Acute Injuries

Acute Joint Injuries
  • Types of Injuries

    • Traumatic injuries to joint structures:

    • Ligament damage

    • Meniscal injury

    • Labral injury

    • Cartilage damage

    • Bone damage

    • Long-term implication: osteoarthritis

  • Clinical Signs

    • Increased intra-articular fluid leads to joint effusion

    • Assessment Tests:

    • Milking test

    • Sweep test

    • Increased fluid indicates damage

  • Haemarthrosis

    • Presence of blood in the joint

Acute Articular Cartilage Injuries
  • Nature of Injury

    • Chondral fragments shear off articular surfaces due to high-impact trauma

  • Detection

    • MRI and CT scans used for detection

  • Surgical Intervention

    • Arthroscopic options: micro-fracturing

    • MRI is the gold standard for diagnosis

  • Repair Techniques

    • Mosaicplasty for cartilage repair

    • Link to osteoarthritis development

    • Poor long-term results in knee microfracture and ACI (Andia & Maffulli, 2018)

Acute Chondral Injuries
  • Characteristics and Causes

    • Result from extreme forces

    • Poor healing capacity due to limited regenerative ability

  • Statistics

    • Osteochondral fracture incidence in ACL injuries: up to 80%

    • Lateral patellar dislocation osteochondral fractures: 40%-78%

Acute Muscle Contusions
  • Definition and Causes

    • Result from forceful impacts or blunt trauma

    • Symptoms: pain, bruising (ecchymosis), edema, hematoma

  • Classification of Muscle Contusions by Severity

    • Mild "Cork":

    • RTP in 1-3 weeks

    • Soreness post-activity

    • Moderate "Cork":

    • RTP ~4 weeks

    • Minimal stiffness; up to 50% reduced range of motion

    • Severe "Cork":

    • RTP up to 8 weeks

    • Rapid swelling and significant movement loss

  • Bruising Progression

    • Initial red → Blue/purple/black (1-2 days) → Yellow/green (5-10 days) → Yellowish-brown (10-14 days)

  • Commonly Affected Areas

    • Vulnerable: vastus lateralis, biceps brachii

    • Less common: thigh adductors, medial gastrocnemius

Management and Treatment of Acute Muscle Contusions
  • Immediate Care Protocol

    • Use ICE, not HARM (Heat, Alcohol, Running, Massage)

    • Place muscle in a stretched position to aid healing

    • Avoid massage to limit bleeding

Myositis Ossificans Traumatica
  • Definition

    • Bone deposition in muscle during healing, typically 2-4 weeks post-injury

  • Mechanism

    • Fibroblasts differentiate into osteoblasts

    • Local factors: calcium reserve, hypoxia, vascular stasis

    • Systemic factors are unclear

  • Progression Timeline

    • Repeated injuries may exacerbate development

    • Time to development: 4 weeks to 12 months

Severe Complication: Compartment Syndrome
  • Case Study Example

    • Jarryn Geary had compartment syndrome post-quadricep strain

Exercise-Associated Muscle Cramps (EAMC)
  • Symptoms

    • Painful, spasmodic, involuntary contractions (1-3 mins)

    • Often occurs late in games or after exercise

  • Affected Muscle Groups

    • Gastrocnemius (calf), intrinsic foot muscles, quadriceps, hamstrings

  • Causes of EAMC

    • Not necessarily due to dehydration/electrolyte imbalance

    • Linked to altered neuromuscular control

    • No significant underlying diseases found

  • Management of EAMC

    • Effective Treatment:

    • Passive stretching for relief

    • Pickle juice may reduce cramp duration

Delayed Onset Muscle Soreness (DOMS)
  • Definition

    • Soreness after vigorous exercise, peaking 24-48 hours post-exercise

    • Associated with eccentric actions

  • Signs and Symptoms

    • Intense pain, local swelling, stiffness, strength deficits, elevated plasma creatine kinase levels

  • Management of DOMS

    • Modify exercise, cryotherapy, stretching, active recovery, turmeric for inflammation reduction

    • Repeated eccentric exercises may protect against future DOMS incidents

  • Proposed Pathway for DOMS

    • Bradykinin release during eccentric exercises upregulates NGF leading to muscle hyperalgesia

    • Cyclooxygenase-2 (COX-2) activation stimulates nociceptors, developing an adaptive mechanism to reduce future incidences.