Chapter 14
Innate Defenses
our internal bodies must strive to maintain a sterile environment in a world full of microbes
first-line defenses
cells of the immune system
cell communication
Sensor Systems
phagocytosis
Inflammation
Fever
Innate immunity - host defenses (anatomical barriers, sensor systems, and phagocytic cells)
Pattern recognition - what sensor systems are used to detect certain microbes
LPS and peptidoglycan
molecules that envoke immune response are called antigens
Adaptive immunity - immunity that comes from exposure to microbes and is a specialized defense system of vertebrates
First Line Defenses
Barriers make it difficult for microbes to get to our insides
Barriers are in direct contact with the outside environment
Anatomical barriers - skin and mucous membranes
provide a physical barrier and have innate antimicrobial properties such as antimicrobial secretions and benefits from normal flora
Physical Barriers
all exposed surfaces are lined with epithelial cells tightly packed resting on a thin fibrous basement membrane
Skin
most difficult to penetrate - two layers
Dermis - tightly woven fibrous connective tissue
Epidermis - layers of epithelial cells becoming progressively flattened toward the exterior
outermost layers consist of dead cells with water
repelling Keratin protein creating an arid environment
Dead cells come off and take microbes with them
mucous membranes
Digestive, respiratory and genitourinary tract
Constant mucous or other secretions to wash away microbes
some can use cilia (respiratory tract) or peristalsis (digestive tract) to propel microbes toward areas of easy elimination or in the case of the genitourinary tract the flow of urine
Antimicrobial Substances
substances on skin and mucous membranes that inhibit or kill microbes
Sweat - High Salt
Lysozyme - degrades peptidoglycan in tears, saliva, mucus
also inside phagocytic cells, blood, and fluid surrounding tissues
peroxidase enzymes - Saliva and milk. Break down hydrogen peroxide producing harsh oxidizing compounds
catalase-producing bacteria less susceptible
Lactoferrin - Saliva, mucus, Mild (Transfirin in blood and tissue fluids.)
Iron-binding protein effectively sequesters iron thereby withholding it from Microbes
Defensin - Short antimicrobial Peptides produced by neutrophils and epithelial cells. insert into bacterial membrane-making pores that disrupt cell integrity
Normal Microbiota/ Flora
competitive exclusion of pathogens
covering attachment Sites
consuming available nutrients
Toxic compound production
Disruption of normal microbiota can predispose a person to infections
essential for immune system development in infants
Cells of the Immune System
Travel through the body's circulatory system moving from one part of the body to another
some of these cells are in both innate and adaptive immune response
Always found in blood, numbers increase during infection
reserves found in bone Marrow. All blood cells come from hematopoietic Stem cell
Three General Categories
Red blood cells - erythrocytes, carry oxygen
platelets - megakaryocytes, clotting
white blood cells - leukocytes: hosts defenses
Cytokines - colony-stimulating factors cause stem cells to develop into different kinds of blood cans. Some cars mature and others differentiate as they enter tissues
Polymorphonuclear Leukocytes - PMN
Granulocytes
cytoplasmic granules containing biologically active chemicals
neutrophils
phagocytize (engulf) and destroy foreign material.
Granules contain antimicrobial substances and degrative enzymes
most abundant
Basophils
Allergic reactions and inflammation.
Granules contain histamine and other chemicals that increase capillary permeability
similar to mast ceil but found in blood
Eosinophils
Thought to function in expelling parasitic worms
involved in allergic reactions - causing and alleviating symptoms
Granules contain antimicrobial substances and histaminase
Leukocytes
Mononuclear Phagocytes - MPS
monocytes circulate in the blood for a few days then enter tissues and differentiate into macrophages or dendritic cells
Macrophages
differentiated form of monocyte
phagocytic cells in almost all tissues but abundant in the liver, spleen lymph nodes, lungs, abdominal cavity
names change based on tissue
survives for months
Dendritic Cells
usually differentiated from monocyte
function as scouts in various tissues
engulf material in tissues and bring it to adaptive immune cells for inspection
lymphocytes
responsible for adaptive immunity
Two major groups
B cells and T cans
great septicity in antigen recognition
Generally resid in lymph nodes
Natural killer- not specific
Cell Communication
Immune System Cells need to communicate with each other and their environment to Successfully combat invasion or trauma
Surface Receptors
proteins that generally span the membrane
eyes and ears
enable inside of the cell to sense and respond to what is outside the cell
receptors are specific to a certain ligand compound
when a ligand binds, the receptor potion becomes modified in some ways, causing a cell response
cells can change the winds and amounts of receptors they make to adjust conditions
Cytokines
protiens made for the purpose of communication.
voices
Bind Cytokine receptors on the surface of the internal cans that the cytokines regulate
can cause changes in cell growth, differentiation, movement or cause cell death
Doesn't last long, take many to produce large effect.
can act locally or systemically
often act together or in a sequence cascade
pro-inflammatory cytokines CTNF-Alpha, IL-1, IL-6 and others) contribute to inflamation
often involved in antibody response
Adhesion Molecules
one cell surface and allow cen to adhere to other cells or to 'gras certain cells as they pass by
when tissues need phagocytic cells, the cells that line the blood vessels synthesize adhesions that adhere to passing phagocytic cells
other adhesion molecules allow cells to create direct contact whereby cytokines or other molecules can be delivered to a particular cell
Sensor systems
complement system
series of proteins always circulating in the blood and tissue fluid that work in conjunction with "complement the adaptive immune response.
