neurology

TABLE OF CONTENTS

  1. Embryology
  2. Cellular Function
  3. Ascending Spinal Cord Tracts
  4. Descending Spinal Cord Tracts
  5. Brainstem Anatomy
  6. Cerebral Cortex
  7. Radiculopathy
  8. Spinal Cord Syndromes
  9. Invasive Spinal Cord Disease
  10. Demyelinating Disease
  11. Conduction Physiology
  12. Neuromuscular Junction (NMJ)
  13. Cranial Nerves 1-6
  14. Cranial Nerves 7-12
  15. Vision Neurology
  16. Auditory Sensation
  17. Vertigo
  18. Pediatric Brain Tumors
  19. Adult Primary Brain Tumors
  20. Ischemic Cerebrovascular Accidents
  21. Aneurysms and Intracranial Hemorrhage
  22. Cerebellum and Ventricles
  23. Diencephalon
  24. Basal Ganglia
  25. Neurotransmitter Activity in Psychiatric Disease
  26. Dementia
  27. Headache
  28. Seizures
  29. Traumatic Brain Injuries

EMBRYOLOGY

A. Notochord

  • Derived from mesoderm.
  • Induces ectoderm to form neuroectoderm at ~2.5 weeks gestation.

B. Neural Tube

  • Arises from medial ectoderm.
  • Fuses dorsally to form the neural tube at ~3 weeks gestation.

C. Meninges

  • Dura Mater:
    • Derived from mesoderm.
  • Arachnoid Mater:
    • Comes from neural crest cells.
  • Pia Mater:
    • Also derived from neural crest cells.
  • Epidural Space:
    • Associated with epidural anesthesia and hematomas.
  • Subdural Space:
  • Contains potential space for subdural hematomas.
  • Subarachnoid Space:
    • Contains cerebrospinal fluid (CSF).
    • Involvement in lumbar punctures and hemorrhage.

D. Origins of Nervous Tissue

  • Forebrain/Prosencephalon:

    • Diencephalon: Hypothalamus, thalamus, pineal gland.
    • Telencephalon: Cerebral hemispheres, basal ganglia.
  • Midbrain/Mesencephalon:

    • Midbrain structures.
  • Hindbrain/Rhombencephalon:

    • Metencephalon: Cerebellum, pons.
    • Myelencephalon: Medulla oblongata.

E. Neural Tube Defects

  • Anencephaly:
    • Caused by failure of rostral neuropore closure, leads to fatal absence of forebrain.
  • Lissencephaly:
    • Characterized by absent cortical sulci and gyri.
  • Spina Bifida:
    • Occulta: Vertebral defect with skin manifestations.
    • Meningocele: Meninges through defect.
    • Myelomeningocele: Spinal cord through defect, associated with Chiari II malformations.

CELLULAR FUNCTION

A. Neuronal Cells

  • Schwann Cells (PNS):

    • One cell myelinates one axon.
    • Derived from neural crest cells, S100 positive.
    • Effective in Wallerian degeneration and regeneration.
    • Relevant conditions: GBS, acoustic neuroma.
  • Oligodendrocytes (CNS):

    • One cell myelinates multiple axons.
    • From neuroectoderm, ineffective in regeneration.
    • Associated with MS, PML, oligodendroglioma.
  • Astrocytes:

    • Maintain blood-brain barrier.
    • From neuroectoderm, GFAP marker.
    • Associated with glioblastoma multiforme.
  • Microglia:

    • CNS phagocytes, from mesoderm, involved in HIV-associated dementia.

B. Neuronal Anatomy

  • Soma: Contains organelles, Nissl substance (rough endoplasmic reticulum).
  • Axon: Delivers action potentials.
  • Dendrites: Receives signals, contains Nissl substance.
  • Neuron Injury: Cellular edema, peripheral nucleus, chromatolysis.

C. Wallerian Degeneration

  • PNS:

    • Axonal retraction keeps proximal axonal membrane.
    • Schwann cells break down myelin, facilitating regeneration.
    • Macrophages clear debris.
  • CNS:

    • Oligodendrocytes poorly manage debris and regeneration.
    • Macrophages must cross the blood-brain barrier.

D. Sensory Receptors

  • Aδ and C fibers: Responsible for pain transmission.
  • Meissner Corpuscles: Senses fine touch and vibration.
  • Pacinian Corpuscles: Detect pressure and high-frequency vibrations.

ASCENDING SPINAL CORD TRACTS

A. Spinothalamic Tract

  • Lateral Spinothalamic: Pain, temperature.

    • Decussates 1-2 levels above.
  • Anterior Spinothalamic: Crude touch, pressure.

