Eating Disorders
behavioral disorder→ dont have to do with mood, have to do with behavior
anorexia nervosa
very brief history
12th and 13th century religious themes to starvation
kosher, lent, fasting holidays
St. Catherine of siena 12th century
16th century: no longer accepted by catholic church
17th century: english physician Richard Morton in 1689 gave the first clear medical description → “wasting” disease
Lasegue(1873) and Gull(1874) recognized the physiological aspects of the condition
in 1973, Hilde Bruch published eating disorders: obesity, anorexia nervosa, and the person within
anorexia nervosa: restriction of caloric intake PLUS distorted self-image PLUS intense fear of gaining weight PLUS lack of recognition of seriousness(always below normal weight regardless of subtype)
subtypes
restricting (most familiar) → caloric restriction
binge eating/purging→ caloric conscious but sometimes has periods of binge eating and purging (over exercising or vomiting to counteract the binge eating)
who is at risk
anyone whose job involves appearance
physical consequences: all symptoms associated with starvation
characteristic behavior:
calorie counting
checking weight (several times a day)
excuses/distractions
epidemiology: varies from 0.9% of females to 0.3% of males (possible as high as 5% to 20% of the general population)
chronic relapsing → may come back and go away (not one and done)
eating disorders→ highest morbidity rate of any psychiatric disorder
5-10% death rate within 10 years
18-20% death rate within 20 years
reasons: caloric malnutrition → weakened immune system
comorbidity: depression and anxiety
Bullimia Nervosa
core symptoms
behavior→ purging
thoughts → feeling out of control, impulsive
within 10% of normal weight (unlike anorexia who are always underweight)
recurrent inappropriate compensatory behaviors after binging (1000s of calories)
purging(throwing up)
excessive exercise
fasting
frequency:
DMS: average of once a week for 3 months
subtypes:
purging (most common)
vomiting, laxatives, enemas
nonpurging (6%-8%)
excess exercise, fasting
binge eating disorder: was considered for research in DSM 4→ added in the DSM 5
eating in a discrete period of time an amount of food that is definitely larger than what most people would eat in a similar period of time
a sense of lack of control
report of distress over binge eating
3 or more symptoms
eating rapidly
eating past the point of feeling full
eating large amounts of food when not physically hungry
eating alone due to embarrassment
feeling disgusted with oneself, depressed, or guilty
severity dependent no frequency of eating binges
1/3 of the people who seek medical treatment for obesity
controversial
diagnosing the normal
other eating disorders(RARE)
pica: eating non organic substances
chalk, glue, paper
a lot of young kids → eating rocks
sign of malnutrition or intellectual disability
rumination disorder: someone regurgitates what they ate, chew and then swallow it
avoidant/restrictive food intake disorder: as an adult is very restrictive on what food they eat (more intense than just a picky eat)
excessive to the point where individual becomes malnourished
example: someone who only eats dominos pizza and nothing else
eating disorder possible causes:
biological factors
genetics
risk of anorexia from relatives is 11.4 times greater than normal controls
risk of bulimia for relatives is 3.7 times higher than healthy controls
genome-wide association for anorexia nervosa on chromosome 12, which is associated with type 1 diabetes
brain abnormalities
anorexia: ventromedial hypothalamus (homeostasis and appetite)
damage to frontal and temporal cortex seem to be linked to the development of anorexia nervosa in some cases and bulimia nervosa and others
set points:
our bodies have a well-established tendency to resist variation from some sort of biologically determined set point or weight that our individual bodies try to “defend”
serotonin(found in brain but mostly in stomach)
neurotransmitter serotonin is implicated in obsessionality, mood disorders, and impulsivity; also modulate appetite and feeding behavior
active area of research
reward sensitivity
new research centers on brain pathways and neurotransmitters (such as dopamine) involved in reward processing
theory that reward and punishment systems get contaminated
normally rewarding stimuli such as food become aversive
stimuli associated with self-starvation become valued
the garcia effect
sociocultural factors
the media and publications exert influence in defining the culture’s “ideal body shape” and creating pressures to be thin. AKA the “thin ideal”
socioeconomic status: eating disorders more common among middle to upper class people
family influences
more than 1/3 of patients reported that family dysfunction contributed to the development of anorexia
parents overly preoccupied with thinness, dieting, and good physical appearance
associated family behaviors include rigidity, parental overprotectiveness, excessive control, and marital discord (family systems theory)
more sick the child is, the less the parents fight and more stable the family becomes (unconscious)
rigidity and control
bulimic family characteristics include:
high parental expectations
perceiving one’s family to be less cohesive (less control)
psychodynamic
personality based factors (very difficult personality between each disorder)