circulate in their inactive form, activation sets of a cascade of events that cooperate with host defense systems to remove and destroy invading microbes
Activation of one complement proton allows it to become enzymatically active, Cleaving and activating millions of the next protein in the cascade. Each of those activates multiple Molecules of the next proteion in the cascade
stringent controls at various points
three pathways lead to a complement system
Alternative Pathway
Lectin pathway
Classical pathway
Complement System - 3 Outcomes
Inflammation
compliment components C3a and C5a induce changes in epithelial cells that line blood vessels leading to vascular permeability associated with inflammation
C5 is a chemoattractant living phagocyte in the area
Lysis of Foreign Cells
complexes of (Sb, 16, CG molecules spontaneously assemble in the membrane of foreign ceils producing structures called Membrin attack complex (mac) creating pores in the membrane and interfering with the integrity of the cell
Gram + cells peptidoglycan kyer makes them less susceptible but Gram-bacteria outer layer is sensitive
Opsonization
C3b binds foreign material making them sensitive to phagocyte ingestion because they have C3b receptors on their surface
C3b coated material is opsonized - prepared for eating
C3b is an opsonin
Inflammation
Inflammatory response triggered when tissues have been damaged
skin penetrated, invasion
Cardinal signs of inflammation: swelling, redness, heat, pain, loss of fiction
The purpose is to contain the site of damage, localize response, eliminate invaders, and restore tissue function
Macrophages and eosinophils at the Site of damage, neutrophils, and monocytes recruited
Acute Infection - short-term inflammation, characterized by neutrophils
chronic inflammation - long-term inflammation, occurs when acute inflammation is insufficient to limit infection, characterized by macrophages, giant cells, and granulomas
Inflammatory processes
Dilation of small blood vessels
greater blood flow, slower rate
leakage of fluids
migration of leukocytes from the bloodstream to tissues
endothelial cells "grab" phagocytes, slow them down
phagocytes squeeze between cells of vessel - diapedesis
Clotting factors wall off-site of infection
Dead neutrophils, tissue debris accumulate as pus
Outcomes of Inflammation
can help prevent a problem but causes damage doing it
Toxic substances from dead phagocytes can be released
damage inflicted by these effects vary in severity based on length of inflammation and where inflammation occurs
Fever
Important host defense of vertebrates
Temperature regulated by the brain
during infection, the regulator changes its setting achieving higher temperatures than normal
pro-inflammatory cytokines released by macrophages signal to the brain
Fever-inducing substances are called pyrogens
endogenous pyrogens - produced by the body-cytokines
exogenous pyrogens - produced externally by microbial products
fever inhibits the growth of many pathogens by
exceeding microbes’ temperature range for growth
Activating and Speeding up other host defenses
Sensors that Detect Long dsRNA
most cans do not contain ds RNA greater than 30bp
usually a sign of Viral infection
RNA viruses, retroviruses, DNA viruses
Long ds RNA induce alpha-beta interferon synthesis and secretion.
Attach to receptors on infected cells and their neighbors using the expression of several inactive antiviral protiens
iAVPs are activated by dsRNA and become AVP which degrades RNA, stop protien synthesis, leads to apoptosis
only infected cells are sacrificed
Phagocytosis
Cells that engulf and digest material
Pattern recognition receptors on phagocytes determine which particles to engulf
Macrophages handle Butine situations with a few bacteria, is too many - produce cytokines to attract phagocytes
Process of Phagocytosis
medically important because bacteria often evolve to evade one of them
Chemotaxis
chemoattractants recruit phagocytes to the Site of tissue damage or infection.
Chemical stimuli include products of microorganisms, complement component CSa, and phospholipids released by damaged host cells
Recognition and Attachment
phagocytic cells have receptors to bind to invading microbes
Direct binding occurs through pattern recognition receptors identifying microbe compounds
bind to mannose
Indirect binding occurs when the particle is first opsonized which increases a phagocytic ability to attach and engulf
bind to opsonins
Engulfment
phagocytotic cell engulfs creating a membrane-bound vacuole called a phagosome - which does not destroy
pseudopods form around material
Fusion of Phagosome with Lysosome
phagosome transported along the Cytoskeleton to fuse with the lysosome
lysosome filled with digestive enzymes
phagolysosome
Destruction of Pigestion
Involves aerobic respiration of sugars creating oxygen diproducts.
ph decreases, enzymes degrade, detentions damage membrane of invader, lactoferrin ties up iron
Exocytosis
membrane-bound vesicle l fuses with can membrane dumping contents out of a cell
Attributes of Macrophages
scavengers of dead cells and debris
function as a first response
call for reinforcements
can live for weeks or months
alwas present in tissues
TLRs on their surface allow them to detect microbes, then produce pro-inflammatory cytokines to stimulate other cans of the immune system
gain assistance from certain T cells
activate macrophages
upon chronic infection, macrophages form giant cells
form granulomas with t cells
quarantine organisms
Attributes of Neutrophils
Critical during the early stages of inflammation, first cell type recruited from the bloodstream to of damage
only live 1-2 days in tissues but have more destructive power than macrophages
kill microbes through phagocytosis and can lay traps
can reless the content of their granules and DNA
DNA Shares microbe a and granule contents digest them