Lecture 9 2020-2021
Control Points in Gene Expression
Various control points are crucial for regulating gene expression.
Post-transcriptional control occurs after mRNA is synthesized in the nucleus.
Key areas of control include:
mRNA degradation
Inactive mRNA transport
mRNA processing
Protein translation
Protein activity and localization control.
Alternative mRNA Splicing: The Concept
Alternative splicing is a process allowing the production of multiple mRNA variants from a single gene.
Exons: coding sequences that remain in the mRNA post-splicing.
Introns: non-coding sequences that are removed during splicing.
Splice variants: different mRNAs produced from various combinations of exons.
Mechanisms of Alternative Splicing
Cells exploit alternative splicing to generate diversity in protein outputs from one gene.
Key types of alternative splicing include:
Intron retention
Mutually exclusive exons
Alternative splice sites (5' & 3')
Exon inclusion/skipping
Tissue-specific regulation
Prevalence in Eukaryotes
Alternative splicing is common, with over 90% of genes in mammalian cells subject to splicing variations.
It impacts gene product diversity and mRNA properties (e.g., stability).
Alternative Splicing in Disease: Apoptosis
Splicing of apoptosis-regulated genes determines cell survival:
Anti-apoptotic vs Pro-apoptotic isoforms influence whether a cell undergoes apoptosis.
Example: Drosophila Sex Determination
Mechanisms of sex determination via alternative splicing:
The sex lethal (Sxl) gene plays a pivotal role in females (X:A ratio).
Males splice transformer (tra) gene to create a dysfunctional protein, while females produce a functional Tra protein.
Activation of DSX transcription factor occurs differently in males and females due to the presence of Tra.
Example of Human Disease: Myotonic Dystrophy
Myotonic Dystrophy is linked to splicing defects:
Dominantly inherited muscular dystrophy affecting muscle function.
Caused by CTG repeat expansions in the DMPK gene.
The expanded RNA sequesters critical splicing regulators, leading to specific disease symptoms.
Mdm2 Splicing in Cancer
In cancer, Mdm2 gene undergoes alternative splicing, producing variants that may contribute to tumor diversity and progression.
It regulates p53, a crucial protein in protecting against DNA damage.
Regulatory Elements of Splicing
Alternative splicing can be influenced by enhancers and silencers:
Specific RNA sequences can promote or inhibit splicing site choices.
Exonic and Intronic Splicing Enhancers (ESE/ISE) and Splicing Silencers (ESS/ISS) regulate splicing efficiency and choice.
Fate of RNA Binding Proteins
The Fox family of RNA binding proteins regulate alternative splicing in specific tissues, promoting the evolution of complex organisms.
They recognize specific motifs vital for splicing regulation in metazoans.
Trans-Splicing Reactions
Trans-splicing involves joint processing of exons from different pre-mRNA molecules; rare in multicellular organisms but prevalent in some organisms like trypanosomes.
Short Sequence Leader RNAs (SL RNA) play roles in this process.
Summary of Regulated Alternative mRNA Splicing
Alternative splicing significantly enhances genomic coding potential.
Generates diverse mRNA forms, including tissue and developmental specificity.
Particularly critical in non-dividing cells, particularly in neuronal and muscle cells.
mRNA Processing in Eukaryotes
Three stages of RNA processing:
5' capping
RNA splicing: removal of introns and linkage of exons.
3' polyadenylation for mRNA stability.