CH06 Cocaine and other stim
Drug Use and Misuse
Author and Edition: Ninth Edition by Stephen A. Maisto, Mark Galizio, Gerard J. Connors.
Chapter Overview: Focus on Cocaine, Amphetamines, and Related Stimulants.
Approach to Discussing Drugs/Drug Classes
History, Prevalence, etc.
Pharmacology
Effects
Controlled Stimulants
Table 6.1 - Controlled Stimulants
Cocaine: No brand name; slang terms include angel powder, diosa blanca.
Dextroamphetamine: Brand names Adderall, Vyvanse; slang includes Addy, speed.
Methamphetamine: Brand name Desoxyn; slang terms chicken feed, speed.
Methylphenidate: Brand names Ritalin, Concerta; slang kiddie coke, Vitamin R.
Methcathinone: No brand name; slang cat.
Mephedrone: No brand name; slang bath salts, bubbles, meow-meow.
The Coca Leaf
Brief History of the Coca Leaf
Indigenous Usage: Chewed by indigenous peoples of Bolivia, Ecuador, Argentina, and Peru for thousands of years.
Cultural Significance: Important in Incan culture; used for medicinal and stimulant purposes.
European Awareness: First noticed in the 1800s with the isolation of cocaine in 1850.
Methods of Absorption: IV injection or smoking provides rapid absorption compared to chewing leaves.
Early Use of Cocaine
19th-Century Introduction
Freud's Advocacy: Sigmund Freud promoted cocaine for treating various medical issues.
Surgical Use: First local anesthetic used in surgeries; declined by end of 19th century due to abuse.
Regulation: Harrison Narcotics Act of 1914 established control over cocaine and similar substances.
The Amphetamines
History and Use
Transition from Cocaine: Shift to amphetamines after the Harrison Narcotics Act; synthesized in the late 19th century.
Medical Applications: Initially used for colds, obesity, narcolepsy, and ADHD by the 1920s.
Military Use: Widely given to WWII soldiers for performance enhancement.
Post-War Issues: Became problematic due to misuse and dependency.
Injection Effects of Amphetamines
Immediate Effects: Intense "flash" after injection; repeated use results in stimulation without the initial "rush."
Side Effects: Appetite suppression, formication, paranoid delusions, and stimulant psychosis with overdose.
Withdrawal Symptoms: Commonly include depression post-heavy use.
Cocaine Epidemic II
Reemergence of Cocaine
Glamorization: In the 1970s, cocaine became more accessible and glamorized.
Crack Cocaine: Emerged in the late 1970s; produced intense but brief highs.
Market Growth: Developed rapidly due to affordability and intense cravings.
Development of Crack Cocaine Use and Problems
Crisis Reports: By late 1980s, crack use increased dramatically; rise in overdose deaths.
Statistics: Survey data indicating fluctuations in crack and powdered cocaine use among high school seniors from 1986 to 2016.
Cocaine Drug Wars
Colombian Cartel Control: Historical monopolization of cocaine distribution by Colombian cartels.
Government Action: U.S. and Colombian governments attempted to restrict smuggling; led to violence.
Production Shift: Decline in Colombian production offset by increases in Peru and Bolivia.
The Coca Leaf in Latin America
Cultural Continuance: Coca cultivation continues in Andean countries; government support.
Evo Morales: Bolivian President's symbolic consumption of coca at UN to represent traditional use.
Legislation Against Crack Cocaine
Anti-Drug Acts: Enacted severe penalties for crack cocaine at disproportionate rates.
Racial Disparities: Laws affected African American communities significantly; measures taken to address inequities.
The Return of Meth
Methamphetamine Overview
Historical Use: Meth reappeared in the U.S. in the early 1990s; largely spread via illegal labs.
Demographic Usage: Evolved from a "biker drug" to mainstream usage in club settings.
Public Health Risks: Risks associated with illegal labs include explosions and biohazards.
Bath Salts: New Synthetic Stimulants
Existence and Risks: Rarely seen before 2009; linked to acute psychosis and overdose deaths.
Chemical Properties: Classified as synthetic analogs of cathinone, which mimics stimulants found in the khat plant.
Mechanism of Stimulant Action
How Stimulants Affect the Brain
Neurotransmitter Interaction: Primarily affect monoamines (dopamine, norepinephrine, serotonin).
Action Mechanism: Block reuptake or increase release of these neurotransmitters.
Pharmacokinetics of Stimulants
Administration Routes and Effects
Onset and Duration: Varies significantly between oral and IV use; inhalation provides rapid effects.
Metabolism: Cocaine metabolizes quickly while amphetamines have longer action durations.
Acute Effects at Low and Moderate Doses
Physiological Effects
Sympathomimetic Effects: Increase heart rate, respiration, and blood pressure.
Appetite Suppression: Used for weight loss but often leads to weight regain after cessation.
Behavioral Effects
Mood Enhancements: Increased sociability, alertness, and performance in physical tasks.
Learning Challenges: State-dependent learning can impede recall without drug influence; potential increase in errors.
Acute Effects at High Doses
Risks Associated with High Doses
Stimulant Psychosis: Can cause paranoia and compulsive behaviors; treatable with antipsychotics.
Overdose Risks: Severe health consequences including respiratory collapse and heart attacks.
Effects of Chronic Use
Tolerance Development
Types of Tolerance: Acute versus chronic tolerance, with variances in heart rate and blood pressure responses.
Dependence and Withdrawal
Withdrawal Symptoms: Primary symptoms are psychological (depression, agitation, sleep disturbances).
Craving Phases: Initial crash followed by extinction, linked to environmental cues.