Cardiovascular Medications Study Notes
Cardiovascular Medications Overview
- Cardiovascular Medications: Drugs that affect cardiovascular function.
- Types:
- Inotropic drugs
- Antiarrhythmic drugs
- Antianginal drugs
- Antihypertensive drugs
- Diuretics
- Antilipemic drugs
Inotropic Agents
- Definition & Function:
- Agents that alter myocardial contractility (the force of heart contractions).
- Can have positive (+) or negative (-) effects.
- Primary Classes:
- Digitalis (e.g., Digoxin)
- Phosphodiesterase Inhibitors (PDE Inhibitors, e.g., Milrinone)
Digitalis (e.g., Digoxin)
- Mechanism of Action:
- Increases intracellular calcium, leading to more forceful contractions.
- Acts on CNS to slow heart rate (HR).
- Indications:
- Treatment of Atrial Fibrillation with Rapid Ventricular Response (A-Fib w/ RVR)
- Atrial Flutter, Supraventricular Tachycardia (SVT), Paroxysmal Atrial Tachycardia (PAT).
- Increases refractory period, reducing heart rate.
Interactions
- Numerous drug interactions that can affect efficacy or increase toxicity:
- Drugs reducing therapeutic effects
- Drugs increasing dysrhythmias
- Herbal Supplements: St. John’s Wort can interfere with Digoxin efficacy.
Adverse Effects of Digoxin
- Signs of toxicity:
- Nausea, vomiting, abdominal pain, diarrhea
- CNS symptoms: headache, irritability, depression, insomnia, confusion
- Visual disturbances: blurred/yellow vision
- Cardiac issues: bradycardia, complete heart block
Nursing Process Considerations for Digitalis
- Assessment:
- Monitor vital signs, potassium (K) levels, digoxin levels, signs/symptoms (S/Sx) of toxicity.
- Diagnosis:
- Problems: Decreased cardiac output (CO), risk for injury.
- Planning:
- Set goals based on diagnosis
- Implementation:
- Administer as per protocol, monitor closely for adverse effects.
- Evaluation:
- Assess if goals of improved cardiac output and negative adverse reactions are met.
Phosphodiesterase Inhibitors (PDE)
- Examples: Inamrinone, Milrinone
- Uses:
- Short-term treatment of Congestive Heart Failure (CHF).
- Administered intravenously.
- Mechanism of Action:
- Increases calcium influx into cells and promotes smooth muscle relaxation, reducing preload and afterload.
Adverse Effects of PDE Inhibitors
- Potentiate risk of hypotension, dysrhythmias, and cause nausea.
- Must monitor potassium levels closely, especially with K-wasting diuretics.
Antidysrhythmic Medications
- Purpose: Treat arrhythmias; can worsen existing rhythms.
- Classes:
- Class I: Sodium (Na+) channel blockers
- Class IA: Quinidine, Procainamide
- Class IB: Lidocaine, Phenytoin
- Class IC: Flecainide, Propafenone
- Class II: Beta Blockers (e.g., Propranolol, Metoprolol)
- Class III: Potassium (K+) channel blockers (e.g., Amiodarone)
- Class IV: Calcium (Ca2+) channel blockers (e.g., Verapamil, Diltiazem)
Class IA Antidysrhythmics — Details
- Mechanism of Action: Reduce phase 0 depolarization by blocking Na+ channels, slowing action potential.
- Indications: Treat various arrhythmias such as:
- Atrial Fibrillation
- Ventricular Tachycardia
- Key Considerations:
- Monitor ECG for changes.
- Watch for potential drug interactions, especially with digoxin and warfarin.
Class IV Antidysrhythmics — Details
- Function: Inhibit Ca2+ influx; can act as pseudo + inotropes.
- Adverse Effects: Postural hypotension, bradycardia, AV block.
Antianginal Drugs
- Subtypes:
- Nitrates (e.g., Nitroglycerin)
- Routes of Administration: Sublingual, oral, IV, transdermal.
- Patient Education:
- Check and store medications properly.
- Notify healthcare provider of severe or persistent symptoms.
Antihypertensive Drugs
- Classes:
- ACE Inhibitors (e.g., Lisinopril)
- Angiotensin II Receptor Blockers (ARBs)
- Calcium Channel Blockers
- Beta Blockers
- Considerations:
- Monitor vital signs, renal function, and electrolytes.
- Educate patients on potential side effects and lifestyle modifications to manage blood pressure.
Common Adverse Effects of Antihypertensives
- Dizziness, hypotension, dry cough (ACE inhibitors), hyperkalemia, and fatigue.
Antilipemic Agents
- Include: Statins, Niacin, Bile-acid sequestrants.
- Functions: Lower LDL cholesterol, increase HDL cholesterol, and manage triglyceride levels.
- Clinical Considerations: Monitor lipid profiles and educate about dietary management.
Statins - Key Points
- Mechanism: Inhibit cholesterol production.
- Adverse Effects: Muscle pain, liver dysfunction, rhabdomyolysis.
- Nursing Points:
- Monitor CK levels, liver function tests, and educate regarding potential side effects and interactions.