Skeletal System
Anatomy & Physiology \n Summer SEMESTER 2022
Chapter 7.1: Introduction to the Skeletal System:
Components of the skeletal system:
Bones:
Primary organs of the skeletal system
Lightweight, but strong
Provide support for the body:
Keeps us upright
Provide protection:
Protects vital organs
Act as levers for movement:
It helps to make a coordinated effort to walk
Hematopoiesis: formation of blood cells
In red bone marrow : the center of the bone contains stem cells (progenitor cells) that help to produce WBC, RBC, and Platelets
Mineral Storage:
Calcium, phosphate
Cartilage:
A somewhat flexible, semi-rigid connective tissue
Covers the joint surface
Covers the growth plates
The growing tissue at the ends of long bones
Eventually, the cartilage is replaced by bone, and the lengthening of bones stops
Reduces Friction: allows bones to move against each other
Absorbs shock in moveable joints: cushions the force of the bones coming together
Ligaments: connects bone to bone
Help to attach one bone to the next helping to keep the bones in the right place
Tendons: connects muscle to bone
Allows for normal movement to occur
Joints:
Places of contact between:
Bones and Bones:
Ex: Vertebral disc are joints that allow the spine to move
For knees: there is a synovial cavity that contains synovial fluid (which allowed shock absorption and lubes the bones
Bones and cartilage:
An inverse relationship between mobility and stability
With joints that have more stability (skull and the spine), there is not a lot of mobility
With joints that have more mobility (knee, elbow) there is not a lot of stability
Chapter 7.2e: Microscopic Anatomy - Bone Connective Tissue:
Cells of bone:
Osteoprogenitor cells: beginning (come from stem cells in the bone marrow)
Bone stem cells
Can differentiate into osteocytes and osteoblasts
Osteoblasts: anabolic effect (building up)
Formed from osteoprogenitor stem cells
Synthesize and secrete semisolid organic bone matrix called osteoid
Become entrapped within the matrix
Osteoids and osteoblasts mix together to form the surface of the bone
After finishing secreting osteoid they Differentiate into osteocytes
Involved with building up bone
Osteocytes: cells
Mature bone cells derived from osteoblasts
Maintain bone matrix
Osteoclasts: catabolic effect (breaking down)
Large, multinuclear, phagocytic cells
Derived from fused bone marrow cells
A ruffled border increases the surface area exposed to bone
Involved in bone resorption
Bone Formation:
Begins with the secretion of the osteoid by osteoblasts
Osteoid contains collagen, glycoproteins, etc.
Calcification occurs, deposition of hydroxyapatite
Inorganic components of the bone matrix:
Calcium phosphate crystals (Ca3(PO4)2) and calcium hydroxide interact to form hydroxyapatite (Ca10(PO4)6(OH)2) crystals
Other substances incorporated into crystals
Calcium carbonate, sodium, magnesium, sulfate, fluoride
Hydroxyapatite hardens the matrix and accounts for the strength and rigidity of bones
Bone Formation:
Process requires
Vitamin D - enhances calcium absorption from the GI tract
Vitamin C - is required for collagen formation
Calcium and phosphate for calcification
Eventually, cartilage is replaced by bone, and growth stops
Bone resorption:
Bone matrix is destroyed by substance released from osteoclasts
Proteolytic enzymes released from lysosomes withing osteoclasts
Chemically digest organic matrix components
Freed calcium and phosphate ions enter the blood
Occurs when blood calcium levels are low
Increase bone resorption if blood calcium levels are low
This is not something you wish to speed up as low calcium levels will increase bone fragility
Chapter 7.5b: Bone Remodeling:
Bone Remodeling:
- Continuous removal of old bone and replacement with new bone
- Continues throughout adulthood
- Occurs at surfaces of a bone
- Occurs at different rates
- 20% of the skeleton is replaced yearly
- Dependent upon coordinated activities of osteoblasts, osteocytes, and osteoclasts
- Influenced by hormones
Chapter 7.5c: Hormones That Influence Bone Growth and Remodeling
Hormones:
- Molecules released from one cell into the blood
- Travel throughout the body to affect other cells
- Initiate specific cellular changes
- Some alter rates of osteoblast and osteoclast activity
- Affect bone composition and growth patterns
Growth Hormone:
Somatotropin - Growth hormone
Increase the growth of every organ/ bone in the body
Produced by the anterior pituitary gland
Increase at puberty
Stimulates the liver to produce the hormone, somatomedin
Both directly stimulate the growth of cartilage at the epiphyseal plate and bone elongation
Thyroid Hormone:
- Secreted by the thyroid gland
- Stimulates metabolic rate of osteoblasts: if there is not enough thyroid function then osteoblast creation is decreased (leading to soft bones)
Sex Hormones:
Estrogen and testosterone
Secreted in large amount at puberty
Dramatically accelerate bone growth
Stimulate osteoblasts and increase the rate of cartilage growth and bone formation in the epiphyseal plate
Glucocorticoids:
Group of steroid hormones
Released from the adrenal cortex
Reduce bone growth and bone thickness. Increase in bone fractures and bone fragility
High amounts increase bone loss
Impairs growth at the epiphyseal plate in children
Must monitor if the child receiving high doses of glucocorticoids.
Chapter 7.6: Regulating Blood Calcium Levels:
Regulating calcium concentration in blood is essential Calcium is required for:
- Initiation of muscle contraction
- Exocytosis of molecules from cells, including neurons
- Stimulation of the heart by pacemaker cells
- Blood clotting
Two primary hormones raise blood calcium:
- Calcitriol
- Parathyroid hormone
Chapter 7.6a: Activation of Vitamin D to Calcitriol:
UV light converts 7-dehydrocholesterol to vitamin D3 (cholecalciferol)
Released into the blood
Absorbed in the small intestine from diet
Vitamin D3 circulates throughout the body
Converted to calcidiol by liver enzymes
Both steps 1 & 2 occur continuously
Calcidiol circulates in the blood
Converted to calcitriol (the active form of Vitamin D3) by kidney enzymes
Parathyroid hormone (PTH) increases rate, so more calcitriol formed
Calcitriol stimulates the absorption of calcium ions from the small intestine into the blood and increased renal calcium reabsorption.
The more PTH there is the faster calcitriol is formed
Chapter 7.6b: Parathyroid Hormone and Calcitriol:
Parathyroid hormone (PTH):
- Secreted and released by parathyroid glands in response to reduced blood calcium levels
- Accelerates formation of calcitriol by kidney
PTH and calcitriol interact with major organs:
Bone:
- Act synergistically to increase osteoclasts activity and thus the resorption of bone matrix, releasing calcium into the blood
- PTH will digest our bones to raise calcium levels
Kidneys:
- Increase calcium reabsorption by the kidney
Small Intestine:
- Only calcitriol increases the absorption of calcium from the small intestine into the blood
Chapter 7.6c: Calcitonin:
Calcitonin:
Aids in regulating blood calcium levels
Less significant role than PTH or calcitriol
Released from the thyroid gland in response to high blood calcium levels
Inhibits osteoclast activity, thus less calcium is released into the blood (Promotes deposition of Ca2+ into bones)
Stimulates kidneys to increase the loss of calcium in the urine
Reducing blood calcium levels