Eating Disorders: Anorexia & Bulimia Nervosa - Comprehensive Notes

Eating Disorders: Anorexia & Bulimia Nervosa

Overview
  • Eating disorders are characterized by irregular eating habits and extreme distress or concern about body weight or shape.

  • These disorders involve inadequate or excessive food intake, which can harm a person’s well-being.

  • Common forms include anorexia nervosa, bulimia nervosa, binge-eating disorder, pica, rumination disorder, avoidant/restrictive food intake disorder (ARFID), and other specified feeding or eating disorder (OSFED).

What are Eating Disorders?
  • Eating disorders involve repeated disturbances of eating or eating-related behavior, resulting in altered food consumption or absorption.

  • These disturbances significantly diminish physical health or psychosocial functioning.

  • Continuum of Eating Disorders:

    • Anorexia nervosa: Eating too little or self-starvation.

    • Bulimia nervosa: Chaotic eating patterns.

    • Obesity: Eating too much.

  • Historical Context:

    • Middle Ages: Documentation of willful dieting leading to self-starvation in female saints.

    • Late 1800s: Doctors described young women using self-starvation to avoid obesity.

    • 1960s: Anorexia nervosa established as a mental disorder.

    • 1979: Bulimia nervosa described as a distinct syndrome.

Types of Eating Disorders
  • Anorexia Nervosa:

    • Life-threatening disorder characterized by refusal or inability to maintain a minimally normal body weight.

    • Intense fear of gaining weight or becoming fat.

    • Significantly disturbed perception of body shape or size.

    • Inability or refusal to acknowledge the seriousness of the problem.

  • Bulimia Nervosa:

    • Recurrent episodes (at least twice a week for 3 months) of binge eating followed by inappropriate compensatory behaviors to avoid weight gain.

    • Compensatory behaviors include purging, fasting, or excessive exercising.

  • Binge-Eating Disorder (BED):

    • Recurrent episodes of binge eating without the recurrent use of inappropriate compensatory behaviors.

    • Does not occur exclusively during bulimia nervosa or anorexia nervosa.

    • No compensatory methods for overeating, such as self-induced vomiting.

  • Pica:

    • Persistent eating of non-nutritive substances (e.g., hair, dirt, paint chips) for at least one month.

  • Rumination Disorder:

    • Repeatedly and persistently regurgitating food after eating.

    • Not due to a medical condition or another eating disorder.

  • Avoidant/Restrictive Food Intake Disorder (ARFID):

    • Persistent failure to meet appropriate nutritional or energy needs.

    • Due to lack of interest in eating, aversion to sensory characteristics of food, or fear of choking.

  • Other Specified Feeding or Eating Disorder (OSFED):

    • Eating behaviors that cause clinically compelling distress and impairment in functioning.

    • Do not meet the full criteria for other feeding and eating disorders.

Causes
  • A specific cause for eating disorders is unknown; dieting may be the initial stimulus.

  • Biologic Factors:

    • Studies indicate that anorexia nervosa tends to run in families.

    • Genetic vulnerability may result from personality type or susceptibility to psychiatric disorders.

  • Developmental Factors:

    • Onset of anorexia nervosa often occurs during adolescence or young adulthood.

    • Causes may be related to developmental issues.

  • Family Influences:

    • Girls growing up amid family problems and abuse are at higher risk for anorexia and bulimia.

    • Disordered eating can be a response to family discord.

  • Sociocultural Factors:

    • Adolescents idealize actresses and models with the “perfect” body, even if they are underweight or use special effects.

    • Pressure from others contributes to eating disorders.

Statistics and Incidences
  • Obesity is a major health problem in the United States.

  • Millions of women either starve themselves or engage in chaotic eating patterns leading to death.

  • 30% to 35% of normal-weight people with bulimia have a history of anorexia nervosa and low body weight.

  • About 50% of people with anorexia nervosa exhibit bulimic behavior.

  • More than 90% of cases of anorexia nervosa and bulimia occur in females (American Psychiatric Association, 2000).

  • The prevalence of both eating disorders is estimated to be 1% to 3% of the general population in the United States.

Clinical Manifestations
  • Symptoms of Anorexia Nervosa:

    • Fear of gaining weight or becoming fat, even when severely underweight.

    • Body image disturbance.

    • Amenorrhea or absence of menstrual period.

    • Depressive symptoms (e.g., depressed mood, social withdrawal, irritability, insomnia).

