Cardiac Arrythmias (YT)
Heart Rhythms and Origin
Rhythm classifications based on their site of origin:
Sinus rhythms: Originate from the sinoatrial (SA) node.
Atrial rhythms: Originate from the atria.
Ventricular rhythms: Originate from the ventricles.
Sinus rhythm: Normal heart rhythm controlled by the SA node.
The Sinoatrial Node
The SA node fires 60 to 100 times per minute, establishing a normal heart rate of 60 to 100 beats per minute.
Common variations include:
Sinus bradycardia: Means the SA node fires less than 60 times, resulting in a heart rate of less than 60 bpm; can be normal during sleep.
Sinus tachycardia: Means the SA node fires more than 100 times, resulting in a heart rate of over 100 bpm; can be normal during exercise.
Both sinus bradycardia and sinus tachycardia could be normal or clinical based on underlying causes.
Cardiac Arrhythmias from the Atria
Cardiac arrhythmias from other parts of the atria are always clinical conditions.
Common atrial arrhythmias include:
Atrial flutter: Caused by a reentrant electrical impulse loop usually in the right atrium.
Regular and rapid atrial rate of 250-400 beats per minute.
Ventricular rate is slower, typically a three-to-one heart block (one impulse in three reaches the ventricles).
On ECG, characterized by absence of normal P waves and presence of flutter waves (F waves) in sawtooth patterns.
Atrial fibrillation: Involves multiple chaotic impulses initiated from ectopic sites in the atria.
Causes quivering instead of contraction of the atria.
Atrial rate can be high, but most impulses don’t pass through the AV node, leading to an irregular ventricular rate (can range from <60 to >100 bpm).
On ECG, absence of P waves and irregular narrow QRS complexes, with a baseline that may appear undulating or flat.
AV nodal reentrant tachycardia (AVNRT): Small reentrant pathway involving the AV node.
Atrial and ventricular rates are identical, fast and regular, ranging from 150 to 250 beats per minute.
Ventricular Rhythms
Ventricular rhythms are dangerous, termed lethal rhythms.
Ventricular tachycardia (V tach): Results from a single strong firing site within the ventricles.
Common in individuals with heart structural issues (e.g., scarring from a heart attack).
Produces ventricular premature beats, regular and fast (100-250 bpm);
On ECG: wide, bizarre QRS complexes with absent P waves.
Can occur in brief episodes (<30 seconds); sustained VTAC (>30 seconds) requires immediate treatment to avoid cardiac arrest.
Potential progression to ventricular fibrillation.
Ventricular fibrillation (V fib): Caused by multiple weak ectopic sites in the ventricles.
Results in chaotic electrical signals causing the ventricles to quiver instead of contract, leading to minimal or no blood pumped.
It can rapidly result in cardiac arrest.
On ECG: irregular random waveforms without identifiable P wave, QRS complex, or T wave; amplitude decreases over time, evolving from coarse to fine V fib to eventual flatline.