Vaginal Infections
anatomy
Birth Canal → lies between the bladder and the rectum → lower end has the Bartholin’s glands → lined with stratified squamous epithelium that contains glycogen.
glycogen conversion into lactic acid (4-4.5) catalyzed by lactobacillus → helps to regulate pH of vagina → to prevent the risk of prevent vaginal infections (increase in pH level)
pap smear test (not tested) → for cervical cancer → if detected early, good prognosis is very high → check for cancerous cells by going in all the way.
main focus on the organism that colonize the vagina is LACTOBACILLUS 80-90% ; but there are others like Corynebacteria, streptococcus, staphylococcus epidermidis, Gardnerella vaginalis, Peptostreptococcus, Bacteroidesm, E.coli and candida albicans → try not to upset the flora to prevent the overgrowth of these bacteria that cause infection.\
factors that affect the vagina ecosystem
hormonal changes → affects amount of glycogen and the amount of discharge in the area.
aging → menopause etc.
certain diseases (diabetes mellitus: immunocompromised)
use of medication that upset/change the flora (antibiotics/contraceptives)
hygiene
douching/rinsing or washing the inside of your vagina with soap
sexual intercourse (seminal pH is 4-4.5, but it becomes more alkaline during sexual activity)
the number of sexual partners that you have → different organisms
physiology
normal discharge can be around 1.5g of vaginal fluid → odorless, clear or white, thin, viscous or sticky
increase in discharge is common in pregnancy, ovulation, menses or with sexual excitement or emotional flares (moody)
alternation in vaginal secretions can also indicate whether there is higher or lower amount of irritation to the area.
common vaginal infections and differences
Bacterial Vaginosis
Trichomoniasis
VVC (Vulvovaginal Candidiasis) → only one that can be treated w/o prescription antibiotics
all these complications of BV and Trichomoniasis can lead to pelvic inflammatory disease, urinary tract infection, endometriosis and infertility (most important).
BV | Trichomoniasis | VVC | |
prescription? | yes | yes | yes/no |
most common (1st) | 2nd most common | ||
Name of Causative Bacteria | G.vaginalis Anaerobes. | Trichomonoiasis vaginalis | Candida Albicans/Candida Fungi (80-92%) — about 50% of women will have one or more infections caused by this by age 25. OR
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Risk Factors |
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Signs and Symptoms |
| presence of erythema and itching |
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Difference in Odour | Fishy Odour | Foul smell | No smell |
Discharge | Thin and sometimes foamy | Frothy | Thick white, cottage cheese like. |
Color of Discharge | Off-white. discoloured (green/grey) | Yellow-green/grey | White |
Complications | PID/UTI/Pre-term labor/Low birth weight/infertility or transmission of HIV | PID/UTI/Pre-term labor/Low birth weight/infertility or transmission of HIV |
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Exclusion for self-treatment → if you are pregnant, or below 12 years old, concurrent symptoms like fever or pain on back, shoulder or abdomen, on medication that can predispose to VVC (corticosteroids), medical disorders that can predispose to VVC or recurrent VVC (>3 infections per year or in the past 2 months)
antifungal products used → imidazole antifungals (miconazole, clotrimazole) → for uncomplicated VVC → exist as vaginal creams, suppositories and tablets.
advantage → superior efficacy → improved patient compliance due to ease of use → less frequent local reactions and short treatment duration
Medications that need to memorise
Miconazole Nitrate 2% | clotrimazole 1% | Isoconazole | Fluconazole |
Cream → apply daily BD (outer) | Cream → apply BD daily (outer) pessaries → vaginal tablets (inserted) pessaries → 100 mg (1 tab 6 days) pessaries → 200 mg (1 tab 3 days) pessaries → 500 mg (1 table single app) | ovule 600 mg→ 1 tablet single app | 150 mg tablet → single oral dose Triazole Itraconazole and Fluconazole (no need insert and no need prescription → but try not to give because its a very strong antifungal and wouldn’t want any antifungal resistance. |
Mechanism of action | alters the membrane permeability of fungal cells inhibits the CYP450 enzyme → lanosterol-14-alpha-demethylase → reduce ergosterol reduced ergosterol content causes increase in lanosterols like methylated sterols → structural damage to fungal membranes → loss of function for the bacterial cell due to leakage of cellular components | alters the membrane permeability of fungal cells inhibits the CYP450 enzyme → lanosterol-14-alpha-demethylase reduced ergosterol content causes increase in lanosterols like methylated sterols → structural damage to fungal membranes → loss of function for the bacterial cell. | alters the membrane permeability of fungal cells inhibits the CYP450 enzyme → lanosterol-14-alpha-demethylase reduced ergosterol content causes increase in lanosterols like methylated sterols → structural damage to fungal membranes → loss of function for the bacterial cell. |
They are very safe and appreciably absorbed → only potential vulvovaginal burning/itching/irritation → no drug interactions except for warfarin → only harmful if you are allergic to azoles
alternative therapies
use lactobacillus acidophillus preparation
sodium bicarbonate sitz path (1 tsp in 1 pint (450 ml) of water) → 15 mins
gentian violet/potassium permanganate → dilute in 1 pint and use for cleansing of vagina (antiseptics)
itchiness → use hydrocortisone 1% or benzocaine.
patient-counselling
symptoms should improve after 2-3 days and disappear/resolve within a week
avoid tight clothing like jeans or underwear → gives high moisture and humidity while allows microbes to grow.
seek medical evaluation if symptoms persist after a week or if recur after 2 months or occur 3 times within 12 month interval
don’t use perfumed soaps while washing
avoid/don’t douch