Basal Cell Carcinoma and Squamous Cell Carcinoma Study Notes
Overview of Skin Cancers
Focus on basal cell carcinoma (BCC) and squamous cell carcinoma (SCC)
Learning Objective: Describe risk factors, pathophysiology, and clinical manifestations of BCC and SCC
Classification of Skin Cancers
Skin cancers classified into two categories:
Non melanoma skin cancers (NMSC): 70% BCC, 30% SCC
Melanoma skin cancer
Pathophysiology of Skin Cancers
Common factor: High levels of UV light exposure
**BCC Specific Factors:
Chemical exposure
SCC Specific Factors:
Human Papillomavirus (HPV) infection
Melanoma Specific Factors:
Familial gene mutation (inherited mutations)
Cellular Origins of Skin Cancers
Cell Types Affected:
Basal Cell Carcinoma (BCC): DNA mutation in stratum basale cells (stem cells)
Squamous Cell Carcinoma (SCC): DNA mutation in epidermal keratinocytes
Key concept: Disordered replication leads to DNA mutations, resulting in dysplasia and eventual neoplasia
Differences Between BCC and SCC
Aggressiveness:
BCC – least aggressive
SCC – more aggressive, potential to metastasize
Skin Lesions: Both types present as skin lesions
Basal Cell Carcinoma (BCC)
Definition: Most common skin tumor (70% of NMSCs)
Anatomy & Growth Patterns:
Occurs in sun-exposed skin, predominantly on the head and neck
Grows slowly and rarely metastasizes
Visible Types:
Superficial:
Morphology: Red, defined lesions, macular, sometimes pearly appearance
Nodular:
Morphology: Translucent papule or nodule, possible small dilated vessels, sometimes ulcerated and bled
Morphoeic:
Morphology: Pale, sclerotic lesion, waxy/white/yellowish, invasive, requires aggressive treatment
Pathophysiology Details:
Initiated by excessive UV exposure, leading to DNA damage of pluripotent stem cells
Role of Tumor Suppressor Genes:
Approximately 50% of BCCs involve mutations in tumor suppressor genes, obstructing DNA repair and apoptosis
Common Risk Factors:
Light skin, red hair, blue eyes, freckles, older age
Clinical Features/Manifestations:
Elevated nodule with central depression, possible tissue destruction (less than a rodent ulcer)
Diagnosis Methods:
Observation and biopsy to confirm BCC
Management Tactics:
Surgical excision, particularly challenging for morphoeic types due to undefined borders
Squamous Cell Carcinoma (SCC)
Definition: Invasive malignant tumor arise from stratum spinosum epithelial cells
Development: Often preceded by actinic keratosis (thickened, scaly patches due to UV exposure)
Key Risk Factors:
Primary factor: Sun exposure
HPV infection (certain strains)
Immunosuppression, vitamin D deficiency
Etiology:
Risk factors that may also contribute to tumor initiation
Actinic keratosis can progress to SCC but not all do
Gene mutations (tumor suppressor genes p53, notch1, notch2, RAS)
HPV plays a role in 25% of SCC cases, inhibiting DNA repair and promoting survival of mutated cells
Pathophysiology Details:
Excessive UV exposure causes alterations in keratinocyte signaling, leading to oxidative stress and subsequent mutations
Clinical Features/Manifestations:
Persistent non-healing ulcer, scaly crust, possible bleeding, localized invasion
Diagnosis Methods:
Biopsy to distinguish SCC from other forms of skin cancer (BCC, melanoma)
Management Tactics:
Surgical excision usually performed in outpatient settings; may also require radiation or chemotherapy based on patient history and biopsy results
Conclusion
Understanding risk factors, pathophysiology, and clinical manifestations of BCC and SCC is crucial for effective diagnosis and management.