Anxiety
ANXIETY INTRODUCTION
Definition of Anxiety:
An anxiety is a universal human experience, categorized as one of the most fundamental emotions.
It encompasses feelings of apprehension, uncertainty, or dread, resulting from a real or perceived threat.
Normal Anxiety:
Considered a healthy reaction; it serves as an essential evolutionary strategy for survival.
ANXIETY PATHOPHYSIOLOGY
Key Mediators in Central Nervous System:
Norepinephrine
Serotonin
Dopamine
Gamma-Aminobutyric Acid (GABA)
Role of the Autonomic Nervous System:
Mediates most symptoms of anxiety; particularly the sympathetic nervous system.
Causes of Anxiety Disorders:
Caused by the interaction of several factors, including genetic vulnerability.
Factors interact with situations, stress, or trauma, leading to clinically significant syndromes.
ANXIETY PATHOPHYSIOLOGY - Rule Out Medical Causes
Necessary Steps for Diagnosis:
Table 15.7 (see Psych Workbook for details).
Rule out other causes that may include:
Caffeine-containing beverages
Over-the-counter (OTC) medicines
Herbal medications
Street drugs
Basic Laboratory Studies:
Complete blood cell count
Chemistry profile
Thyroid function tests
Urinalysis
Urine drug screening
ANXIETY LEVELS OF ANXIETY
Peplau’s Levels of Anxiety:
Mild
Moderate
Severe
Panic
ANXIETY LEVELS OF ANXIETY - Mild Anxiety
Characteristics:
Normal occurrence in daily life; allows clear perception of reality.
Enhances information processing and problem solving.
Physical Symptoms:
Slight discomfort, restlessness, or minor tension-relieving behaviors, e.g., nail biting, foot or finger tapping, fidgeting.
ANXIETY LEVELS OF ANXIETY - Moderate Anxiety
Characteristics:
Perceptual field narrows; details are often excluded from attention.
Exhibits selective inattention where only certain environmental aspects are focused on.
Cognitive Function:
Clear thinking is hampered; suboptimal learning and problem solving can occur.
Physical Symptoms:
Activation of sympathetic nervous system, increased pulse, increased respiratory rates, perspiration, gastric discomfort, headache, and urinary urgency.
ANXIETY LEVELS OF ANXIETY - Severe Anxiety
Characteristics:
Greatly reduced perceptual field, focusing on one specific detail or scattered details.
Excludes most external stimuli; learning and problem solving become impossible.
Physical Symptoms:
Pounding heart, trembling, hyperventilation, feelings of impending doom, headache, nausea, and dizziness.
ANXIETY LEVELS OF ANXIETY - Panic Anxiety
Characteristics:
Most extreme anxiety level leading to highly dysregulated behavior and loss of reality.
Could involve pacing, running, screaming, or total withdrawal.
Physical Symptoms:
Include racing heart, sweating, chills, hot flashes, trembling, shortness of breath, weakness, dizziness, tingling or numbness in hands, headache, nausea, abdominal cramping, chest pain.
DEFENSE MECHANISMS
Definition:
Psychological strategies employed unconsciously to protect individuals from anxiety generated by unacceptable thoughts or feelings.
ANXIETY RISK FACTORS
General Risk Factors:
White race
Female gender
Family history of anxiety or Major Depressive Disorder (MDD)
Disturbed family environment
Early parental loss
Childhood sexual abuse
Substance use disorder
Traumatic events
Lower educational attainment
Low self-esteem
ANXIETY RISK FACTORS - Specific Types
Vary by Anxiety Disorder Type:
Shyness or distress in new situations during childhood
Exposure to stress or negative life events
Family history of anxiety or mental disorders.
ANXIETY SIGNS & SYMPTOMS
Physical and Psychological Symptoms:
Restlessness, feeling wound-up or on-edge
Easily fatigued
Difficulty concentrating
Irritability
Somatic complaints such as headaches, muscle aches, stomach aches
Difficulty controlling worry
Sleep problems, including difficulty with sleep onset or maintenance.
ANXIETY DESIRED OUTCOMES
Objective Goals:
Experience decreased anxiety
Demonstrate effective coping with anxiety
Show improved knowledge of treatment plans.
ANXIETY INTERVENTION GUIDELINES
Treatment Approaches:
Counseling, milieu therapy, promotion of self-care activities
Pharmacological and biological interventions
Health education interventions.
Techniques:
Relaxation methods like progressive relaxation.
Identification of community resources for specialized treatment.
Support groups for patients and families specific to anxiety disorders.
ANXIETY GENERAL INTERVENTIONS TO DECREASE ANXIETY
Strategies:
Foster a calm environment by reducing stimuli.
Provide reassurance of support.
Assist in calming anxiety from severe or panic to mild with slow deep breathing or prescribed medication.
Encourage discussion about feelings and concerns.
Reframe problems into solvable formats.
Identifying triggers of anxiety prior to onset.
ANXIETY GENERAL INTERVENTIONS TO DECREASE ANXIETY - Techniques
Teaching Methods:
Teach relaxation techniques such as deep breathing, meditation, or progressive muscle relaxation.
Identify and address negative self-talk.
Refer patients and significant others to support groups and self-help programs.
ANXIETY GENERAL INTERVENTIONS TO IMPROVE COPING
Recommendations:
Monitor positive coping skills and encourage new alternatives to ineffective ones.
Explain the fight-or-flight response and how controlled breathing can invoke a relaxation response.
Teach visualization and other relaxation techniques.
ANXIETY GENERAL INTERVENTIONS TO IMPROVE COPING - Techniques
Cognitive Behavioral Techniques:
Keep focus on manageable, clearly defined problems.
Utilize role-play for stressful situations.
