Obstetrics and Gynecology Flashcards
Endometriosis
Uterus not enlarged, bleeding into the peritoneal cavity.
Symptoms: Dysmenorrhea and dyspareunia (painful sex, especially during menstruation).
May displace cervix, causing cervical motion tenderness.
Risk factors: Nulliparity and low BMI.
Can cause infertility by disrupting pelvic anatomy, oocyte release, and sperm entry.
Treatment: NSAIDs, oral contraceptives, Mirena, GnRH agonists, cauterization, or removal of ovaries.
Uterine Polyps
Light abnormal uterine bleeding (AUB) with a normal-sized uterus.
Pedunculated fleshy growth that may be calcified.
Hyperechoic.
Doesn't affect ovulation; menses are regular with light intermenstrual bleeding due to friability and vascularity.
Fibroids
Irregular-sized, bulky, enlarged uterus due to smooth muscle proliferation within the myometrium.
Typically cause regular (ovulatory) but heavy, prolonged menses (menorrhagia), can cause AUB.
Growth increased by estrogen, pregnancy, and OCPs.
Globular masses can cause breech presentation.
Adenomyosis
Symmetrically (diffusely) enlarged, boggy (soft) uterus.
Endometrial glands within the myometrium.
Menorrhagia and dysmenorrhea, but no intermenstrual bleeding.
Treatment for AUB: IV estrogen or high-dose OCPs to stabilize the endometrium; high-dose progestin if estrogen is contraindicated.
Third Trimester Bleeding
Placenta Previa
Abnormal implantation where the placenta grows across the os.
Cervical dilation causes placental tearing.
Initial reassuring fetal monitoring.
Diagnosis: Transabdominal followed by TVUS.
Management: No sex, no digital cervical exam, inpatient admission for bleeding episodes.
Risk factors: Previous placenta previa, prior caesarean, multiple gestation, advanced maternal age.
Most cases resolve spontaneously by trimester 3; persistent cases require caesarean delivery at 36-37 weeks.
Vasa Previa
Blood vessels connect the uterus to an accessory lobe and lie across the os.
Tearing during cervical dilation leads to fetal exsanguination.
Diagnosis: NST showing fetal distress, bradycardia, and sinusoidal pattern.
Treatment: Urgent C-section.
Uterine Rupture
Risk factors: Previously scarred uterus (C-section), oxytocin-induced forceful contractions.
Loss of fetal station.
Fetal HR: Uterine rupture if 80 bpm at nadir; peak to nadir >30 sec.
Sudden decrease in HR with prolonged fetal HR deceleration.
Mother experiences increasing lower abdominal pain; vaginal bleeding is not always present.
Fetal parts may partially expel into the maternal abdomen, causing a palpable irregular abdominal protuberance.
Treatment: Crash C-section.
Vaginal Hematoma
Risk factors: Operative vaginal delivery, infant >8.8lb, nulliparity, prolonged 2nd stage of labor.
Vaginal purplish mass +/- hypovolemic shock.
Minimal external bleeding but significant blood loss as blood collects in the paravaginal potential space.
Treatment: Observation for non-expanding hematomas; embolization or surgery for expanding hematomas.
Uterine Inversion
Risk factors: Nulliparity, macrosomnia, placental accreta, rapid labor and delivery.
Due to excessive fundal pressure and traction on the umbilical cord before placental separation.
Smooth round mass protruding through the vagina/cervix; massive postpartum hemorrhage and hypovolemic shock with lower abdominal pain.
Uterine fundus no longer palpable transabdominally.
Management: Aggressive fluid replacement, manual replacement of uterus, placental removal, and uterotonic drugs after replacement.
Placental Abruptio
Placenta tears off the endometrium but remains in place.
Pain can be in the back or abdomen.
Firm uterus with unusually low-amplitude high-frequency contractions.
Concealed bleeding can cause uterine distension.
Large force needed to tear placenta off: HTN/cocaine use; MVA.
Tender, hypertonic uterus.
Diagnosis: US/NST.
Kleihauer-Betke test for fetomaternal hemorrhage to determine the necessary dose of anti-D Ig after delivery of Rh+ fetus to Rh- mother.
