Renal Disorders Part2
Medical Management of Acute Kidney Injury (AKI) and Chronic Kidney Disease (CKD)
Underlying Causes
Identifying and correcting the underlying cause of AKI is crucial to prevent progression to significant kidney damage.
Assessment based on:
Electrolyte Testing: Determines the metabolic response and needs for renal replacement therapy.
Comorbidities: Consider diabetes, cardiac issues, hypertension.
Nutritional Status: Importance of adequate protein intake due to possible protein breakdown issues associated with AKI.
Nutritional Management
Protein Intake:
Focus on replacing protein without exceeding levels that might provoke uremia.
Targeted protein intake is vital for growth and tissue repair, especially in AKI patients.
Carbohydrate Intake:
Increased carbohydrate intake helps meet caloric requirements while sparing protein for vital functions.
Nutritional Restrictions:
Sodium, Potassium, Phosphorus: Restrict high levels found in foods (e.g., bananas, citrus fruits, dairy) based on lab work results.
Nursing Management
Fluid and Electrolyte Monitoring:
Track intake and output; aim for minimal volume IV fluids or medications.
In case of low blood pressure, consider albumin instead of normal saline for bolus therapy.
Assessment of Urine Output:
Monitor urine output and edema; implement strategies to control progression and educate the patient.
Metabolic Rate Management:
Address fever/infection immediately as they increase metabolic demand, employing treatments like antipyretics and antibiotics.
Promoting Pulmonary Function:
Encourage mobility to prevent atelectasis and respiratory infections through techniques like turning, coughing, and deep breathing.
Infection Control:
Use sterile techniques for procedures (e.g., catheterization) to reduce risk of infection.
Skin Care:
Monitor for skin breakdown; bathe with tepid water, moisturize, and regularly turn patients to protect skin integrity.
Chronic Kidney Disease Management
Assessment and Diagnosis:
Regularly evaluate renal function through GFR, serum creatinine, and BUN levels.
Uremia Monitoring:
Recognize that increased levels of urea can cause severe multisystem effects.
Stage Identification:
CKD progression leads to cardiovascular risks from hypertension, anemia, and inadequate blood sugar management.
Signs of Progression:
Watch for electrolyte imbalance symptoms like hyperkalemia leading to dysrhythmias, and recognize the need for early management to slow deterioration.
Treatment Options
Medication Management:
Include phosphate binders (e.g., calcium-based binders) to manage phosphorus intake, alongside calcium and vitamin D supplements.
Antihypertensive agents, dietary sodium restrictions, and fluid management are also essential.
Dietary Strategies:
Formulate diet plans to optimize protein and carbohydrate intake while restricting potassium and phosphorus to prevent build-up and manage symptoms.
Dialysis Consideration:
For patients requiring hemodialysis, ensure they understand the schedule (e.g., M/W/F or T/Th/S) and community resources available to them for support.
Patient Education
Fluid Management:
Urge patients to adhere to fluid restrictions (e.g., 1000 mL/day for anuric patients) and strategies to manage thirst effectively.
Dietary Restrictions:
Educate on renal diet specifics, avoiding foods high in potassium and phosphorus, including limiting processed and high-sodium products.
Awareness of Symptoms:
Teach patients and families about potential complications of CKD and signs to monitor for worsening conditions.
Community Resources:
Provide information on available support services, treatment options, and lifestyle changes to improve quality of life.