Neurologic Trauma: Head Injuries & Spinal Cord Injuries

Head Injury: Scope & Prevention

  • Broad spectrum of trauma-induced damage to scalp, skull, or brain
  • U.S. annual burden:
    • 2.9 million2.9\text{ million} emergency-department (ED) visits for traumatic brain injury (TBI)
    • 56,80056{,}800 deaths (≈30%30\% of all injury-related fatalities)
  • Most common etiology of TBI = falls
  • High-risk cohorts:
    • Children 04 yr0\text{–}4\text{ yr}
    • Adolescents 1519 yr15\text{–}19\text{ yr}
    • Adults 65 yr\ge 65\text{ yr}
    • Higher incidence in males
  • Primary strategy = prevention (helmets, fall precautions, seat belts, violence reduction)

Brain Damage: Primary vs. Secondary

  • Primary injury – occurs at moment of impact:
    • Contusions, lacerations, skull or external hematomas, fractures, subdural hematomas, concussion, diffuse axonal injury (DAI)
  • Secondary injury – evolves minutes → days post-trauma:
    • Cerebral edema, ischemia, biochemical & inflammatory cascades, ↑ intracranial pressure (ICP)
  • Progressive pathophysiologic sequence:
    1. Traumatic impact
    2. Brain swelling / bleeding ↑ intracranial volume
    3. Rigid cranium prevents expansion → ICP rises
    4. Cerebral vessels compressed → ↓ cerebral blood flow (CBF)
    5. Cerebral hypoxia & ischemia; risk of herniation
    6. CBF may cease → brain death

Glossary Check: Concussion vs. Contusion

  • Concussion = temporary neurologic dysfunction with no visible structural injury
  • Contusion = bruise of cortical surface; may include hemorrhage & edema

Scalp Wounds & Skull Fractures

  • Scalp wounds: profuse bleeding, ↑ infection risk
  • Skull fracture manifestations vary by site/severity
    • Localized, persistent pain common
    • Basilar skull fracture hallmark signs:
    • CSF rhinorrhea / otorrhea (clear drainage) → positive halo sign
    • Battle sign (post-auricular ecchymosis)
    • Epistaxis or ear bleeding

Classification of Traumatic Brain Injury (TBI)

  • Closed (blunt): acceleration–deceleration without skull violation
  • Open (penetrating): scalp & skull breached by object/force
  • Concussion: brief LOC ± retrograde amnesia; usually GCS 131513\text{–}15
  • Contusion: more severe; prolonged unconsciousness, focal deficits, vitals changes
  • Diffuse Axonal Injury (DAI): widespread shearing of axons → immediate coma, poor prognosis

Intracranial Bleeding Phenotypes

  • Epidural Hematoma (EDH)
    • Arterial bleed between skull & dura mater
    • Classic "lucid interval" then rapid LOC decline
    • Surgical emergency: burr holes or craniotomy; ICP control; airway support
  • Subdural Hematoma (SDH)
    • Venous bleed between dura & arachnoid
    • Acute: 2448 h24\text{–}48\text{ h} onset; Sub-acute: 48 h48\text{ h}2 wk2\text{ wk}
    • Chronic: weeks–months; minor trauma often forgotten; fluctuating symptoms
    • Therapy: craniotomy & evacuation, ICP management
  • Intracerebral Hemorrhage / Hematoma
    • Bleeding inside brain parenchyma; traumatic or spontaneous (HTN, AVM)
    • Care: supportive, ICP control, fluid & BP optimization; surgical removal if accessible

Concussion Observation Parameters (Discharge Teaching)

  • Monitor and immediately report:
    • Any ↓ LOC or difficulty arousing
    • Worsening headache, dizziness, confusion, irritability, anxiety
    • Speech or motor deficits
    • Persistent nausea / vomiting
  • Wake & assess frequently for first 2448 h24\text{–}48\text{ h}

Assessment & Diagnostics for Head Injury

  • Immediate physical & neurologic exam; Glasgow Coma Scale (GCS)
  • Assume cervical spine injury → rigid collar until cleared
  • Imaging:
    • CT scan (first-line)
    • MRI for diffuse, posterior fossa, brain-stem lesions
    • PET for cerebral metabolism/function

Acute Management Goals (Head Injury)

  • Preserve cerebral homeostasis; prevent secondary damage:
    • Stabilize cardio-respiratory status → maintain cerebral perfusion pressure (CPP)
    • Control hemorrhage & hypovolemia
    • Optimize blood gases (PaO<em>2>60mmHg<em>2 >60\,\text{mmHg}; PaCO</em>2</em>2 targets individualized)
    • Treat ↑ ICP & edema (osmotherapy, drainage)
    • Surgical intervention when indicated (craniotomy, burr holes)
    • Continuous ICP monitoring; CSF drainage PRN

Supportive Measures (Head Injury)

  • Airway & ventilation (intubation/mech vent prn)
  • Seizure prophylaxis (levetiracetam, phenytoin, etc.)
  • NG tube for gastric decompression & aspiration prevention
  • Fluid/electrolyte balance; strict I&O
  • Analgesia & anxiolysis (avoid oversedation masking neuro exam)
  • Early nutrition (enteral within 2448 h24\text{–}48\text{ h})

