Unit 06 Pt5
Unit Overview
Unit 06: The Cytoskeleton in BIOL 331 – Advanced Cell Biology
Text Ref: MBoC (7th Ed): Chapter 16, Pages 1016 - 1018
Key Topics by Authors
Authors:
Hubert, Robert. Gould's Pathophysiology for the Health Professions, 6th Edition. Elsevier (HS-US), 2017
Banasik, Jacquelyn. Pathophysiology, 6th Edition. Elsevier (HS-US), 2018
Outline of Topics:
Cell Polarity Governed by Small GTPases in Budding Yeast
PAR Proteins and Anterior–Posterior Polarity in Embryos
Conserved Complexes Polarize Epithelial Cells and Control Growth
Dynamic Cell Polarity in Cell Migration
External Signals Directing Cell Migration Direction
Cytoskeletal Communication Supporting Cell Polarity and Locomotion
The Crawling Cell
Types of Cells That Move by Crawling:
Examples: Amoebae, bacteria, yeast, neural crest cells in development, germ cell migration in Drosophila, adult migratory cells (neutrophils, macrophages, osteoclasts, fibroblasts), and cancer cells (metastasis).
Mechanism of Cell Crawling
Crawling Process:
Protrusion: Actin-rich structures extend forward.
Attachment: The actin cytoskeleton connects to the substratum.
Traction: The cytoplasm is drawn forward.
Characteristics: Depending on cell types, movements can be smooth (e.g., fish keratocytes) or jerky.
Protrusions of the Leading Edge
Types of Protrusions:
Filopodia: Long, thin, 1-D structures (e.g., growth cones of neurons).
Lamellipodia: Wide sheets, 2-D structures filled with a cross-linked actin mesh (e.g., seen in epithelial cells and fibroblasts).
Invadopodia (Podosomes): Larger projections, 3-D forms crucial for tissue barrier crossing (metastasis).
Bleb: Formed when the plasma membrane detaches from the cortex.
Case Study: Keratocytes
Description::
Epithelial cells from frog or fish epidermis, specialized for rapid movement (~30 μm/min).
Characterized by large lamellipodial extensions and small trailing cell bodies.
Notably used in wound healing; allows for observation of movement over time.
Lamellipodia Dynamics
Characteristics:
Actin networks are stationary regarding the substrate; exhibits treadmilling where plus ends point towards migration direction.
Branching of actin is facilitated by ARP complexes, creating a characteristic 70° angle.
Maintaining Unidirectional Movement
Requirements for Movement:
Interacting proteins for filament nucleation at leading edge must coordinate with depolymerization behind.
Cofilin preferentially binds to D-form actin, promoting treadmilling essential for directional movement.
Actin-Associated Proteins
Functional Role:
Focal adhesions connect migrating cells to the extracellular matrix.
Sarcomere-like contractions of myosin orient actin filaments to facilitate forward motion, ensuring engagement of integrins.
Regulation of Actin Rearrangement in Migration
Importance of Coordination:
Cell migration necessitates synchrony between protrusion and retraction.
Rho protein family (Cdc42, Rac, Rho) regulates actin dynamics and polarization.
Rho Proteins Overview
Functionality:
Small monomeric GTPases act as molecular switches in signaling pathways.
Activated Cdc42 induces filopodia, Rac drives lamellipodia, and Rho promotes stress fibers and focal contact formation.
Cdc42 Activation and Actin Polymerization
Mechanism:
Cdc42-GTP stabilizes WASp proteins to promote actin nucleation through the ARP 2/3 complex, enhancing cellular motility.
Other Examples of Cell Migration
Chemotaxis:
Cell movement in response to chemical gradients (e.g., neutrophils to bacterial infection, Dictyostelium towards cAMP).
Discussion Questions (Unit 06 – Part 5)
How does a cell crawl and what protrusions are involved?
Why are keratocytes an effective model for studying cell movement?
What roles do Rho family proteins play in cellular dynamics?
What factors stimulate cellular movement and how is this initiated?