Intro to ECGs/EKGs Notes
Intro to ECGs/EKGs
Expectations and Objectives
- The goal is not to become an expert in reading ECGs in one lecture.
- The aim is to familiarize with identifying rhythms and determining heart rates for board exams and clinical practice.
- This lecture covers basic physiology of the heart and components of rhythm strips.
- Clinical applications and decision-making will be discussed in the lab due to multifactorial considerations.
- The content is based on the textbook, providing background information on why certain rhythms occur.
- Learning objectives focus on understanding and interpreting ECGs.
Cardiac Cycle
- The cardiac cycle includes atrial contraction (extra cardiac kick), ventricular contraction, and valve opening/closing.
- ECG correlates:
- P wave: Atrial contraction
- QRS complex: Ventricular contraction
- T wave: Ventricular repolarization
Purpose of ECGs
- Monitor heart activity.
- Electrocardiogram provides the most cardiac function information non-invasively.
- Monitors heart's electrical activity (action potential), not actual contraction force.
- Methods:
- Exercise ECGs (stress tests)
- Holter monitors (continuous monitoring during exercise or at home)
Technical Terms
- Aberrant: Abnormal, diverging from the expected rhythm pattern.
- Arrhythmia: Absence of rhythm, pause, or impulse. Often referred to as dysrhythmia (bad rhythm).
- Dysrhythmia: Abnormal rhythm, not necessarily bad, but often used to describe aberrant rhythms.
- Ectopic: Abnormal place or position (e.g., ectopic heartbeat starts from somewhere other than the SA node).
- Sinus: Related to the SA node (e.g., sinus rhythm is normal rhythm originating in the SA node).
Basic Principles
- Automaticity: Heart's ability to contract without external nerve stimulation.
- Rhythmicity: Regular pattern of heartbeats (regular is good; irregular may cause concern).
- Conductivity: Nerve impulses travel through cardiac muscles.
Conduction System
- Impulse originates at the SA node (right atrium).
- Travels through:
- Internodal pathways
- Bachmann bundle (to left atrium)
- AV node (provides pause for atrial kick)
- Bundle of His
- Right and left bundle branches (along the septum)
- Purkinje fibers (ventricular muscles)
Components of Conduction System
- SA node: Pacemaker cell in the right atrium.
- Functions rhythmically.
- At rest, beats in a regular rhythmic pattern.
- Internodal tracts transmit from the SA node to the AV node.
- AV node passes impulses to the ventricles.
- AV bundle (bundle of His) splits into right and left bundle branches, then Purkinje fibers.
Depolarization and Repolarization
- Depolarization: Changes in heart's electrical charges, leading to contraction.
- Resting membrane potential is slightly negative.
- Depolarization makes it less negative.
- Repolarization: Restoration of membrane potential.
- Involves movement of sodium, potassium ions, and sodium-potassium pump.
Action Potential in Conducting Cells
- Resting state is approximately -60 millivolts.
- Slow influx of sodium makes it less negative.
- Rapid influx of calcium makes it sharply positive (depolarization).
- Outflux of potassium restores the resting potential (repolarization).
Action Potential in Cardiac Muscles
- Rapid depolarization followed by a plateau phase.
- Plateau phase: Slow calcium channels open, leading to contraction.
- Long repolarization period.
- Absolute Refractory Period: Muscles will not contract, regardless of the strength of the impulse.
- Relative Refractory Period: Muscles might contract if the impulse is strong enough.
- Significance: Long period allows the heart to fully contract, maximizing stroke volume.
Skeletal Muscle vs. Cardiac Muscle
- Skeletal muscle contraction is quick with a rapid tension release.
- Cardiac muscle builds tension over time for a sustained contraction.
- Skeletal muscle has a short refractory period, while cardiac muscle has a long absolute refractory period.
- Cardiac muscle's long refractory period prevents off-rhythm contractions.
Measuring Depolarization with ECGs
- Electrode placement (leads) is essential.
- Positive lead: Wave of depolarization moving towards it causes an upward deflection.