Fetal Heart Rate Decelerations
VEAL CHOP
Early decelerations: Head compression.
Late decelerations: Fetal hypoxemia and forceful contractions; stop oxytocin, give mother O2, change position (left lateral with legs raised), IV fluids.
Variable decelerations: Cord compression/abnormal position; abrupt decline and return to baseline.
Cord compression with <50% contractions well tolerated; maternal repositioning to reduce cord compression, amnioinfusion if no improvement.
Most ominous when repetitive and severe (below 60 bpm); if recurrent, perform Caesarian.
Prolonged deceleration: Decrease of 15 bpm below baseline for 2-10 minutes.
Sinusoidal tracing: Severe hypoxia (e.g., in Vasa Previa and Rh disease).
Umbilical Cord Prolapse
Abrupt, prolonged deceleration or bradycardia.
Contraception
Long Acting Reversible Contraception (LARC)
Most effective, even compared to tubal ligation/vasectomy.
Non-IUD: Subcutaneous etonogestrel arm implant (lasts 3 years); irregular bleeding/spotting common initially.
IUD
CI: Acute pelvic infection, severe uterine cavity distortion, AUB, Wilson disease.
Hormonal (5 years): CI in current breast cancer; can cause irregularity.
Copper (10 years): Longest acting.
Moderate Efficacy
Injections (3 months).
Patches (E+P): Highest risk of DVT/PE.
Rings (E+P): Minimal breakthrough bleeding.
OCPs: Inhibit LH surge -> no ovulation; metabolized via P450; used in AUB/choriogestational trophoblastic disease/molar pregnancy.
Mini Pill (Progesterone only): Daily compliance is crucial.
Low Efficacy
Condoms and diaphragms (diaphragm > cup).
Hysteroscopic Sterilisation
Insertion of nickel into FTs causing gradual scarring (backup birth control for 3 months until occlusion confirmed via hysterosalpingogram).
CI: Previous tubal ligation; allergy to contrast media/nickel; uterine/FT pathologies; recent/active pelvic infection.
OCP Combo Pills
Inhibits LH surge.
CI: >35 if smoking >15 cigarettes per day; SLE; migraine with aura; breast cancer; cirrhosis; RF for CV disease; thromboembolism; Hx of stroke, hepatocellular adenoma.
Oestrogen containing contraceptives CI <1 month postpartum (increased risk of thromboembolism).
Medroxyprogesterone Acetate Injection
Thickens cervical mucus; decidualises endometrium -> atrophy; impairs Tubal peristalsis.
Osteoporosis risk in over 35s.
Progestin only IUD does not affect thrombotic state -> systemic forms still not used in high risk pt.
Protective against endometrial cancer.
Oestrogen only contraception is CI in breastfeeding as can decrease milk production.
Emergency Contraception
Copper IUD: 0-120hr, >99% effective.
Ulipristal (Progestin R blocker): 0-120hr, 98-99% effective (delays ovulation).
Levonorgestrel: 0-72hr.
High dose OCPs: 0-72hr.
Oestrogen-progestin Contraceptive
Pros
Endometrial and ovarian cancer risk reduction, menstrual regulation, hyperandrogenism tx.
Cons
Thromboembolism; HTN; Stroke; MI, hepatic adenoma, cervical cancer, moderate breast cancer risk, mood changes (mainly due to progesterone).
Primary Amenorrhea
Absence of menarche in girls aged 13 or older with no secondary sexual characteristics or absence of menarche by 15 w secondary sexual characteristics
Absent Uterus? Go to Karyotype
46 XY = AI
46 XX = Mullerian Agenesis
Uterus Present? Check FSH
Low FSH = Central cause: Hypothyroidism, Prolactinoma, Functional Hypothalamic Amenorrhea (FHA), Cushings Syndrome
Norm FSH = Imperforate Hymen
High FSH = Peripheral cause: Primary Ovarian Insufficiency or Turners
Mullerian Agenesis (Mayer-Rokitansky-Kuster-Hauser Syndrome): MCC, normal secondary sexual characteristics, absent uterus and upper vagina (46XX with intact ovaries - hormonally normal), management includes renal tract evaluation and vaginal dilation.