Nursing Surveillance (Head Injury)

  • Serial neurologic checks: GCS, pupils, motor, vitals
  • Monitor labs: serum/urine osmolality, Na$^+$, glucose
  • Skin integrity, DVT prophylaxis, stimulus control, sleep hygiene
  • Family education & coping support

Traumatic Brain Injury: Potential Complications

  • ↓ Cerebral perfusion, cerebral edema, herniation
  • Hypoxia/ventilatory failure
  • Fluid, electrolyte, nutrition imbalance
  • Post-traumatic seizures

Spinal Cord Injury (SCI): Epidemiology & Etiology

  • 294{,}000AmericanslivewithSCIrelateddisability</li><li>Commonmechanisms:motorvehicleaccidents(MVA),falls,violence(GSW),sports</li><li>Demographics:Americans live with SCI-related disability</li> <li>Common mechanisms: motor-vehicle accidents (MVA), falls, violence (GSW), sports</li> <li>Demographics:78\%male;averageageatinjurymale; average age at injury43\text{ yr}
  • Risk factors: youth, male, alcohol/drug use
  • Leading mortality causes: pneumonia, pulmonary embolism (PE), sepsis

SCI Pathophysiology

  • Primary injury: contusion, laceration, compression at impact → often permanent
  • Secondary injury: edema, ischemia, hemorrhage escalate neurologic loss
  • Prompt interventions attempt to limit secondary spread

Spinal & Neurogenic Shock

  • Spinal Shock
    • Immediate areflexia below lesion; flaccid paralysis & sensory loss
    • Usually resolves within \approx 4\text{ wk}(variable)</li></ul></li><li>NeurogenicShock(aboveT(variable)</li></ul></li> <li>Neurogenic Shock (above T6 lesions)
      • Loss of sympathetic tone → ↓↓ BP, HR, CO; peripheral vasodilation & venous pooling
      • Anhidrosis below level of lesion

    Autonomic Dysreflexia (Hyperreflexia)

    • Acute life-threatening emergency once spinal shock resolves; lesions above T6
    • Triggers: bladder distention (most common), bowel impaction, skin stimuli
    • Presentation: pounding headache, sudden HTN, profuse diaphoresis, nausea, nasal congestion, bradycardia
    • Immediate nursing actions:
      1. Sit patient upright (↓ BP)
      2. Rapidly search & alleviate cause:
      • Drain bladder (catheter check/insert/irrigate)
      • Rectal exam for fecal mass (use anesthetic gel 10\text{–}15\text{ min} prior to removal)
      • Inspect skin, clothing, lines
      1. If BP remains ↑, give ganglionic blocker (e.g., hydralazine IV)
      2. Flag chart: "Risk for autonomic dysreflexia"
      3. Teach patient prevention strategies

    Respiratory & Airway Care in SCI

    • Continuous monitoring: pulse oximetry, ABG, lung sounds
    • Aggressive pulmonary toilet: humidification, hydration, assisted cough, incentive spirometry, cautious suctioning

    Mobility & Musculoskeletal Management (SCI)

    • Maintain spinal alignment; log-roll only per order or with stable spine
    • Specialized rotating bed if available; otherwise turn with precautions
    • Passive ROM (PROM) ≥4$$×/day; gradual elevation to sitting
    • Cervical collar / halo as prescribed
    • Monitor orthostatic BP with position changes

    Skin, Bowel & Bladder Programs (SCI)

    • Frequent inspection & pressure off-loading; high-protein, high-calorie diet
    • Indwelling or intermittent catheterization schedule; never massage calves/thighs (DVT risk)
    • High-fiber diet, stool softeners, scheduled bowel regimen

    Traction & Pin Care

    • Cleanse pin sites per protocol; monitor for infection
    • Maintain hygiene around traction devices

    SCI Complications

    • Deep-vein thrombosis (DVT)
    • Orthostatic hypotension
    • Autonomic dysreflexia
    • Spasticity, infection, sepsis in long-term paraplegia/tetraplegia

    Planning & Goals: Traumatic Brain Injury vs. SCI

    • Head Injury Goals
      • Patent airway & optimal CPP
      • Fluid/electrolyte & nutritional balance
      • Normothermia; skin integrity; prevention of secondary injury & sleep disturbance
      • Education & coping support; absence of complications
    • SCI Goals
      • Effective ventilation; airway clearance
      • Improved mobility; prevention of injury from sensory loss
      • Intact skin; urinary & bowel control; pain reduction
      • Recognition & prevention of autonomic dysreflexia; absence of complications
      • Achievement of functional mobility, sexual expression, long-term self-management knowledge

    Quick-Fire True/False & MCQ Review

    • Contusion equals temporary loss of neuro function? → False (that’s concussion)
    • Clear nasal CSF leak indicates basilar fracture? → True
    • Never massage immobile patient’s calves/thighs? → True (risk of thromboembolus)
    • Autonomic dysreflexia: which action is NOT appropriate? → Lowering to flat side-lying position is contraindicated; always sit up first.