Urogenital Tract Infections Lecture Notes
Urogenital Tract Infections
Lecture Aims
- Describe the anatomy of the urogenital tract.
- Describe innate immunity mechanisms of the urogenital tract.
- Describe different infections of the urinary tract.
- Causative organisms, epidemiology, risk factors.
- Describe a number of genital tract infections including:
- Causative organisms, epidemiology, risk factors.
- Bacteria, viruses, fungi, protozoa.
Why People Get Infectious Diseases
- Factors include physical, social, behavioral, cultural, political, and economic factors.
Urinary Tract Anatomy
- Upper urinary tract:
- Ureters and kidneys
- Ureters drain urine through vesicoureteral valves to the bladder.
- Lower urinary tract:
- Bladder and urethra
- Urine is voided through the urethra via the urethrovesical valve.
- The urethra is colonized by a range of organisms.
Is Urine Sterile?
- Historically thought of as sterile.
- Now known to have its own microbiome based on 16s sequencing data.
Infections of the Urinary Tract
- Infection can occur at several sites within the urinary tract:
- Urinary tract infection (UTI) or cystitis: Bladder (common); ascending or descending; complicated or uncomplicated
- Pyelonephritis: Kidney; acute or chronic
- Prostatitis (enlarged prostate): Prostate; can cause chronic intermittent UTI in males
- Urethritis: Urethra; not really a UTI; normally associated with STIs (e.g., gonorrhoea)
Ascending UTI
- Colonization: Pathogen colonizes the periurethral area and ascends through the urethra upwards towards the bladder.
- Uroepithelium penetration:
- Fimbriae allow bladder epithelial cell attachment and penetration.
- Following penetration, bacteria continue to replicate and may form biofilms.
- Ascension:
- Once sufficient bacterial colonization occurs, bacteria may ascend on the ureter towards the kidney.
- Fimbriae may aid in the ascension process.
- Bacterial toxins may also play a role by inhibiting peristalsis (reducing the flow of urine).
- Pyelonephritis: Infection of the renal parenchyma causes an inflammatory response called pyelonephritis.
- While infection of the renal parenchyma is usually the result of bacterial ascension, it can also occur from hematogenous spread.
- If the inflammatory cascade continues, tubular obstruction and damage occur, leading to interstitial edema.
- This may lead to interstitial nephritis, causing acute kidney injury (AKI).
Ascending UTI - Causes
- Bacteria in urethral opening ascend urethra, leading to bladder infection.
- Can result from:
- Fecal soiling or contamination (more common in children and incontinent adults).
- Incomplete emptying of the bladder:
- Bacteria in bladder are normally voided in urine or removed by the immune system.
- Incomplete emptying may leave a "reservoir" of organisms.
- Urethrovesical reflux: Reflux of urine (from urethra to bladder).
- Prostatic seeding: Chronic prostate infection; intermittent UTIs in males.
- Instrumentation: Urethral organisms enter bladder via instruments (catheterization).
UTI - Causes
- Descending UTI:
- Pathogen travels via blood (hematogenous spread) or lymph from a source elsewhere in the body.
- Organisms enter the bladder via the kidney.
- Main organisms:
- S. aureus
- Candida albicans
- Mycobacterium tuberculosis
- Uncommon.
UTI - Risk Factors
- Uncomplicated UTI:
- No urinary tract defects, renal impairment, or comorbidities.
- Afebrile, non-pregnant, immunocompetent.
- Risk factors:
- Female gender (comparatively short urethra increases risk).
- Sexual activity increases risk.
- Menopause (linked to decline in estrogen).
- Complicated UTI:
- Patients not described above.
- Risk factors:
- Altered immune status: diabetes or pregnancy.
- Calculi within the urinary tract.
- Urinary tract abnormality (congenital or acquired).
- Urinary catheterization.
Organisms Responsible for UTI
- Uncomplicated UTI:
- UPEC (Uropathogenic E. coli): 75 \%
- K. pneumoniae: 6 \%
- S. saprophyticus: 6 \%
- Enterococcus spp.: 3 \%
- GBS: 3 \%
- P. mirabilis: 2 \%
- P. aeruginosa: 1 \%
- S. aureus: 1 \%
- Risk factors:
- Female gender
- Older age
- Younger age
- Complicated UTI:
- UPEC: 65 \%
- K. pneumoniae: 11 \%
- Enterococcus spp.: 7 \%
- Candida spp.: 8 \%
- P. aeruginosa: 2 \%
- P. mirabilis: 2 \%
- S. aureus: 2 \%
- GBS: 1 \%
- Risk factors:
- Indwelling catheters
- Immunosuppression
- Urinary tract abnormalities
- Antibiotic exposure
Genital Tract Infections
- Affect any gender.
- May or may not be sexually transmitted.
- Sexually transmitted infection (STI) includes both:
- Asymptomatic infection
- Symptomatic disease
- Female genital tract microbiome is protective:
- Dominated by Lactobacillus species.
