Lecturer: Dr. C. Borz
Topic: Chronic Venous Disease - Overview of problems, diagnosis, and management.
Prevalence: Affects 40% of adults.
Components: - Varicose veins
Postthrombotic syndrome
Venous ulcers
Spider veins (telangiectasias)
Central Venous System: Includes IVC, SVC, iliac, and subclavian veins.
Peripheral Venous System: Includes extremity and head/neck veins. - Divided into superficial and deep veins with interconnections.
Lower Extremity Veins: - Greater (long) and lesser (short) saphenous veins, including tributaries (superficial veins).
Deep veins responsible for 90% of blood flow and patency.
Valves: - Bicuspid valves prevent retrograde flow in saphenous veins.
Perforating Veins: - Interconnections between superficial and deep veins.
Incompetency: Leads to retrograde flow and varicose veins.
Muscle Contraction: Aids in blood return to the heart.
Risk Factors for DVT: - Virchow’s triad: stasis, endothelial injury, hypercoagulable states (e.g. pregnancy, contraceptives, surgery, sepsis, obesity, trauma).
Consequences: - Superficial and deep venous thrombosis
Pulmonary embolism
Varicose veins and edema
Chronic venous insufficiency
Secondary varicose veins (due to tumor compression or DVT)
C - Clinic: - C1 to C6 (includes spider veins, edema, skin lesions, etc.)
E - Etiology: - Hereditary, primary, secondary
A - Anatomy: - Superficial, deep, perforating
P - Pathophysiology: - Reflux, obstruction, or both mechanisms
C (Clinic): - C0: No clinical signs
C1: Spider veins
C2: Varicose veins (>3 mm)
C3: Edema
C4a: Hyperpigmentation or eczema
C4b: Lipodermatosclerosis or white atrophy
C5: Healed venous ulcer
C6: Active venous ulcer
E (Etiology): - Ec: Congenital, Ep: Primary, Es: Secondary (postthrombotic)
A (Anatomy): - As: Superficial veins, Ap: Perforator veins, Ad: Deep veins, An: Unspecified veins
P (Pathophysiology): - Pr: Reflux, Po: Obstruction, Pn: Unspecified
Symptoms: - Heaviness, fatigue, night cramps, superficial thrombophlebitis, hemorrhage
Clinical Findings: - Varicose veins and skin changes examined in a standing position.
Can investigate distribution to identify saphenofemoral incompetence.
Mid-thigh Perforating Veins: Can initiate anatomical abnormalities even if the sapheno-femoral junction is intact.
Importance of Valves: - Failure at the junction causes progressive incompetency of valves downstream.
Hydrostatic Pressure: - Causes dilation/tortuosity of tributaries exposed to increased pressure.
Detailed anatomy of veins involved in varicose conditions including the femoral vein and superficial veins.
Initial Examination: - Assess the severity and extent while the patient stands.
Skin Examination: - Look for swelling, ulcers, and varicose eczema.
Varicosities: - Documenting presence above the knee can suggest sapheno-femoral incompetence.
Diagnostic Tools: - Tourniquet Test
Doppler Ultrasound: For assessing venous incompetency and patency.
Other Techniques: Venography and plethysmography.
Differential Diagnosis: Includes hernias, lymph node diseases, arterial diseases, and cancers.
Complications: - Chronic venous insufficiency, thrombosis, rupture.
Obesity may be an aggravating factor.
Surgical Options: - High Saphenous Ligation: Ligation of the saphenous vein at the sapheno-femoral junction.
Process: - Incision and exposure of the vein, ligation of tributaries, closure to prevent reconnection.
Stripping Mechanism: The stripper is introduced into the saphenous vein aiding removal by pulling it out through small incisions and tearing off tributaries.
Post-operative Care: Important to minimize subcutaneous bleeding and manage bruising post-surgery.
Chronic Venous Disease: Recognized as a progressive inflammatory disorder leading to potential complications.- Requires a multidisciplinary team approach for effective management: Includes GP, surgeons, and dermatologists.