Eating Disorders

behavioral disorder→ dont have to do with mood, have to do with behavior

anorexia nervosa

  • very brief history

    • 12th and 13th century religious themes to starvation

      • kosher, lent, fasting holidays

    • St. Catherine of siena 12th century

    • 16th century: no longer accepted by catholic church

    • 17th century: english physician Richard Morton in 1689 gave the first clear medical description → “wasting” disease

    • Lasegue(1873) and Gull(1874) recognized the physiological aspects of the condition

    • in 1973, Hilde Bruch published eating disorders: obesity, anorexia nervosa, and the person within

anorexia nervosa: restriction of caloric intake PLUS distorted self-image PLUS intense fear of gaining weight PLUS lack of recognition of seriousness(always below normal weight regardless of subtype)

  • subtypes

    • restricting (most familiar) → caloric restriction

    • binge eating/purging→ caloric conscious but sometimes has periods of binge eating and purging (over exercising or vomiting to counteract the binge eating)

  • who is at risk

    • anyone whose job involves appearance

  • physical consequences: all symptoms associated with starvation

  • characteristic behavior:

    • calorie counting

    • checking weight (several times a day)

    • excuses/distractions

  • epidemiology: varies from 0.9% of females to 0.3% of males (possible as high as 5% to 20% of the general population)

  • chronic relapsing → may come back and go away (not one and done)

  • eating disorders→ highest morbidity rate of any psychiatric disorder

    • 5-10% death rate within 10 years

    • 18-20% death rate within 20 years

    • reasons: caloric malnutrition → weakened immune system

      • comorbidity: depression and anxiety

Bullimia Nervosa

  • core symptoms

    • behavior→ purging

    • thoughts → feeling out of control, impulsive

    • within 10% of normal weight (unlike anorexia who are always underweight)

  • recurrent inappropriate compensatory behaviors after binging (1000s of calories)

    • purging(throwing up)

    • excessive exercise

    • fasting

  • frequency:

    • DMS: average of once a week for 3 months

  • subtypes:

    • purging (most common)

      • vomiting, laxatives, enemas

    • nonpurging (6%-8%)

      • excess exercise, fasting

binge eating disorder: was considered for research in DSM 4→ added in the DSM 5

  • eating in a discrete period of time an amount of food that is definitely larger than what most people would eat in a similar period of time

  • a sense of lack of control

  • report of distress over binge eating

  • 3 or more symptoms

    • eating rapidly

    • eating past the point of feeling full

    • eating large amounts of food when not physically hungry

    • eating alone due to embarrassment

    • feeling disgusted with oneself, depressed, or guilty

  • severity dependent no frequency of eating binges

  • 1/3 of the people who seek medical treatment for obesity

  • controversial

    • diagnosing the normal

other eating disorders(RARE)

  • pica: eating non organic substances

    • chalk, glue, paper

    • a lot of young kids → eating rocks

      • sign of malnutrition or intellectual disability

  • rumination disorder: someone regurgitates what they ate, chew and then swallow it

  • avoidant/restrictive food intake disorder: as an adult is very restrictive on what food they eat (more intense than just a picky eat)

    • excessive to the point where individual becomes malnourished

    • example: someone who only eats dominos pizza and nothing else

eating disorder possible causes:

biological factors

  • genetics

    • risk of anorexia from relatives is 11.4 times greater than normal controls

    • risk of bulimia for relatives is 3.7 times higher than healthy controls

    • genome-wide association for anorexia nervosa on chromosome 12, which is associated with type 1 diabetes

  • brain abnormalities

    • anorexia: ventromedial hypothalamus (homeostasis and appetite)

    • damage to frontal and temporal cortex seem to be linked to the development of anorexia nervosa in some cases and bulimia nervosa and others

  • set points:

    • our bodies have a well-established tendency to resist variation from some sort of biologically determined set point or weight that our individual bodies try to “defend”

  • serotonin(found in brain but mostly in stomach)

    • neurotransmitter serotonin is implicated in obsessionality, mood disorders, and impulsivity; also modulate appetite and feeding behavior

    • active area of research

  • reward sensitivity

    • new research centers on brain pathways and neurotransmitters (such as dopamine) involved in reward processing

    • theory that reward and punishment systems get contaminated

      • normally rewarding stimuli such as food become aversive

      • stimuli associated with self-starvation become valued

      • the garcia effect

sociocultural factors

  • the media and publications exert influence in defining the culture’s “ideal body shape” and creating pressures to be thin. AKA the “thin ideal”

  • socioeconomic status: eating disorders more common among middle to upper class people

family influences

  • more than 1/3 of patients reported that family dysfunction contributed to the development of anorexia

    • parents overly preoccupied with thinness, dieting, and good physical appearance

    • associated family behaviors include rigidity, parental overprotectiveness, excessive control, and marital discord (family systems theory)

      • more sick the child is, the less the parents fight and more stable the family becomes (unconscious)

    • rigidity and control

  • bulimic family characteristics include:

    • high parental expectations

    • perceiving one’s family to be less cohesive (less control)

psychodynamic

  • personality based factors (very difficult personality between each disorder)