subject guide notes
D1.3.1—Gene mutations as structural changes to genes at the molecular level
most common mutations are: insertions, deletions, and substitutions
cause structural changes to gene
D1.3.2—Consequences of base substitutions
SNPs are the result of base substitution mutations & are most common type of genetic variation
due to degeneracy of genetic code, the change in the DNA sequence may or may not hv an effect on structure of protein
depends if substitution is synonymous or non-synonymous
D1.3.3—Consequences of insertions and deletions
frameshift mutations are caused by either insertions or deletions
alter the reading frame of the codons DNA
changing even one nucleotide in the DNA sequence, means that the wrong mRNA sequence is transcribed (the mRNA sequence is missing a codon, or has the wrong codon)
this means there is wrong order of amino acids during translation, and therefore the wrong protein is synthesised, or the protein has the wrong shape & loses its functionality
D1.3.4—Causes of gene mutation
can occur by errors in DNA replication or repair, or can be caused by mutagens
2 types of mutagens - chemical & radiation
examples of chemical mutagens: mustard gas, nitrous acid, formaldehyde, and more
specifically for nitrous acid, it can convert cytosine to uracil through deamination, making it mutagenic
this can result in errors during DNA replication & repair, potentially causing mutations
exposure to radiation can damage DNA, resulting in single-strand breaks, double-strand breaks, or chemical modifications to DNA bases
can disrupt DNA replication
can cause cancer
ex: single-stranded break in occurring in DNA template strand can impact movement of replication fork
can disrupt continuity of replication fork, resulting in replication errors, or breaks in replication all together
D1.3.5—Randomness in mutation
mutations can occur anywhere in base sequence of genome
certain bases do hv higher susceptibility to mutations than others
ex: cytosine has higher mutation rate than other nitrogenous bases, as it can undergo a reaction called deamination, where it loses an amino group
results in conversion of cytosine to uracil
environmental conditions could also impact frequency & types of mutations
D1.3.6—Consequences of mutation in germ cells and somatic cells
mutations in somatic cells can cause diseases in a person’s lifetime
mutations in somatic cells aren’t heritable
so the mutations may cause cancer during the person’s lifetime, but the mutations wont be passed on to offspring
mutations in germ cells are heritable
can alter chromosome number or gene sequence in gametes
can increase the offspring’s susceptibility to certain diseases, or can cause genetic disorders
D1.3.7—Mutation as a source of genetic variation
gene mutation is the original source of genetic variation
can be harmful or beneficial for organism, but in the long term, they’re essential for evolution by natural selection
D1.3.8—Gene knockout as a technique for investigating the function of a gene by changing it to make it inoperative
gene knockout helps scientists understand the role of certain genes, in the organism’s growth or development
understanding the gene’s role can help improve treatments
library of knockout organisms are available for scientists
are organisms that hv been genetically modified to hv one or more of their genes, knocked out
D1.3.9—Use of the CRISPR sequences and the enzyme Cas9 in gene editing
is composed of enzyme Cas9
scientists created single-guide RNAs to target specific genes for modification or deletion
it will target & bind to a specific DNA sequence
will guide Cas9 enzyme to that location
allows it to make cuts to the DNA, resulting in double-strand breaks
scientists can then add, modify, or delete the DNA at the location of the cuts
CRISPR technology has been used in:
gene therapy - has potential to correct mutations that cause diseases (such as sickle-cell anemia)
agriculture, in order to increase crop yield & disease resistance
disease modelling in animals
genetic engineering of microorganisms & can enhance their ability to produce important compounds
D1.3.10—Hypotheses to account for conserved or highly conserved sequences in genes
one hypothesis is functional constraints
are selective pressures that prevent accumulation of mutations, as these can impact an organisms survival
second hypothesis is slower rates of mutations
natural selection pushes mutation rates down by power of random genetic drift
HBA chain (codes for haemoglobin alpha chain) is example of highly conserved sequence