- Negative lead: Wave of depolarization moving away from it causes a negative deflection.
- Repolarization has the opposite effect.
- Law of Averages: The left ventricle is larger, so its impulse magnitude is greater and more towards the left.
ECG Leads
- 12-lead ECGs: Multiple views of the heart.
- Rhythm strip: Single lead view.
- 12-lead ECG includes six chest leads and six limb leads.
12-Lead ECG vs. Single-Lead
- 12-Lead ECG:
- Determines ischemia or infarction.
- Compares to previous recordings.
- More descriptive.
- Assesses heart rate, rhythm, hypertrophy, and infarction.
- Single Lead:
- Assesses heart rate, rhythm, and abnormal rhythms.
Electrode Placement
- Electrode placement is different from lead placement.
- Electrodes can be positive or negative, depending on the lead.
- Standard placement: Right arm, left arm, right leg (ground), left leg, and precordial leads (V1-V6).
- Avoid placing electrodes on large muscle portions to reduce noise.
Limb Leads
- Lead I, II, III, AVR, AVL, AVF.
- Lead I: Left lateral lead (left arm positive, right arm negative).
- Lead II: Inferior lead (left foot positive, right arm negative).
- Lead III: Inferior lead (left foot positive, left arm negative)
- AVR: Right arm positive, left arm and left leg negative.
- AVL: Left arm positive, right arm and left foot negative.
- AVF: Left foot positive, arms negative.
- Right foot acts as a ground.
Chest (Precordial) Leads
- V1-V6 placed horizontally across the chest to view the heart in the transverse plane.
- Each lead views cardiac activity from a different angle.
- V1 is on the right side of the sternum, while V6 is towards the midaxillary line.
Understanding Deflections
- If depolarization moves towards a positive lead, there's an upward deflection.
- If it moves away, there's a downward deflection.
- V1: Normally has a large downward deflection because the majority of the impulse goes towards the left ventricle.
- V6: Normally has a large upward deflection because the impulse goes towards the left ventricle.
ECG Strip
- Typically a six-second strip with tick marks at the top (each tick mark represents three seconds).
- Each large box: 0.2 seconds.
- Each small box: 0.04 seconds.
- Amplitude: Each large box is 0.5 millivolts.
ECG Components
- P wave: Atrial depolarization.
- PR interval: Includes P wave and pause segment (0.12-0.2 seconds).
- PR segment: End of P wave to beginning of QRS complex.
- QRS complex: Ventricular depolarization.
- R wave: First upward deflection.
- Q wave: First downward deflection.
- S wave: Downward deflection after the R wave.
- R prime: Second upward deflection after the S wave.
- ST segment: After the S wave to the beginning of the T wave.
- T wave: Ventricular repolarization. Atrial repolarization is not seen because it occurs during the QRS complex.
Normal vs. Abnormal ECG
- Normal P wave, PR interval, QRS complex, and T wave.
- Expected time frame for each component.
Reading ECGs: Two Approaches
- Approach 1 (Textbook): Evaluate P wave, PR interval, then heart rate, etc.
- Approach 2 (Suggested): Start big (observe patient, rhythm) and move to small (evaluate waves and intervals).
Suggested ECG Reading Approach
- Observe the Patient: Is the patient in distress? Expect normal ECGs if the patient is stable; suspect abnormalities if distressed. Note any symptoms. (chest pains, fatigue, shortness of breath)
- Evaluate the Rhythm: Is it regular or irregular? Regular rhythms suggest normalcy, while irregular rhythms need further investigation.
- Estimate the Heart Rate:
- Method 1: Count QRS complexes in a six-second strip and multiply by 10.
- Method 2: Use bold boxes to estimate rate based on 300, 150, 100, 75, 60.
- Method 3: Count small boxes between QRS complexes and divide 1500 by that number.
P Wave Abnormalities
- Normal P wave: Rounded, symmetrical, upright, < 0.12 seconds, < 2.5 mm
- Right Atrial Enlargement: Large amplitude.
