CARDIO
Cardiovascular System Overview
The cardiovascular system consists of the heart and blood vessels, responsible for circulating blood and facilitating gas exchange.
Anatomy of the Heart
Location: The heart is situated in the mediastinum.
Structure and Layers of the Heart
Pericardium: A double-walled sac containing the heart; consists of:
Parietal Layer: Outer layer
Visceral Layer (Epicardium): Covers the heart
Pericardial Cavity: Contains serous fluid to reduce friction during heartbeats.
Heart Wall: Composed of three layers:
Epicardium: Outer layer
Myocardium: Middle muscular layer (thickest)
Endocardium: Inner lining of the heart chambers.
Chambers of the Heart
Four Chambers:
Atria (2 superior chambers)
Ventricles (2 inferior chambers)
Septum: Divides the heart into right and left sides.
Interatrial Septum: Separates the atria
Interventricular Septum: Separates the ventricles.
Blood Vessels Related to the Heart
Right Atrium receives blood from:
Superior Vena Cava: Drains blood from the upper body
Inferior Vena Cava: Drains blood from the lower body
Coronary Sinus: Drains blood from the heart.
Left Atrium receives blood from:
Pulmonary Veins: Carry oxygenated blood from the lungs.
Right Ventricle pumps blood to:
Pulmonary Artery: Carries deoxygenated blood to the lungs.
Left Ventricle pumps blood to:
Aorta: Distributes oxygenated blood to the body.
Heart Valves
Tricuspid Valve: Between right atrium and right ventricle.
Pulmonary Valve: Between right ventricle and pulmonary artery.
Bicuspid (Mitral) Valve: Between left atrium and left ventricle.
Aortic Valve: Between left ventricle and aorta.
Function: Prevent backflow of blood during heart contraction.
Cardiac Muscle Tissue
Structure: Composed of branching cells connected by intercalated discs which include:
Desmosomes: Provide structural support.
Gap Junctions: Allow electrical impulses to pass between cells, facilitating rapid contraction.
Cardiac Conduction System
Components: SA Node, AV Node, Bundle of His, Purkinje Fibers.
Function: Facilitates pacemaker activity and coordinates heart contractions.
SA Node: Pacemaker of the heart, generates impulses at a rate of 70-100 beats/min.
Action Potential in Cardiac Muscle
Phases:
Phase 0: Rapid depolarization via Na+ influx.
Phase 1: Initial repolarization.
Phase 2: Plateau phase due to Ca2+ influx.
Phase 3: Repolarization via K+ efflux.
Phase 4: Resting membrane potential.
ECG (Electrocardiogram)
Records electrical activity of the heart:
P Wave: Atrial depolarization.
QRS Complex: Ventricular depolarization.
T Wave: Ventricular repolarization.
Cardiac Cycle
Definition: Series of events during one heart beat including systole (contraction) and diastole (relaxation).
Events During Systole:
Isovolumic Contraction: Ventricles contract, no volume change.
Ejection Phase: Blood is expelled into arteries.
Events During Diastole:
Isovolumic Relaxation: ventricles relax; pressure drops.
Rapid Inflow: Blood fills ventricles via AV valves.
Cardiac Output
Formula: CO = Stroke Volume x Heart Rate.
Typical Values: 70 mL/beat (SV) and 75 beats/min (HR) gives an average CO of 5.25 L/min.
Regulation of Heart Function
Stroke Volume Factors:
Preload: Degree of stretch before contraction.
Contractility: Force of contraction.
Afterload: Resistance the ventricle must overcome to eject blood.
Heart Rate Factors:
Affected by autonomic nervous system and hormones (epinephrine increases HR).
Blood Vessel Anatomy and Functions
Types: Elastic arteries, muscular arteries, arterioles, capillaries, venules, and veins.
Functionality: Vessels conduct, distribute, and exchange blood with tissues.
Blood Pressure and Resistance
Definition: BP is the force exerted by circulating blood against blood vessel walls.
Factors Affecting BP: Vessel lumen size, blood viscosity, and total vessel length.
Shock Types
Hypovolemic Shock: Due to reduced blood volume.
Cardiogenic Shock: Due to heart failure.
Vascular Shock: Due to inappropriate vasodilation.
Obstructive Shock: Due to physical blockage of blood flow.
Homeostatic Responses to Shock
Renin–angiotensin system responds to reduced BP to restore blood volume and pressure.
Activation of sympathetic response and release of local vasodilators helps adjust blood flow during stress.
Summary of Important Hormones
Epinephrine/Norepinephrine: Increases heart rate and force of contraction.
ADH: Increases blood volume by promoting water reabsorption.
ANP: Decreases blood volume and pressure.
Inotropic Agents: Affect contractility positively or negatively.
Important Concepts:
Frank-Starling Law: The more the heart is stretched (higher EDV), the stronger the contraction.