Sinus Rhythms
Sinus Rhythms
Sinus Rhythm
- All originate in the sinoatrial (SA) node.
- ECG features common to all sinus rhythms:
- Upright P waves, all similar in appearance.
- Normal duration PR intervals ( to seconds).
- Normal duration QRS complexes ( – sec) if no ventricular conduction disturbances are present.
EKG Characteristics
- The SA node discharges and follows the normal conduction pathway
- P Waves
- Uniform in appearance, positive (upright) in lead II, one and only one precedes each QRS complex
- PR Interval
- to seconds and constant from cardiac cycle to cardiac cycle
- QRS complex
- – sec
- Rhythm
- Regular
- Rate
- 60 to 100 beats/min
Sinus Bradycardia
- The SA node discharges slower than in NSR but follows the normal conduction pathway.
EKG Characteristics
- P Waves
- Uniform in appearance, positive (upright) in lead II, one and only one precedes each QRS complex.
- PR Interval
- to seconds and constant from cardiac cycle to cardiac cycle
- QRS Complex
- – sec
- Rhythm
- Regular
- Rate
- Less than 60 beats/min
Monitoring the Patient
- Sinus bradycardia can be normal in athletes and during sleep, or in response to a vagal maneuver.
- Determine if the patient is symptomatic or not.
Causes of Sinus Bradycardia
- Vagal stimulation
- Drugs like beta blockers/CCB/Morphine/Anticholinesterase
- Ischemia
- Increased Intracranial Pressure (ICP)
- Athlete (normal variant)
Hemodynamic Responses
- Sinus bradycardia produces various hemodynamic responses.
Signs and Symptoms of Sinus Bradycardia
- Hypotension
- Pale, cool skin
- Weakness
- Angina
- Dizziness or syncope
- Confusion or disorientation
- Shortness of breath
Interventions for Sinus Bradycardia
- Nursing Interventions
- Monitor vital signs (including SpO2).
- Administer oxygen if hypoxic.
- Trendelenburg position (¯ HOB) for hypotension.
- Assess for angina.
- Assess for shock (cool clammy skin, ALOC - altered level of consciousness).
- Establish IV access.
- Identify and treat possible causes (hold negative chronotropic medications).
- Pharmacologic Interventions
- If symptomatic (chest pain, shortness of breath, altered level of consciousness):
- Atropine (initial dose is mg IVP q3 – 5 minutes for a max of mg); Isuprel
- Stop offending drugs
- If symptomatic (chest pain, shortness of breath, altered level of consciousness):
- Electrical Interventions
- Artificial pacing if patient is hemodynamically compromised
- Transcutaneous / temporary pacing
- Artificial pacing if patient is hemodynamically compromised
Sinus Tachycardia
- SA node discharges more frequently than in NSR but follows the normal conduction pathway.
EKG Characteristics
- P Waves
- Uniform in appearance, positive (upright) in lead II, one and only one precedes each QRS complex; at very fast rates
- PR Interval
- to second and constant from cardiac cycle to cardiac cycle
- QRS Complex
- – sec
- Rhythm
- Regular
- Rate
- to beats/min
Underlying Causes
- The clinical significance of this dysrhythmia depends on the underlying cause
- Caused by vagal inhibition or sympathetic stimulation
- Increased Circulating Catecholamines
- Associated with physiologic (increased temperature) and psychologic stressors (anxiety)
- Stimulants, exercise, fever, hypoxemia/hypoxia, & alterations in fluid status (hypovolemia)
- Drugs can increase rate
- Assess for symptoms of low cardiac output (cardiac failure/HF)
- PE/hyperthyroidism
- Response to pain
Manifestations
- Dizziness
- Dyspnea
- Hypotension
- Angina in patients with CAD
Interventions
- Nursing Interventions
- Vital Signs (including SpO2)
- Oxygen if hypoxic
- Trendelenburg position (¯ HOB) for hypotension
- Assess for angina
- Assess for shock (cool clammy skin, ALOC)
- IV Access
- Identify and treat possible causes (Pain? Fever? Anxiety?)
- Vagal Maneuvers (if rate > 160)
- Pharmacologic
- Beta Blockers; Calcium Channel Blockers
- Electrical
- Usually, NA for rates of 160 or less
Treatment
- Guided by cause (e.g., treat pain)
- Vagal maneuver
- β-blockers
Sinus Arrhythmia
- Originates in SA node but the impulses are discharged irregularly.
EKG Characteristics
- P Waves
- Uniform in appearance, positive (upright) in lead II, one and only one precedes each QRS complex
- PR Interval
- to second and constant from cardiac cycle to cardiac cycle
- QRS:
- Narrow (<0.12 sec); sometimes wide
- Rhythm
- Irregular
- Rate
- 60 to 100 beats/min; frequently increases with inspiration & decreases with expiration; sometimes slower
Sinus Pause/Sinus Arrest
- SA node fails to discharges and then resumes when the SA node resets itself or when a lower pacemaker begins to discharge.
- The pause (arrest) time interval is not a multiple of the normal R-R interval
EKG Characteristics
- P Waves
- normal in appearance, positive (upright & uniform), except in areas of pause (arrest)
- PR Interval
- to second
- QRS Complex
- – sec
- Rhythm
- Irregular whenever a pause (arrest) occurs
- Rate
- Normal to slow; determined by duration & frequency of sinus pause (arrest)
Sinus/Sinoatrial Block
- SA block occurs in some multiple of the R-R interval; after the dropped beat, cycles continue on time
EKG Characteristics
- P Waves
- normal in appearance, positive (upright & uniform), except in areas of dropped beats
- PR Interval
- to second
- QRS Complex
- – sec
- Rhythm
- Irregular whenever an SA block occurs
- Rate
- Normal to slow; determined by duration & frequency of SA block
Sinus Arrest or Sinus Block
- Sinus node fails to initiate impulse
- Causes: vagal responses, heart disease, and drugs that slow heart rate
- Heart rate can be normal or slow
- Irregular Rhythm
- Can decrease cardiac output causing syncope or dizziness
Common Sinus Rhythms
- Normal Sinus Rhythm (NSR)/SR
- Sinus Bradycardia
- Sinus Arrhythmia
- Sinus Tachycardia
- Sinus Arrest/Pause
- Sinus Block