- Left Atrial Enlargement: Prolonged duration.
Ventricular Hypertrophy
Right Ventricular Hypertrophy
Increased R wave in V1 and increased S wave in V6.
R wave in V1 is larger than the S wave.
Left Ventricular Hypertrophy
Increased R wave in left leads, increased S wave in right leads.
Use math to determine if the sum of R and S waves exceeds normal measurements.
Rhythm Abnormalities
Normal Sinus Rhythm: Normal rhythms originating from the SA node.
Sinus Pause: SA node fails to initiate an impulse for one cycle. Generally benign.
PREMATURE ATRIAL COMPLEX
Premature Atrial Complex: The SA node fires early causes an irregular rhythm. Generally Benign.
- No significant signs or symptoms
WANDERING ATRIAL PACEMAKER
Wondering Atrial Pacemaker: The P waves look different, meaning the impulse is initiated outside the SA node.
- May require medical treatment to prevent atrial fibrillation
ATRIAL FLUTTER
Atrial Flutter: Rapid succession of atrial depolarization caused by an ectopic focus that depolarizes at a rate of 250-350 times per minute
- Characterized by a sawtooth pattern. Treatment: Medication.
ATRIAL FIBERLATION
Atrial Fibrillation: Erratic quivering, causes by multiple ectopic foci creating an irregular, and irregular heart rhythm.
- Requires Treatment such as drugs or a pacemaker
- May decrees cardiac output, but not necessarily life threatening
HEART BLOCKS
FIRST DEGREE AV BLOCK
First-Degree AV Block: Prolonged PR interval. Treatment usually not warranted.
SECOND DEGREE AV BLOCK TYPE 1
Second-Degree AV Block Type 1 (Wenckebach/Mobitz I): Lengthening PR interval until a QRS complex is dropped. Treatment usually not necessary.
SECOND DEGREE AV BLOCK TYPE 2
Second-Degree AV Block Type 2 (Mobitz II): Non-conduction of impulse to the AV node. Requires medical attention because leads to possibility of a complete heart block
THIRD DEGREE AV BLOCK
Third-Degree AV Block: Medical Emergency, because the p waves and QRS, the ventricles depolarize from somewhere other then the AV node are not corrdinating.
- Complete AV Block: Lack of coordination between P waves and QRS complexes.
Ventricular Arrhythmias
PREMATURE VENTRICULAR COMPLEX (PVC)
Premature Ventricular Complex (PVC): Can happen with healthy people
Paired together, Origin(multifocal) and frequent meaning more then 6 and triplets are more serious can causes, ventricular fibrillation.
- Travels along the regular pathway
UNIFOCAL PVC
Unifocal PVC: regular PVC
PVCs are identical. Means that the impulse starts from the same place with the same pathway
MULTIFOCAL PVC
Multifocal PVCs: PVCs looks different, with different pathways. Starting different paths
Irregular Pvcs
RUNNER OF V-TACH
three beat, R on T, Run of V tach: Medical Attention Required
The r happens before the repolarization through the, probably the relative refractory period, then can leads to ventricular fibrillation.
VENTRICULAR TACHYCARDIA
V1 is where looks like relatively normal if V1 looks like the abnormal on Medical Attention Required
TORSADES DE POINTES
- Rare ventricular tachycardia, increasing/decreasing amplitude. Medical emergency, cardioversion is used.
VENTRICULAR FIBERLATION
- Erratic quivering of the ventricular muscle. No pulse or blood ejected. Medical emergency requiring resuscitation.
STEMI and NSTEMI
- STEMI (ST-Elevation Myocardial Infarction) indicates ischemia or infarction.
- ST Segment: Period from end of S wave to beginning of T wave.
- ST-segment elevation: ischemic area
- ST-segment depression: injured cardiac muscle
- T-wave inversion: repolarization traveling differently than expected.
Extra Considerations
- Tick marks indicate pacemaker: Atrial pacing or ventricular pacing.
- Implanted cardioverter defibrillator (ICD): Detects tachycardia and delivers a shock.