- Create mildly acidic environment (pH < 4.5).
- Produce hydrogen peroxide, lactic acid, bacteriocins.
- Activates complement.
Overview of Genital Tract Infections - Bacterial
- Syphilis:
- Organism: Treponema pallidum
- Gonorrhoea:
- Organism: Neisseria gonorrhoeae
- Chlamydia:
- Organism: Chlamydia trachomatis
- Bacterial vaginosis:
- Organisms: Gardnerella vaginalis, anaerobes
- MG:
- Organism: Mycoplasma genitalum
- Donovanosis:
- Organism: Klebsiella granulomatis
- Chancroid:
- Organism: Haemophilus ducreyi
- Puerpural sepsis:
- Organisms: Various (e.g., Streptococcus pyogenes)
- Septic abortion:
- Organisms: Various (e.g., Clostridium)
Overview of Genital Tract Infections - Viral, Fungal, Protozoal
- Viral
- Genital herpes:
- Organism: Herpes simplex virus
- Human papilloma virus:
- Organism: Human papilloma virus
- Fungal
- Protozoal
- Trichomoniasis:
- Organism: Trichomonas vaginalis
- HIV is sexually transmitted, but does not actually cause a genital tract infection (covered in a later lecture).
Burden of Disease (USA)
- 2018: in USA estimated 26 million new STIs.
- Almost 16 billion in costs.
- Syphilis in newborns still increasing compared to previous years.
- Not all STIs are reportable – many are under-reported.
Gonorrhoea
- Infection caused by Neisseria gonorrhoeae
- Gram-negative; fastidious.
- Obligate human pathogen.
- Facultative intracellular pathogen.
- Transmitted sexually or during childbirth (vertical transmission).
- Gonococcal ophthalmia an issue in developing countries.
- Organism infects mucous membranes
- Range of symptoms or asymptomatic.
- May clinically resemble Chlamydia infection.
- Diagnosed by nucleic acid amplification test (NAAT) +/- culture.
- Infection still relatively common due to two important factors:
- Large reservoir of asymptomatic carriers, particularly females.
- Increasing (and alarming) resistance to antibiotics.
Gonorrhoea Symptoms
- Males: asymptomatic or mild symptoms
- Most common symptom is acute urethritis.
- Second common symptom is urethral discharge / pus.
- Symptoms appear one week after infection.
- If untreated: epididymitis, infertility (rare).
- Females: most remain asymptomatic
- Primary site of infection is the endocervix.
- If untreated: may spread to uterus or fallopian tubes.
- Leads to pelvic inflammatory disease.
- Complication: scarring of fallopian tubes; infertility.
- Rectal and oropharyngeal gonorrhoea can occur
- Most common in men who have sex with men (MSM).
Chlamydia
- Infection caused by Chlamydia trachomatis
- Gram negative-like, coccoid bacteria.
- Obligately intracellular (can't synthesize ATP).
- Infection also known as non-gonococcal urethritis (NGU) or non-specific urethritis (NSU).
- Most common STI in many countries.
- In Australia in 2021:
- 86,916 chlamydia notifications compared to
- 26,577 gonorrhoea notifications
- Most cases remain undiagnosed and untreated.
- Diagnosed by NAAT or antigen testing.
Chlamydia Symptoms
- Males: asymptomatic (\sim 50 \%. or mild symptoms
- Pain on urination, discharge
- Females: asymptomatic (\sim 70 \%.)
