Study Notes on Antibiotics Use in Food Animals
ANTIBIOTICS USE IN FOOD ANIMALS
Presented by: Eoin Ryan MVB MVM DECBHM
Contact: shumphreysdvm@gmail.com
LEARNING OBJECTIVES
1) Understand the basic principles of antibiotic selection.
2) Differentiate between MIC (Minimum Inhibitory Concentration) and Kirby-Bauer method.
3) Review the fundamentals of each pertinent antibiotic class, which includes:
Mechanism of Action (MOA)
Spectrum of activity
Time vs concentration dependency
Tissue penetration (hydrophilic vs lipophilic)
Adverse side effects
Special restrictions (where applicable)
PRINCIPLES OF ANTIMICROBIAL SELECTION
Are antibiotics indicated?
What is the known/suspected organism?
What antibiotics are the known/suspected organism susceptible to?
Can therapeutic concentrations be achieved and maintained at the site of the infection?
Work within the limitations of the label (or legal Extralimital Drug Use - ELDU).
ARE ANTIBIOTICS INDICATED?
Questions to assess:
Is there an infectious agent involved in the disease process?
Investigate through diagnostics.
Are antimicrobials necessary to clear the infection?
Does the animal have a reasonable chance of clearing the infection with antibiotics?
Are there any alternative treatments that can be implemented concurrently with antibiotics?
Quote: "The best antibiotics are a scalpel and a drainage needle."
WHAT IS THE KNOWN/SUSPECTED ORGANISM?
Ideal scenario: Take actions to identify the infectious agent involved.
Easy and cost-effective methods:
Cytology
Gram stain
Best approach (though costly):
Culture
PCR (Polymerase Chain Reaction)
Reality: The infectious agent is often unknown.
Collect samples for identification at initial evaluation.
If treatment must begin before identification:
Predict likely organisms based on experience and knowledge.
WHICH ANTIBIOTICS ARE THE BACTERIA SUSCEPTIBLE TO?
Ideal scenario: Identify the organism known to be susceptible to certain antibiotics.
Qualitative susceptibility testing methods:
Kirby-Bauer (Disc diffusion susceptibility test)
Quantitative susceptibility testing method:
Minimum Inhibitory Concentration (MIC)
Reality: Often the antimicrobial susceptibility is unknown.
Submit samples for susceptibility testing.
In the interim, predict likely susceptibility based on geographical patterns and past cases.
WILL OUR ANTIBIOTICS REACH AND WORK AT THE SITE OF INFECTION?
This factor is influenced by the physiology and pathological state of the infection site.
Examples:
Encapsulated abscess
Fibrin accumulation
Inflamed or “leaky” central nervous system (CNS)
Pharmacokinetics/Pharmacodynamics (PK/PD) optimization is essential to ensure therapeutic concentrations are maintained at the site of infection.
PK/PD OPTIMIZATION
Pharmacokinetics: Refers to how the body affects the drug, including:
Absorption
Distribution
Metabolism
Elimination
Pharmacodynamics: Indicates how the drug acts within the body, encompassing:
Mechanism of Action (MOA): bactericidal vs bacteriostatic
Concentration-dependent vs time-dependent effects
Duration of post-antibiotic effect
Toxicity considerations
TIME- VS CONCENTRATION-DEPENDENT KILLING
Time-dependent killing:
Goal: Maintain plasma concentrations above the MIC for more than 50% of the dosing interval.
Concentration-dependent killing:
Goal: Achieve peak plasma concentrations that are 8-10 times higher than the MIC.
Post-antibiotic effect allows trough concentrations to fall below MIC.
Monitor for potential toxicity associated with sustained high trough concentrations.
MECHANISMS OF ANTIMICROBIAL AGENTS
Cell Wall Synthesis Inhibitors:
Beta Lactams:
Penicillins
Cephalosporins
Carbapenems
Monobactams
Vancomycin
Bacitracin
Folate Synthesis Inhibitors:
Sulfonamides
Trimethoprim
Nucleic Acid Synthesis Inhibitors:
DNA Gyrase: Quinolones
RNA Polymerase: Rifampin
Protein Synthesis Inhibitors:
50S subunit:
Macrolides
Clindamycin
Linezolid
Chloramphenicol
Streptogramins
30S subunit:
Tetracyclines
Aminoglycosides
COMMON BACTERIA BY GRAM STAINING
Gram + Aerobes:
Streptococcus
Staphylococcus
Corynebacterium*
Listeria*
Enterococcus
Trueperella*
Gram - Aerobes:
E. coli
Pasteurella multocida
Pseudomonas
Manheimia hemolytica
Histophilus somni*
Enterobacteriaceae
Actinobacillus
Gram + Anaerobes:
Clostridium
Peptostreptococcus
Actinomyces
Gram - Anaerobes:
Bacteroides
Fusobacterium
Porphyromonas
Prevotella
Atypical:
Mycoplasma
* = facultative anaerobe
ANTIBIOTICS FOR LIVESTOCK
Gram + Aerobes:
Beta-lactams
Cephalosporins*
Macrolides
Potentiated sulfas*
Tetracyclines
(Fluoroquinolones* – for Staph)
Rifampin
Gram - Aerobes:
Beta-lactams
Cephalosporins*
Fluoroquinolones*
Potentiated sulfas*
Tetracyclines
Florfenicol
Aminoglycosides
Gram + Anaerobes:
Beta-lactams
Cephalosporins*
Tetracyclines
Macrolides
Florfenicol
Rifampin
Metronidazole
Gram - Anaerobes:
Beta-lactams
Cephalosporins*
Tetracyclines
Macrolides
Florfenicol
Rifampin
Metronidazole
Atypical:
Tetracyclines (+/-)
Macrolides
Florfenicol
Fluoroquinolones
* = limited use in livestock
CONCLUSIONS
Thorough diagnostic efforts are critical prior to treatment to optimize outcomes in clinical cases involving antibiotics. The importance of understanding antimicrobial resistance, patient history, and adaptations in treatment strategies is emphasized.
QUESTIONS
Open-ended questions discussed throughout the presentation for further insight into approaches to treatment and management of cases involving antibiotics in food animals.