Hallucinogens
Prevelance
hallucingens use at some point in their lives: 10% of US pop
current users around 0.2% or 485,000 people
wide veritery of substances
produce powerful changes primaraly in sensory perception (trips)
Trageting the serotinin receptors (primarily 5HT2 class)
Natral hallucigens
Lysergic acid diethelamide (LSD) dirrived from ergot alkaliods (fungus)
Psilocbin (mushrroms)
Salvia
mescalin
Sythetic hallugins
Foxy (5-MeO-DIPT)
DMT
Ecstity, molly, MDMA
2CB, 2CTI, 2CC2, ect
Danager both short and long
self injury, bad trips, flash backs, perminate and long term deficits
MDMA can lead to depletion of serotonin in the synapse
Broad mechainism
glutamate neurons in frontal cortex regulate VTA dopamine neurons
serotonin 2A receptors (5HT2A) activate these glutamate neurons and therefore increase dopamine in VTA
Too much dopamine going to Nac leads to hallucinations/psychosis
Activates 2 receptors and makes more dopamine in VTA
Other ways serotin affects dopamine and alter perception
serotin reaches down into neromodulators and effects how they interact with brain
Right brain/left brain
more callaboration between hemispheres