Voluntary Control
Balance
Balance: The ability to maintain postural equilibrium by controlling our Center of Mass (CoM) and Base of Support (BoS)
CoM: The point at which the whole body, or individual segment, mass is equally balanced
Center of gravity (CoG): Projection of CoM on the floor
BoS: Usually only the feet in contact with the ground, but could incorporate other objects used for balance
CoM must stay within the base of support (BoS)
If the CoM leaves the BoS, increased chance of falling
Anticipatory postural adjustment (APA):
Feedforward for anticipatory postural instability
Plan movements based on sensory input
Postural adjustments that occur prior to task
Stabilize in preparation for the task
Need to anticipate instability that occurs with upcoming movement
NO APAs in response to external perturbations
No time to anticipate the perturbation
Walking:
When walking, the BoS reduces down to one foot
Lifting one leg leads to instability in medial lateral direction
Must shift CoM to a location within the remaining BoS
Automatic postural response (APR):
Feedback for unanticipated postural instability (ex: external perturbations)
Muscle/skin/vestibular feedback signalling unplanned movements
Voluntary movement
Primary motor cortex (M1) – area 4
Movement execution
Premotor areas– both in area 6:
Premotor cortex – area 6
Movement preparation
Supplementary motor area – area 6
Movement planning/initiation
Primary somatosensory cortex (S1) – areas 3a, 3b, 1, 2
Proprioception – 3a & 2
Touch – 3b & 1
Sensory input of movement
Posterior parietal cortex – areas 5, 7
Sensory integration, including vision – current state of body
Primary motor cortex (M1) – area 4
Movement execution
Including: Direction, magnitude, force, speed of movement
M1 neurons respond to individual voluntary movements
M1 neurons fire before movement onset
Synapse directly with ⍺MN & interneurons of individual muscles, and groups of muscle around a joint via corticospinal tracts (medial & lateral)
Lateral corticospinal tract: Synapses on distal muscles (hand, foot)
Ventral-medial (anterior) corticospinal tract: Innervates axial muscles
Full myelination of corticospinal tract coincides with walking in infants, and pincer grip
Premotor cortex – area 6
Movement preparation through selection of appropriate motor plans
Premotor cortex neurons synapse with cells in M1 and signal preparation for movement & observing movement
Integrate sensory aspects for motor acts
Information from S1 important to structure movement
Respond to both making a movement, and watching another make a movement
Mirror neurons
Signal incorrect actions, and involved in motor learning skills
As a monkey learns a new task, neurons within premotor cortex alter firing in response to correct or incorrect rules being learned (Butch et al., 2006)
Are sensitive to behavioural context and instructions
Damage:
Causes difficulties in performing movements in response to visual and verbal commands
A loss of self-initiated movement- therefore, damage impacts APAs
Supplementary motor area (SMA) – area 6
Movement planning/initiation
SMA neurons respond to sequences/mental rehearsal of movement sequences and are involved in creating appropriate complex motor output (timing and gain
SMA is involved in the initiation of movements on the contralateral side of the boduy
A “readiness potential” is seen 0.8-1s before movement begins
Posterior parietal cortex - area 5 and 7 (of association complex)
Integrates sensory modalities for motor planning
Sensory integration, including vision – current state of body
Receives input from S1
Important for sensory integration
Area 5
Integrates tactile & proprioceptive information
Area 7
Integrates visual information
Prefrontal cortex (of association complex)
Working memory: Location of objects in space to guide movement
Memory of consequences of actions
Primary somatosensory cortex (S1) – areas 3a, 3b, 1, 2
Proprioception – 3a & 2
Touch – 3b & 1
Sensory feedback of movement
From dorsal column, peripheral sensory information projects, via 3 synapses, to S1
Synapse 1: Medulla (gracile + cuneate nucleus)
Synapse 2: Ventral posterior lateral (VPL) thalamus
Most thalamic fibers from VPL nucleus terminate in 3a and 3b
Fibers then project to areas 1 and 2
3b & 1 = tactile (skin)
3a & 2 = proprioceptive (skin, spindles, joints)
Synapse 3: Sensory cortex
Damage and therapy
Receptive field: Area of the skin that, when activated, elicits a response from a receptor
Projected fields: Area of the skin that a sensation is felt, following stimulation of the brain
Using cortical stimulation to influence sensation: Receptive fields > projected fields
Phantom limb: Following amputation, patients feel the phantom limb, try and move it, and experience tremendous pain
Cortex rewiring: After amputation, sensations in the phantom limb can be felt by stimulating the face, or the proximal limb
Ex: Hand input and face input rewired to go to same place/overlap
Reasons for phantom limb:
Peripheral nervous system
Damage/swelling in the axon of the nerve at the amputation site
Generates random action potentials
Central nervous system
Damage to pain fibers in spinal cord
Other nerves branch to same tracts to the brain, so some regular sensations could be perceived as pain
Cortical reorganization
Some areas take over other areas
E.g. the face takes over the hand area in M1 and S1
Pain persists as there is inconsistent sensory integration/feedback
Mirror therapy: Utilises vision to enable the sensory and motor feedback to align with the phantom sensations
Prosthetics:
After amputation, afferent and efferent pathways remain largely intact
A peripheral nerve interact is used to implement movement commands & transmit sensory feedback from a prosthesis
