FUNDAMENTALS
I. VIRUSES
A. Definitions
- Virus: A minute infectious agent, filterable and sub-microscopic, approximately 20-300 nm in size, that consists of either DNA or RNA wrapped in a protein coat.
- Infectious Agent: An agent composed of nucleic acid (RNA or DNA) and a protein shell (capsid), and in some cases, a lipid envelope, capable of replication only in living cells using the host cell's machinery.
- Virion: The complete virus particle, with full capacity for replication when a susceptible target cell is encountered.
B. Structure
- Capsid: The protein coat surrounding viral nucleic acid, composed of repeating protein subunits called capsomeres. Capsids exhibit either helical or icosahedral symmetry.
- Nucleocapsid: The complex of protein and nucleic acid.
C. Characteristics
- Obligate Intracellular Parasites: Viruses cannot replicate outside living cells. They cannot generate energy or synthesize proteins independently.
- Replication Process: One virus can replicate to produce hundreds of progeny viruses, differing from cellular division mechanisms like binary fission or mitosis.
D. Chemical Composition
- Viruses consist of:
- Nucleic Acid: 1-40%
- Protein: 70-90%
- Lipids: 15-35% (including phospholipids, glycolipids, cholesterol)
E. Size
- Viral particles typically range from 20 to 300 nm in diameter, comparable to sizes of large proteins to small cells.
F. Shape
- Basic shapes of viruses include:
- Icosahedral
- Helical
- Complex (e.g., pox viruses)
G. Taxonomic Characteristics
- Viral classification is less structured than other organisms and is based on features like nucleic acid type, strandedness, presence of an envelope, capsid shape, and host specificity.
H. Viral Constituents
- Some viruses also include additional structures like an envelope derived from the host cell membrane, which may include spike-like proteins (glycoproteins).
II. ENVELOPED VIRUSES
A. Structure And Shape
- Envelope: A lipid-containing membrane acquired during viral maturation. Not all viruses have envelopes.
- Enveloped viruses can exhibit various shapes due to the fluid nature of the envelope.
B. Viral Envelopes
- Envelopes provide:
- Protection against the host immune system.
- Facilitation of fusion with host cell membranes during infection.
C. Characteristics
- Enveloped viruses are more sensitive to heat, detergents, and solvents than non-enveloped viruses, which tend to be more stable in adverse conditions.
- Enveloped viruses require certain environmental conditions to maintain stability.
D. Functions Of Viral Capsid/Envelope
- Protects viral nucleic acid from enzymatic degradation.
- Facilitates introduction of viral genetic material into host cells.
- Stimulates immune response, signaling production of antibodies.
III. NON-ENVELOPED VIRUSES
A. Virus Structure And Shape
- Naked Viruses: Composed only of nucleic acid and capsid proteins. Typically more resistant to environmental conditions.
B. Taxonomic Characteristics
- Different families of non-enveloped viruses (e.g., Picornaviridae) demonstrate varying pathogenicity and infectivity.
C. Viral Nucleic Acids
- The viral genome can be linear or circular, and consist of either single-stranded or double-stranded RNA or DNA.
D. Viral Capsid And Symmetry
- Capsid configurations include:
- Icosahedral (20 triangular faces)
- Helical (hollow coil)
E. Viral Proteins
1. Functions Of Surface Membrane Proteins
- Viral surface proteins are crucial for attachment and entry into host cells, and also play a role in evading the host immune response.
IV. CLASSIFICATION OF VIRUSES
A. Properties Of DNA and RNA Viruses
DNA Viruses
- Usually double-stranded, reside mainly in the nucleus, interact with host transcription machinery.
RNA Viruses
- Generally single-stranded, replicate primarily in the cytoplasm, require RNA-dependent RNA polymerase for replication.
V. VIRAL PATHOGENESIS
A. Factors Involved In Viral Pathogenesis
- Viral factors encoded in the genome influence the virulence and tropism of the virus.
B. Disease Caused By Viruses
- Consequences of viral infections may include cell injury, tissue damage, and immune response variations leading to disease manifestations.
C. Sequential Steps In Viral Replication
- Main Stages Include:
- Attachment
- Entry
- Uncoating
- Replication
- Maturation
- Release
D. Major Paths Of Virion Entry
- Major routes of entry include respiratory tract, oropharynx, genital tract, and skin.
E. Fundamental Aspects Of Viral Pathogens
- Viruses must bypass host defenses, spread effectively, and modulate host responses for successful replication and transmission.
F. Mechanisms Of Virus-Induced Disease
- Pathogenesis can involve direct killing of host cells, immune-mediated damage, and chronic infection with potential for later disease complications.
G. Virus-Host Interactions
- Interactions between viruses and host determine the severity and outcome of infections, including acute and chronic disease states.
H. Cellular Infection Types
I. Steps Of Pathogenesis
- Virus entry, replication, spread, specific cellular infection, host immune interaction, and persistence may occur in various levels depending on the virus.
VI. ANTIVIRALS
A. Control Of Viral Infections
- Antivirals target:
- Viral replication enzymes (e.g., nucleoside inhibitors for reverse transcriptase)
- Proteases inhibiting viral life cycles in infection.
- Entry and exit pathways (e.g., inhibitors for HIV and influenza).
VII. REFERENCES
- Use of antiviral agents, vaccines, historical context, and clinical practices were highlighted in academic resources such as Jawetz, Melnick, & Adelberg's Medical Microbiology.
VIII. PRACTICE QUESTIONS
- Detailed comprehension questions focused on properties, classifications, and behaviors of viruses.
IX. APPENDIX
- Includes additional insights, illustrations, and figures detailing various virus characteristics and examples for a clearer understanding of their operation.