B. Dorsal Column-Medial Lemniscus

  • Decussates at medulla, conveys fine touch and proprioception.
    • Fasciculus Gracilis: T6 and below.
    • Fasciculus Cuneatus: T5 and above.

C. Spinocerebellar Tract

  • Delivers unconscious proprioception, ipsilateral to the cerebellum.

DESCENDING SPINAL CORD TRACTS

A. Corticospinal Tract

  • Lateral Corticospinal: Voluntary movement, decussates at the inferior medulla.
  • Anterior Corticospinal: Remains ipsilateral until termination at specific cervical and thoracic levels.

B. Corticobulbar Tract

  • Leads to head and neck muscle control, innervates certain cranial nerves bilaterally and contralaterally.

BRAINSTEM ANATOMY

A. Midbrain

  • Structures and Functions: CN III and IV nuclei, red nucleus, substantia nigra involved with motor functions.

B. Pons

  • Contains CN V-VIII nuclei, reticular activating system, and longitudinal fasciculi.

C. Medulla

  • Nuclei of CN VIII-XII, vital for autonomic control and reflexes.

CEREBRAL CORTEX

A. Frontal Lobe

  • Contains primary motor cortex, areas for personality and speech regulation (Broca's area).

B. Temporal Lobe

  • Involved in auditory processing (Wernicke's area) and memory.

C. Parietal Lobe

  • Responsible for somatosensory processing and spatial orientation.

D. Occipital Lobe

  • Primary visual processing center.

RADICULOPATHY

General Spinal Cord Function

  • Anatomical considerations of spinal cord and common reflexes per level.
  • Impingement effects and symptoms from nerve root involvement.

SPINAL CORD SYNDROMES

A. Anterior Cord Syndrome

  • Typically due to anterior spinal artery occlusion, leads to bilateral loss of pain and temperature.

B. Brown-Sequard Syndrome

  • Hemisection presents pain and temperature loss contralaterally, proprioception and motor function loss ipsilaterally.

C. Central Cord Syndrome

  • Greater upper than lower extremity weakness; common in elderly with hyperextension injuries.

INFECTIOUS SPINAL CORD DISEASE

A. Tetanospasmin Toxin

  • Binds to Renshaw cells; causes spastic paralysis classic findings.

B. Rabies Virus

  • Retrograde transsynaptic transport to the CNS, hydrophobia as a hallmark.

C. Poliomyelitis

  • Causes asymmetric flaccid paralysis in unvaccinated individuals.

DEMYELINATING DISEASE

A. Multiple Sclerosis

  • Characterized by optic neuritis and neurological deficits; varied presentations like internuclear ophthalmoplegia.

B. Guillain-Barré Syndrome

  • Ascending paralysis after GI infection, classic CSF findings.

CONDUCTION PHYSIOLOGY

A. Resting Potential

  • Neuronal membrane potential, determined primarily by sodium and potassium ions.

B. Action Potential

  • Depolarization followed by repolarization phases of neuronal firing.

NEUROMUSCULAR JUNCTION (NMJ)

A. Overview

  • Communication between motor neurons and muscle fibers; neurotransmitter release and receptor activation.

CRANIAL NERVES

I - VI

  • Overview, function, associated lesions and symptoms.

VISION

A. Pathway and Clinical Presentation

  • From retina to visual cortex; common pathologies affecting this pathway.

AUDITORY

A. Basics

  • Transport currents, anatomy of the auditory system, testing for hearing loss.

VERTIGO

A. Peripheral and Central Causes

  • Presentation: positional vs spontaneous vertigo, clinical evaluations.

PEDIATRIC BRAIN TUMORS

Main Types

  • Include Medulloblastoma, Astrocytoma, and Ependymoma along with clinical presentations.

ADULT PRIMARY BRAIN TUMORS

A. Classification

  • Include Glioblastoma, Astrocytoma, Meningiomas among others, with typical lesions and presentations.

ISCHEMIC CEREBROVASCULAR ACCIDENTS

A. Classification

  • Thrombotic, embolic, and watershed areas of infarcts.

HEMORRHAGIC CEREBROVASCULAR ACCIDENTS

Details

  • Types of hematomas, clinical findings, diagnosis, and management strategies.

DYNAMIC ANATOMY OF BRAINSTEM

A. Functional Sites & Clinical Relevance

  • Breakdown of roles and lesions associated with various cranial nerve nuclei, control pathways, and clinical symptoms.

SPINAL CORD DISEASES AND SYNDROMES

A. Definitions

  • Overview and symptoms of various spinal cord syndromes affecting motor and sensory functions.

CONCLUSIONS

  • Summation of critical points and clinical correlations to guide further study into neurology and associated pathologies.