Chapter 24
Pharmacology: An Introduction - Chapter 24: Antianginal Drugs
Learning Outcomes
24.1 Types and Causes of Angina Pectoris
Describe the main types of angina pectoris and the usual cause of each type.
24.2 Nitroglycerin Mechanism of Action
Discuss the mechanism of action of the nitrate drugs and compare routes of administration and onset of action of the different nitrate preparations.
24.3 Beta-Blockers in Angina Treatment
Explain the mechanism of action of beta-blockers in the treatment of angina and coronary artery disease (CAD).
24.4 Pharmacologic Effects of Calcium Channel Blockers
Recognize the pharmacologic effects produced by the different calcium channel blockers.
24.5 Preferred Therapies for Angina
Discuss the preferred therapies for the different types of angina.
Nitrates
General Effects
Produce a general vasodilation of systemic veins and arteries.
Administered:
During angina attacks to relieve pain.
On a daily basis to prevent angina attacks.
Mechanism of Action
Nitric oxide relaxes vascular smooth muscle.
Vasodilation decreases both preload and afterload on the heart.
Clinical Use:
Sublingual Nitroglycerin:
Relieves acute angina attacks.
Nitroglycerin Ointment (2%):
Helps prevent the occurrence of angina.
Nitroglycerin Extended-Release Capsules:
Used daily as a form of prophylaxis.
Transdermal Nitroglycerin (Nitro-Dur, Minitran):
Used in the prevention of angina.
Intravenous Infusion of Nitroglycerin:
Used during emergency and surgical situations.
Adverse Effects:
Cutaneous flushing
Dizziness
Headache
Weakness
Fainting
Sudden drops in blood pressure can cause reflex tachycardia.
Patient Education:
Patients must carry the prescribed medication at all times.
Tablets must be stored in light-resistant, airtight containers.
Individuals experiencing angina should sit down and place a nitroglycerin tablet under the tongue.
Awareness of side effects is critical for effective management.
Drug Interactions:
Nitrate drugs should not be combined with drugs treating erectile dysfunction.
This combination can cause excessive vasodilation, hypotension, reflex tachycardia, and potentially lead to myocardial infarction.
Calcium Channel Blockers
Mechanism of Action:
Inhibit the influx of calcium ions into vascular smooth muscle.
Enable arteriolar vasodilation and reduce blood pressure.
These are administered daily to prevent angina and are preferred for treating variant or Prinzmetal angina, and to relieve vasospasm.
Drugs with Direct Cardiac Effects:
Verapamil:
Used to treat supraventricular arrhythmias and angina.
Diltiazem:
Possesses greater vasodilating effects compared to verapamil.
Adverse Effects:
Headache
Facial flushing
Dizziness
Hypotension
Minor gastrointestinal disturbances
Overdose Symptoms for Verapamil and Diltiazem:
Bradycardia
Atrioventricular (AV) block
Decreased myocardial contraction
Possible heart failure
Contraindications:
The use of calcium channel blockers is contraindicated with beta-blockers due to risk of excessive cardiac depression and heart failure.
Ranolazine (Ranexa)
Overview:
Ranolazine is a piperazine antianginal agent.
It exerts antianginal and anti-ischemic effects without altering heart rate or blood pressure.
Indicated for chronic angina pectoris as a maintenance medication.
Adverse Effects:
Dizziness
Constipation
Headache
Nausea
QTc prolongation
Preferred Therapy for Angina Pectoris
Exertional, Fixed, or Stable Angina:
Preferred treatments include nitrates, beta-blockers, and calcium channel blockers.
Variant or Prinzmetal Angina:
Preferred treatments include calcium channel blockers and nitrates.
Unstable Angina:
Managed with antiplatelet and anticoagulant therapy, along with lifestyle changes to provide better patient outcomes.