CVA
Cerebral Vascular Accident (CVA)
Introduction
Presenter: Amanda Couitt, PT, DPT
Objectives
Understand the etiology of CVA
Identify types of CVA
Recognize functional deficits and symptoms related to the location of the lesion in CVA
Arteries of the Brain
Major arteries involved in cerebral blood flow include:
Anterior Cerebral Artery
Middle Cerebral Artery
Posterior Cerebral Artery
Ophthalmic Artery
Internal Carotid Artery
Basilar Artery
Vertebral Artery
Arterial Blood Flow and Correlating Tissue
Understanding which arteries supply blood to specific areas of the brain is essential for evaluating CVA implications.
Anterior, Middle, Posterior, Basilar, Internal Carotid, Ophthalmic, and Vertebral arteries each play unique roles in cerebral perfusion.
Overview of Cerebral Vascular Accident (CVA)
Commonly referred to as a stroke.
Can be caused by two main mechanisms:
Ischemia (blockage): Limits or occludes blood flow to the brain leading to tissue damage.
Hemorrhage (bleed): Causes cell death and increases pressure within the skull.
Functional impairments and limitations depend on the location of the brain damage.
Ischemic Stroke
Defined as: Blood clot lodged in an artery supplying the brain.
Two types of ischemic strokes:
Embolic Stroke: Clot forms in the circulatory system and travels to brain.
Thrombotic Stroke: Clot forms directly within a brain artery.
Embolic Stroke
Process: A blood clot or plaque forms in the circulatory system and migrates to the brain.
Atherosclerosis: The buildup of fatty plaque in arteries that can lead to embolic strokes.
Etymology: "athero" = pasty material; "sclerosis" = abnormal hardening of tissue.
Thrombotic Stroke
Occurs when a blood clot forms within an artery supplying blood to the brain, disrupting blood flow and contributing to cell death.
Hemorrhagic Stroke
Occurs when blood vessels in the brain burst, spilling blood into the surrounding tissues.
Effects:
Increased pressure on nearby tissues leading to cell death.
Types of hemorrhagic strokes include:
Intracerebral Hemorrhage: Bleeding directly into brain tissue.
Subarachnoid Hemorrhage: Bleeding into the space between the brain and the skull.
Aneurysm: Weakened area causing arterial bulging and potential rupture.
Intracerebral Hemorrhage
Caused by the bursting of a blood vessel bleeding into brain tissue; often linked to hypertension.
Etymology: "Hyper" = high; "tension" = pressure in the vessels.
Subarachnoid Hemorrhage
A blood vessel bursts near the brain's surface, leading to leakage between the brain and skull.
Effects:
Causes arterial spasms that reduce blood flow, potentially leading to a stroke.
Aneurysm
Defined as a weak spot in an arterial wall that can swell, potentially rupturing and spilling blood into brain tissue.
Results in increased pressure and possible reduced perfusion, leading to cell death.
smokers are at a higher risk of developing aneurysms due to the damaging effects of nicotine and other harmful substances on blood vessels.
Transient Ischemic Attack (TIA)
Brief period during which blood flow to the brain is temporarily blocked or decreased.
Symptoms typically resolve within 24 hours and may include:
Numbness
Weakness
Loss of vision
Trouble speaking
Loss of balance or coordination.
Risk Factors for CVA
Common risk factors include:
High Blood Pressure
Diabetes
Smoking
Alcohol and Drug Use
Obesity
Diet
Coronary Artery Disease
Hyperlipidemia
Atrial Fibrillation
Peripheral Artery Disease
Age
Previous TIA
Genetics
Environmental factors such as air pollution.
Other conditions include:
Migraine
Sleep Apnea
Lupus
Sickle Cell Disease
Chronic Stress
Recognizing Stroke: FAST/BEFAST Acronym
Acronym used for quick identification and treatment of stroke:
Facial Drooping
Arm Weakness
Speech Difficulty
Time to Call for assistance.
Common Symptoms of CVA
Include:
Hemiplegia (complete paralysis on one side)
Hemiparesis (weakness on one side)
Aphasia (language impairment)
Ataxia (lack of coordination)
Dysphagia (difficulty swallowing)
Apraxia (difficulty with motor planning)
Paresthesia (burning or prickling sensation in the hands).
Aphasia and Cerebral Cortex Involvement
Two principal types of Aphasia:
Broca's Aphasia: Affects speech production.
Wernicke's Aphasia: Affects language comprehension.
Right Cerebral CVA
Effects observed include:
Left-sided neglect: Inattention causing the person to ignore left-sided stimuli.
Short-term memory challenges.
Decision-making difficulties and impulsive behavior.
Lack of insight into deficits and impaired spatial awareness.
Left Hemisphere of the Cerebral Cortex
Primarily affects speech and language in most individuals.
Symptoms may include the following:
Speech difficulties (aphasia).
Cautiousness and slowness in reactions.
Potential visual neglect on the right side.
Facial weakness and unclear speech; swallowing difficulties.
Impacts on logic and analytical capabilities.
Hemiplegic Gait
Describes the walking patterns seen in individuals with hemiplegia following a stroke.
Symptoms of a Stroke in the Brain Stem
Symptoms related to cranial nerve deficits may include:
Altered smell, taste, hearing, or vision (total or partial).
Eyelid drooping (ptosis).
Ocular muscle weakness.
Difficulty swallowing.
Altered breathing and heart rate.
Cerebellar Stroke
Symptoms include:
Altered walking (ataxic gait). Ataxic gait is defined as an unsteady, uncoordinated walk that results from damage to the cerebellum, affecting balance and posture.
Movement coordination issues.
Possible vertigo and balance difficulties.
Prognosis Following a Stroke
Stroke disables approximately 75% of survivors, affecting physical, mental, emotional, or a combination of these domains.
Prognosis varies widely based on:
Size and location of the lesion.
Type of dysfunction corresponding to areas of brain damage.
Additional Deficits/Signs/Symptoms Associated with CVA
Common consequences include:
Pressure sores
Pneumonia
Incontinence
Behavioral changes
Emotional problems may arise, either from direct damage to emotional centers or adaptation challenges, resulting in:
Anxiety/Panic Attacks
Flat Affect
Mania
Apathy
Psychosis
Prognosis of Post-Stroke Depression
Depression affects 30% to 50% of stroke survivors, potentially leading to:
Lethargy
Irritability
Sleep disturbances
Lowered self-esteem
Withdrawal from social interactions.
Rehabilitation Post-CVA
Core components of rehabilitation include:
Physical Therapy (PT)
Occupational Therapy (OT)
Speech-Language Pathology (SLP)
Assistive technologies may assist recovery (e.g., wheelchairs, walkers, canes).
PT focuses on improving joint range of motion, strength, and functional tasks (bed mobility, transferring, walking).
OT concentrates on activities of daily living (ADLs) such as bathing, dressing, eating, as well as instrumental activities (I-ADLs) like home management and community reintegration.
Collaboration between PT and OT aims to enhance awareness and use of the hemiplegic side.
Constraint-Induced Movement Therapy
A rehabilitation approach using repetitive practice to promote use and adaptation of the hemiplegic limb during functional tasks, fostering lasting changes.
Role of Speech and Language Pathology in Rehabilitation
SLP interventions target:
Speech production disorders (e.g., dysarthria, apraxia).
Aphasia and cognitive-communication impairments.
Dysphagia (swallowing difficulties) management.