Androgen Insensitivity Syndrome (defect in androgen receptor): Normal appearing female, decreased pubic and axillary hair, increased height, 46XY, presence of intraabdominal testicles, increased testosterone and LH and estrogen (aromatase conversion of testosterone)
5-alpha reductase deficiency: Virilisation at puberty, 46XY, phenotypically F at birth, impaired testosterone to DHT conversion; normal male testosterone and estrogen levels, male internal genitalia, blind ending vagina, maternal virilisation (aromatase deficiency in foetus).
Turners Syndrome: Increased risk of aortic dissection d/t bicuspid AV; short stature; webbed neck; scoliosis; micrognathia; cubitus valgus renal and ovarian disorders common.
Abnormal Uterine Bleeding
Immature HPO Axis: Anovulatory, painless irregular heavy bleeding, unopposed estrogen, Tx with progesterone or combined oral pill.
Progesterone Challenge: Withdrawal bleeding suggests anovulation, Estrogen challenge if progesterone challenge negative (withdrawal bleeding signifies lack of Estrogen).
Maternal Oestrogen Effects in Newborns: Breast Hypertrophy, Swollen Labia, Physiologic Leukorrhea, Uterine withdrawal bleeding are all normal and transient.
Testes: Should descend by 6 months
Hypospadias (inferior): Ventrally displaced urethral opening, Karyotype and US analysis if severe.
Torsion of appendix testes: Classic ‘blue dot’ pathognomic sign, conservative Tx.
Peyronie Disease: Penile pain, curvature and dorsal nodules/plaques resolves spontaneously in 1-2 yrs in most; NSAIDs for pain and surgery.
Penile Cancer
Bowen disease: Cutaneous SCC in situ can progress to cancer (painless ulcer/nodule with inguinal LAD)
Varicocele: Decreased fertility and testicular atrophy, check for left renal cell cancer - Tx gonadal vein ligation.
Spermatocele: Cystic epididymis accumulations of sperm; usually asymptomatic.
Hydrocele: Transilluminates, collection of serous fluid;
Non communicating in adults - cancer/infection/trauma
Communicating in kids: Incomplete closure of processes vaginalis.
Hematocele: Testicular trauma Blood accumulation in tunica vaginalis; Pain.
Erectile Dysfunction Causes: Vascular, Neurological, Psychogenic (Norm non sexual nocturnal/morning erections), Endocrine (TSH/Prolactin - Hypogonadism), Medications.
Breast Mastitis: Flu like sx + fever; wedge shaped redness; purulent discharge, Dicloxacillin to cover MRSA and penicillin resistant Staph.
Breast fibroadenomas: Work up with US (less than 30) and Mammogram (more than 30).
Fibrocystic Changes of Breast: Diffuse, bilateral premenstrual tenderness - OCPs; danazol; NSAIDs
Galatocele: Benign milk retention cyst Can occur during lactation and Tri3; soft, mobile nontender mass as large as 5 cm
Phyllodes tumour - larger and greater metastatic potential
Paget’s disease of Breast - Erythematous, scaly or vesicular rash affecting nipple and areola Intraductal papilloma: No associated mass or LAD
Physiological Galactorrhea - Bilateral and guaiac negative w/o signs of malignancy
Inflammatory Breast Carcinoma - Edema, diffusely warm, dimpling, peau d’orange.
Tx of Breast cancer: SERMs - Risk of venous thromboembolism; endometrial cancer, Raloxifene - Risk of venous thromboembolism
Breastfeeding CI - Active untreated TB; HIV infection; Active varicella infection; Herpetic breast lesions
Pregnancy
Exercise CI
Increased risk antepartum bleeding eg placenta previa, Risk of preterm delivery eg insufficiency and Cardioresp Disease.