Introduce biofeedback techniques through apps or wearables.
ANXIETY NON-PHARMACOLOGIC TREATMENTS
Behavioral Therapy Techniques:
Modeling
Systematic Desensitization
Flooding
Thought Stopping
Cognitive-Behavioral Therapy (CBT):
Exposure and Response Prevention specifically, with further details in OCD-related content.
ANXIETY PATIENT EDUCATION
Teaching Strategies:
Provide verbal/written material on diagnosis, symptoms, etiology, and treatment options.
Explain anxiety as a result of dysfunctional appraisal and automatic thinking.
Teach behavioral techniques including thought stopping.
Role-play coping strategies for anxiety-provoking situations.
Encourage journals to track anxiety triggers and strategies utilized, rating anxiety from 1 to 10.
ANXIETY HEALTH PROMOTION
Suggestions for Improvement:
Prioritize sleep, exercise (aerobic), and reduction of caffeine.
Incorporate music, pets, and massage as part of stress management.
ANXIETY MEDICATIONS
Review for Pharmacological Management:
Key medication classes include:
SSRIs (Selective Serotonin Reuptake Inhibitors)
SNRIs (Serotonin-Norepinephrine Reuptake Inhibitors)
Benzodiazepines
Buspirone
Tricyclic Antidepressants
Utilize Lehne’s Pharmacology for detailed study and ATAT sections in the Psych Workbook for each medication.
GENERALIZED ANXIETY DISORDER (GAD) INTRODUCTION
Definition:
GAD is one of the most prevalent mental disorders characterized by persistent, excessive, and unrealistic worry about everyday issues such as finances, family, health, and future.
Associated with feelings of being overwhelmed.
GAD ETIOLOGY
Commonly Associated Factors:
Female gender
Being unmarried
Poor health or chronic medical conditions
Low education levels
Presence of stressors
Genetic predisposition (25% likelihood if first-degree relative has GAD)
Environmental factors, including child abuse
Substance abuse
GAD DSM-V CRITERIA
Diagnostic Criteria (according to the DSM-5):
Excessive anxiety and worry for at least six months
Difficulty controlling the worry.
Association with three or more symptoms for at least six months:
Restlessness or feeling keyed up
Easily fatigued
Difficulty concentrating or mind going blank
Irritability
Muscle tension
Sleep disturbances
Resulting distress must significantly impair social and occupational functioning
Not attributable to a physical cause
GAD ASSESSMENT
Challenges with Diagnosis:
Diagnosis can be complex due to common presentation of somatic symptoms rather than psychological symptoms.
Symptoms:
Common somatic complaints include breathlessness, palpitations, fatigue, headaches, dizziness, and restlessness.
Psychological complaints often include vague anxiety, emotional lability, concentration difficulties, and insomnia.
Initial Assessment Focus:
Assess for behavioral or somatic symptoms.
Consider psychosocial stressors, developmental issues, and past medical history (including trauma and substance abuse).
Conduct evaluations to rule out organic causes:
Thyroid function tests
Blood glucose level
Echocardiography
Toxicology screen.
GAD-7 Questionnaire:
A screening tool to help identify and monitor patients with GAD.
GAD RULE OUT MEDICAL CAUSES
Medical Conditions to Consider:
Hyperthyroidism
Pheochromocytoma
Chronic Obstructive Pulmonary Disease (COPD)
Transient Ischemic Attack (TIA)
Epilepsy
Bipolar disorder
Caffeine or stimulant use
GAD INCREASES RISK OF:
Complications:
Development or worsening of other conditions such as:
Depression (often comorbid)
Insomnia
Drug or alcohol misuse
Gastrointestinal problems
Social isolation
Impaired functioning in work or school
Poor quality of life
Potential for suicide.
GAD TREATMENT
Primary Treatment Approaches:
Cognitive Behavioral Therapy and pharmacotherapy are the two main treatments, often utilized together.
Cognitive Behavioral Therapy (CBT):
Involves psychoeducation, restructuring maladaptive thought patterns, and gradual exposure to anxiety-provoking situations.
Pharmacotherapy:
Utilizes various medications for treatment.
GAD TREATMENT - Pharmacotherapy
Antidepressants:
SSRIs and SNRIs are first-line treatments, including:
Escitalopram (Lexapro)
Duloxetine (Cymbalta)
Venlafaxine (Effexor XR)
Paroxetine (Paxil).
Benzodiazepines:
Examples include diazepam and clonazepam for short-term symptom alleviation.
Caution advised in patients with a history of substance abuse due to dependency risks.
Buspirone:
Non-benzodiazepine option, not inducing dependency and with a slower onset (2-3 weeks).
Antipsychotics:
May benefit patients with associated behavioral issues.
GAD TREATMENT - Medication Management
Administration Protocol:
All medications need gradual titration and continued use for a minimum of 4 weeks to evaluate efficacy.
After symptom stabilization, medications should be taken for at least 12 months before tapering.
Monitoring for Adverse Effects:
Potential side effects including weight gain, hyperlipidemia, and diabetes must be monitored closely.
GAD TREATMENT - Patient Education
Importance of Education:
Educating patients on triggers of anxiety is crucial (e.g., caffeine, alcohol, nicotine, managing stress, and optimizing sleep).
Complementary and Alternative Remedies:
Various options exist, but efficacy is often unproven.
Some agents (e.g., Kava) may harm the liver; others (e.g., St. John's Wort) might interact with SSRIs leading to serotonin syndrome.
GAD PROGNOSIS
Overall Outlook:
Prognosis is often guarded due to factors like medication non-compliance driven by cost and side effects.
Relapses are frequent; patients often seek out alternative therapies that may not yield success.
Quality of life for affected individuals remains a concern.