves pairing of heavy/light chains and junctional diversity.

    • Dependent: Involves somatic hypermutation, class switching, and affinity maturation in response to antigen exposure.

Page 19: Functions of Antibodies

  • Class Functions:

    • Neutralization: Inactivates toxins and viruses.

    • Opsonization: Marks pathogens for phagocytosis by immune cells.

    • Complement Activation: Enhances immune response.

Page 20: Fc Receptors

  • Characteristics:

    • Bind specifically to the Fc region of antibodies.

    • Distributed across various immune cell types including NK cells and macrophages.

Page 21: Roles of Fc Receptors

  • Functions:

    1. Enable phagocytosis through antibody binding.

    2. Facilitate ADCC (Antibody-dependent cell-mediated cytotoxicity) by NK cells.

Page 22: Cytokine Production by B Cells

  • Production Rate:

    • Slower than T cells; not a critical feature.

  • Role in T Cell Migration:

    • B cells secrete chemokines to recruit T cells, regulating immune responses.

Page 23: Antibody Subclasses

  • Types of Antibodies:

    • IgM: First antibody produced;

    • IgD: Surface receptor, expressed on activation;

    • IgG: Most abundant, crosses placenta;

    • IgA: Found in mucosal areas;

    • IgE: Associated with allergic responses.

Page 24: IgM & IgD Features

  • IgM: Pentameric structure with high avidity.

  • IgD: Not secreted, involved in B cell signaling.

Page 25: Role of IgG

  • Characteristics:

    • Most prevalent in sera; critical for placental immunity and cellular responses.

  • Subclasses: Different IgG subclasses have varied roles in immunity.

Page 26: IgA & IgE Functions

  • IgA: Primarily at mucosal surfaces; vital for gut defense.

  • IgE: Least common; primarily involved in allergy and parasite defense.

Page 27: Summary of Antibody Functions

  • Functional Activities:

    • Various Ig isotypes involved in neutralization, opsonization, and attack pathways against pathogens.

Page 28: Implications of CD40L Deletion

  • Effects on Antibody Production:

    • Lack of class switching affects the antibody profile.

Page 29: Memory B Cells

  • Differentiation Process:

    • Controlled by BLIMP1; memory B cells have high specificity for pathogens.

Page 30: Plasma Cells

  • Production Capacity:

    • Can produce 2000 antibodies/second but have a short lifespan.

Page 31: Memory B Cells in Response

  • Response Characteristics:

    • Rapid and higher affinity production of antibodies upon antigen re-exposure.

Page 32: Immune Response Dynamics

  • Primary vs. Secondary Response:

    • Distinct differences in antibody concentration and classes produced during infection phases.

Page 33: Dysregulated B Cell Responses

  • Systemic Lupus Erythematosus (SLE):

    • Condition where B cells produce antibodies against self-antigens due to survival of self-reactive B cells.

Page 34: SLE Statistics

  • Demographics & Impact:

    • Predominantly affects women; causes significant health issues including renal complications.

Page 35: Summary of B Cell Development

  • B Cell Receptor Structure:

    • Made of heavy and light chains; VDJ recombination leads to diversity.

Page 36: Summary of Key B Cell Processes

  • Key Processes:

    • Antigen presentation to T cells, engagement in somatic hypermutation and affinity maturation, resultant antibody functions in immune responses.