- Often NGU is untreated
- Chronic infection may lead to
- Pelvic inflammatory disease
- Scarring and infertility
- Increased risk of ectopic pregnancy
- May increase risk of cervical cancer
- Possibly in association with human papillomavirus
- Vertical transmission:
- Can cause pneumonia in infants born to infected mothers
Syphilis
- Infection caused by Treponema pallidum
- Transmitted sexually or vertically
- Sexually transmitted only ~20 \%. of the time (inefficient)
- Organism historically could not be cultured (until 2018)
- Diagnosis is largely by serology
- Culture now possible but growth is extremely slow
- Three disease stages: primary, secondary, latent, tertiary
Syphilis - Stages
- Primary stage syphilis
- Initial sign is small, hard-based chancre (sore)
- Painless ulcer
- Appears at infection site
- Lesions are highly infectious
- Lesions disappear after two weeks
- Bacteria enter bloodstream and lymphatic system:
- Distributes organisms throughout body (systemic infection)
- Secondary stage syphilis
- Skin rashes, caused by circulating immune complexes
- Rash also present in mucous membranes of mouth, throat, cervix
- Lesions also present, patient highly infectious
- Latent syphilis
- No symptoms, stage may last for years
- Tertiary stage syphilis
- Occurs in 15-30 \%. of untreated people
- Most symptoms due to immune reaction to surviving spirochaetes
- Neurosyphilis may occur
- Occurs in 10 \%. patients if untreated
- Personality changes, dementia (paresis), seizures, paralysis, loss of sight, hearing
Syphilis - Congenital & Outbreaks
- Congenital syphilis
- Organism transmitted across placenta
- Range of outcomes depending on how long mother has had syphilis
- Pregnancy in primary or secondary stage likely to result in stillbirth
- Still a major global issue
- Ongoing syphilis outbreak in Australia
- Started in 2011 in QLD
- 2013 started in NT, 2014 WA, 2016 SA
- 2016-2022: 69 cases of congenital syphilis
- 18 resulted in death of the infant
Bacterial Vaginosis
- Most common cause of vaginitis symptoms
- Vaginitis: inflammation, itching, pain
- Symptoms:
- Loose, fishy-odour discharge
- Irritation
- Not sexually transmitted
- Characterized by:
- Substantial depletion of commensal Lactobacillus population
- Increased mix of anaerobic bacteria
- Commensal bacteria that are normally present at low levels
- Includes Gardnerella vaginalis
- Gram-variable coccobacillus
BV Diagnosis
- Diagnosis is by microscopy
- Wet mount: look for characteristic clue cells
- Epithelial cells studded with tiny cocco-bacilli
- Routine culture not valuable
- Other diagnostic features
- High vaginal pH (>4.5)
- Positive Amines test
- In contrast with Candida infection
Viral Infections of the Urogenital Tract
- Genital herpes
- Usually caused by Herpes simplex virus type 2 (HSV-2)
- HSV-1 (usually causes cold sores or blisters) can also cause genital herpes
- Symptoms:
- Burning sensation, pain on urination
- Lesions:
- Appear after incubation period of 1 week
- Fluid within vesicles contain virions:
- Highly infectious
- Virus is transmissible
- Heal after about two weeks
- Disease is also transmitted when no lesions or symptoms are apparent
- Virus is shed through mucosa
Genital Herpes - Latency
- Virus enters lifelong latent state in nerve cells
- Like cold sores (HSV1) and chickenpox virus (Varicella; herpesvirus 3)
- Recurs in >80 \%. of HSV2 cases
- Recurs more in men than women
- Various triggers such as stress, illness (with fever), scratching, menstruation
- Neonatal herpes: virus can cross placenta
- May cause abortion, developmental delay, deafness
- Worse if mother acquires herpes (primary infection) during pregnancy
- No cure: use suppression or management
- Acyclovir alleviates symptoms of primary outbreak
Genital Warts
- Caused by papillomaviruses
- More than 60 serotypes of HPV
- Specific types linked to genital warts (e.g. HPV16)
- Symptoms:
- Infection is usually asymptomatic; no symptoms in 9/10 cases
- Symptoms (females and males) can include:
- Warts/bumps on genitals, groin, anus
- Genital pain, itching, discomfort
- Incubation period is a few weeks to months
- Diagnosed by:
- Examination: appearance, biopsy or Pap test
- DNA test: detects DNA from some of the more likely cancer-causing HPV
Genital Warts - Treatment and Prevention
- Treat lesions topically with cryotherapy or gels
- E.g. Imiquimod gel stimulates body to produce interferon
- But link to cervical cancer is the greatest danger
- Women: cervical cancer
- Men: penile cancer
- Vaccine (Gardasil) against strains associated with cancer
- “Gardasil 9”, 2-3 doses
- HPV types 6, 11, 16, 18, 31, 33, 45, 52 and 58
- Cervarix
Fungal Infections
- Candidiasis or Thrush
- Second most common cause of vaginitis symptoms
- First is bacterial vaginosis
- Caused mainly by C. albicans but also other Candida species
- Symptoms in females:
- Pruritus (itching)
- Discharge: may be slight but usually thick and ‘curd like’
- Symptoms in males:
- Balanitis: inflammation of the head of the penis
- Pruritis, inflammation, swelling
- Not sexually transmitted
- Evidence does not support treating partner with antifungals
- Candida: pH normal (4.5), discharge thick, Amines neg
- BV: pH high (>4.5), discharge thin, Amines pos
Candidiasis - Risk Factors & Diagnosis
- Risk factors for infection are:
- Recent antibiotic use (disrupts bacterial microbiome)
- High oestrogen levels
- E.g. pregnancy, oral contraceptives, pre-menstrual
- Uncontrolled diabetes
- Immune system dysfunction
- 20-30 \%. of women carry Candida in the genital tract
- Usually in low numbers, asymptomatic
- Diagnosis
- Microscopy: examine vaginal swab by wet mount
- Culture
Protozoan Diseases
- Trichomoniasis
- Caused by anaerobic protozoan Trichomonas vaginalis
- Asymptomatic in ~50 \%. of women and ~80 \%. of men
- Symptoms in females:
- Discharge copious “yellow-green frothy”
- Vaginal soreness
- Pain on urination
- Diagnosis
- Wet mount: positive ~70 \%. of cases
- Culture
- Vaginal pH usually elevated