    • Preoccupation with thoughts of food.

    • Feelings of ineffectiveness.

    • Inflexible thinking.

    • Strong need to control environment.

    • Limited spontaneity and overly restrained emotional expression.

    • Complaints of constipation and abdominal pain.

    • Cold intolerance.

    • Lethargy.

    • Emaciation.

    • Hypotension, hypothermia, bradycardia.

    • Hypertrophy of salivary glands.

    • Elevated BUN.

    • Electrolyte imbalances.

    • Leukopenia and mild anemia.

    • Elevated liver function studies.

  • Symptoms of Bulimia Nervosa:

    • Recurrent episodes of binge eating.

    • Compensatory behavior such as self-induced vomiting, misuse of laxatives, diuretics, enemas, or excessive exercise.

    • Self-evaluation overly influenced by body shape and weight.

    • Usually within normal weight range, possible underweight or overweight.

    • Restriction of total calorie consumption between binges, selecting low-calorie foods while avoiding foods perceived as fattening or likely to trigger a binge.

    • Depressive and anxiety symptoms.

    • Possible substance use involving alcohol and stimulants.

    • Loss of dental enamel.

    • Chipped, ragged, or moth-eaten appearance of teeth.

    • Increased dental caries.

    • Menstrual irregularities.

    • Dependence on laxatives.

    • Esophageal tears.

    • Fluid and electrolyte abnormalities.

    • Metabolic alkalosis (from vomiting) or metabolic acidosis (from diarrhea).

    • Mildly elevated serum amylase levels.

Assessment and Diagnostic Findings
  • Diagnostic Tests and Assessment Cues:

    • Physical and mental status evaluation.

      • Physiological condition (weight, vital signs, physical complications).

      • Psychological well-being (body image, mood disturbances, cognitive distortions).

    • Complete blood count (CBC).

      • Hemoglobin levels typically normal, elevations in dehydration.

      • White blood cell count (WBC) typically low due to increased margination.

      • Thrombocytopenia is also observed.

    • Blood chemistries.

      • Hyponatremia (excess water intake or inappropriate secretion of antidiuretic hormone).

      • Hypokalemia (diuretic or laxative use).

      • Hypoglycemia (lack of glucose precursors or low glycogen stores; impaired insulin clearance).

      • Elevated blood urea nitrogen (renal function normal except in dehydration).

      • Hypokalemic hypochloremic metabolic alkalosis (vomiting).

      • Acidosis (laxative abuse).

    • Liver function tests.

      • Minimally elevated, albumin and protein levels usually normal.

Medical Management
  • Medical management focuses on weight restoration, nutritional rehabilitation, rehydration, and correction of electrolyte imbalances.

  • Nutritional rehabilitation and weight restoration.

    • Clients receive nutritionally balanced meals and snacks that gradually increase caloric intake to a normal level for size, age, and activity.

  • Family-based therapy.

    • Individuals with anorexia nervosa may respond best to family-based treatment, also known as the Maudsley method, an established therapeutic modality for achieving and maintaining remission from anorexia nervosa.

  • Cognitive behavioral therapy (CBT).

    • CBT is an evidence-based, effective treatment for bulimia nervosa (BN); behavioral approaches to avoiding undesirable eating habits are used, including diary keeping; behavioral analyses of the antecedents, behaviors, and consequences (so-called ABCs) associated with binge eating and purging episodes; and exposure to food paired with progressive response prevention regarding binge eating and purging.

  • Interpersonal psychotherapy.

    • Interpersonal psychotherapy (IPT) addresses specific issues in the interpersonal arena that create the context for and stimulate dynamic tensions that spur the patient’s symptoms; these generally encompass such processes as grief, role transitions, role conflicts or disputes, and interpersonal deficits.

Pharmacologic Management
  • Several classes of drugs have been studied, but few have shown clinical success.

  • Electrolyte supplements.

    • Electrolyte repletion is necessary in patients with profound malnutrition, dehydration, and purging behaviors; repletion may be done orally or parenterally, depending on the patient’s clinical state.

  • Fat-soluble vitamins.

    • Vitamins are used to meet necessary dietary requirements. They are utilized in metabolic pathways, as well as in deoxyribonucleic acid (DNA) and protein synthesis.

  • Antidepressants, SSRIs.

    • These agents have been reported to reduce binge eating, vomiting, and depression and to improve eating habits, although their impact on body dissatisfaction remains unclear.