Shoulder Dystocia: Failure of usual obstetric manoeuvres to deliver fetal shoulders
Cephalopelvic disproportion: Failure to progress in labour
Hypotonic Contractions: Failure to progress in Labour
HTN and short interpregnancy interval risk of growth restriction
Oligohydramnios- Complications- Meconium aspiration; Preterm delivery; Umbilical cord compression
Polyhydramnios- Complications- Fetal malposition; Umbilical cord prolapse; Preterm labour; PPOM
Monochorionic gestation:- Risk of TTTS and Dichorionic gestation: LAMBDA SIGN
Patients at <32 gestation in preterm labour need: Indomethacin; Bethamethasone; Magnesium sulfate; Penicillin
External cephalic version: >/=37 wks gestation If breech/transverse
Preeclampsia: New onset HTN and proteinuria and/or end-organ damage at >20wks gestation Can present up to 6 wks postpartum - Delivery at 37wks
Preeclampsia with severe features —-> delivery >34wks Indication- obstetrical indications (failed induction); fetal HR abnormalities; Baby >5000g/4500 if gestational diabetes
HELLP Syndrome: Hemolysis, elevated liver enzymes, low platelets - Life threatening - Delivery (warranted at 34 wks or deteriorating maternal/fetal status)
Intrahepatic cholestasis of pregnancy: Generalised pruritusDevelops in third trimester Pruritus worse on hands and feet Complications- Intrauterine demise, preterm delivery, meconium stained amniotic fluid, neonatal RDS Management: Deliver at 37 wks
Acute fatty liver of pregnancy- RUQ pain, mildly elevated transaminases and resultant intrauterine demise Liver failure; Platelets </= 100,000; Immediate delivery
Maternal fever d/t intraamniotic infection (IAI) -> fetal tachycardia (>160 bpm)
Erythroblastosis fetalis- ABO Incompatabilty and Rh Incompatability RhoD IgG normally given at 28 wks and within 72 hrs
Pregnancy 2
PPROM: Rupture of membranes w/o onset of labour External Cephalic Version: Indicated for breech or CI - vaginal delivery, prior classical C Section Complications: Abruptio placentae and Intrauterine fetal demise
Vaginal delivery CI - incomplete or footling breech
Primary Dysmenorrhea- can be accompanied by n/v, diarrhoea or back pain Menopause- Need 12 months no bleeding to dx menopause - clinical dx.
Prolapse can occur post menopause, tx with hysterectomy, bladder tape for stress incontinence,
HRT- Treats vasomotor sx; mood and dryness in F < 60 and Topical estrogen used if mainly vaginal sx
Genitourinary syndrome of menopause- Urinary incontinence, recurrent UTI, Narrowed introitus, Loss of labial volume - Tx: Vaginal moisturiser and lubricant; Topical vaginal oestrogen
Vesicovaginal Fistula: Continuous clear vaginal discharge - Bladder dye testing used to confirm Dx
(Vulvar) Lichen Sclerosus: Vulvar pruritus and Hypopigmented (white) labial lesions Dx - Clinical with punch biopsy to confirm + rule out vulvar cancer and Tx - Superpotent topical corticosteroids eg Clobetasol
Pelvic Inflammatory Disease- Constant Pelvic pain, tenderness of cervix Purulent discharge Complications: Tubo-ovarian abscess; Infertility; Ectopic Pregnancy, Tx : IM Ceftriaxone + ORAL doxycycline if outpatient or IV Cefoxitin/cefotetan + oral doxy if inpatient.
Secondary dysmenorrhea: Sx onset >25yrs, pain is Unilateral and Secondary Amenorrhea- MCC is pregnancy Pregnancy test ==> Serum FSH/TSH/Prolactin
Ectopic Pregnancy Most common site is FT RFs: PID, smoking, presence of IUD; Amenorrhea; Syncope; Intraabdo bleeding Quantitative, Beta hCG Inappropriate rise Beta hCG points to ectopic Progesterone lever US: Use serial beta hCGs Laparotomy in hemodynamically unstable Medical management: Methotrexate - CI Blood dyscrasias Surgical Management:
Exposure to teratogens - Viruses (rubella, CMV) Mercury, alcohol
Ruptured ovarian cyst- Peritoneal signs and referred pain to shoulder Amnioinfusion CI if hx of uterine surgery
Recurrent Pregnancy Loss: Genetic Factors Chromosomal (normal couple; Bal translocations and Trisomies) Causes- Uterine and Cervical Abnormalities