Metabolic Disorders in Cattle Notes
- Milk fever
- Ketosis
- Fat cow syndrome
- Retained placenta
- Displaced abomasum
- Rumen acidosis
- Laminitis
- Udder edema
Milk Fever (Parturient Paresis in Cows)
- Also known as hypocalcemia.
- Characterized by flaccid paralysis occurring most commonly at parturition and marked by changes in mentation, generalized paresis, and circulatory collapse.
- Clinical signs include:
- Sternal Recumbency: Cows may display an 'S' shaped neck with a bulging rectum.
- Serum calcium levels drop sharply from normal (10-12 mg/dL) to critically low levels (2-7 mg/dL).
- Changes in serum phosphorus and magnesium.
- Etiology:
- High-producing dairy cows >5 years old are particularly affected.
- Decreased serum calcium linked to the onset of lactation.
Stages of Parturient Paresis
- Stage 1:
- Cows are ambulatory but show signs of hypersensitivity, excitability, mild ataxia, tremors, and restlessness.
- Stage 2:
- Unable to stand but can maintain sternal recumbency. Signs include low heart rate, reduced body temperature, and GI stasis manifested as bloat and inability to urinate.
- Stage 3:
- Progressive loss of consciousness, muscle flaccidity, unresponsive to stimuli, and risk of severe bloat. Risk of cardiac failure is high.
Treatment of Milk Fever
- Restore normal serum calcium levels quickly:
- IV Calcium Gluconate: Recommended dose is 1 g of calcium per 45 kg (100 lb) body weight.
- SC and oral calcium administration in addition to IV treatment can aid absorption but may take longer to effect.
- Monitor for cardiotoxic effects of calcium; administer slowly, observing heart rhythms.
- Approximately 75% of cows respond within 2 hours of treatment. However, relapses can occur in 25-30% of cases within 48 hours.
Prevention of Milk Fever
- Feeding strategies:
- Low calcium diets during the dry period promote better calcium absorption in preparation for lactation.
- Delayed milking post-calving can help manage calcium levels but may increase the risk of mastitis.
- Dietary Cation-Anion Difference (DCAD): Feeding anionic salts helps maintain appropriate calcium metabolism by lowering blood pH and enhancing calcium resorption from bone.
- Management of Body Condition: Maintain ideal body weight (~3-3.5) prior to calving.
Ketosis
- A condition due to inadequate nutrient intake leading to low blood glucose and fat mobilization, causing elevated ketone levels.
- Symptoms include loss of appetite, weight loss, rumen inactivity, and decreased milk production. Testing blood and urine for ketone levels helps diagnose.
Treatment and Management of Ketosis
- Aim to increase blood glucose quickly through:
- IV Glucose Administration: Quick but may require follow-up treatments.
- Hormonal Treatments such as glucocorticoids can stimulate glucose production.
- Oral Sugar Precursors: Sodium propionate and propylene glycol can enhance glucose availability.
- Preventive measures include avoiding over-conditioning of cows before calving, ensuring high-quality forage post-calving, and proper nutritional management to avoid negative energy balance.
Fat Cow Syndrome
- Typically occurs in over-conditioned cows. Similar symptoms to ketosis, including poor appetite and depression but harder to treat. Prevention focuses on managing weight during dry periods.
Retained Placenta
- Defined as the failure of the placenta to detach from the uterine wall post-calving, commonly affecting high-producing and older cows.
- Prevention strategies include ensuring adequate nutrition and minimizing stress conditions.
Displaced Abomasum (DA)
- A condition where the abomasum twists, commonly occurs within 6 weeks post-calving.
- Symptoms include lack of appetite, low milk yield, and abnormal feces.
- Management involves nutritional strategies, feeding bulky diets, and if necessary, surgical intervention.