ACS Risk Factors and ECG Interpretation
ACS Risk Factors
Important to recognize risk factors for ACS (Acute Coronary Syndrome).
Age:
Women over 55.
Men over 45.
High blood pressure or high cholesterol.
Diabetes: increases risk of vascular disease due to vessel narrowing.
Unhealthy lifestyle.
Family history of ACS.
ACS Presentation
Classic symptoms:
Chest pain.
Jaw pain.
Atypical symptoms:
No chest pain can also be a symptom.
Nausea.
Weakness.
Silent MI: symptoms that aren't chest pain related.
Levine's sign: clutching chest.
Pertinent Negatives for ACS
Shortness of breath.
Nausea.
Angina
Chest pain or discomfort when the heart doesn't get enough oxygen.
Symptom, not a disease.
Usually caused by CHD (Coronary Heart Disease) or CAD (Coronary Artery Disease).
Types:
Stable Angina:
Chest pain upon exertion that resolves with rest.
Predictable.
Usually indicates 70-85% blockage in coronary artery.
Unstable Angina:
Does not resolve with rest or nitroglycerin.
Considered ACS.
Chest pain at rest indicates about 90% blockage.
Chest pain after nitroglycerin usually indicates nearly 100% blockage.
Prinzmetal/Variable Angina:
Blood vessel spasms, causing narrowing.
Can occur at rest.
May resolve with nitro or on its own.
Vitals with ACS
Vitals can vary widely.
Tachycardia or bradycardia.
Hypertension or hypotension.
Do not rule out ACS based solely on vitals.
ECG and ACS
An EKG never rules out ACS. It only rules it in.
EKGs are only about 50% sensitive.
Non-MI conditions can mimic MIs on an ECG.
EKGs are highly specific (if it shows STEMI, it's likely there).
EKGs can identify MIs quickly, unlike labs like troponin.
EKGs can reveal other complications or blockages.
ST Segment Elevation
Identify J point (end of QRS complex).
Measure one small box over from the J point to assess ST segment elevation.
Measurements that matter:
1 mm elevation is significant in all leads except V1, V2, and V3.
In V1, V2, V3, at least 2 mm of elevation is needed.
Elevation must be present in two or more related leads (anatomically).
STEMI
If elevation is only present in lead two need additional confirmation in lead AVF.
Related Leads
v1, v2 is the Septum.
v3, v4 is the anterior.
ECG Limitations and Benefits
Limitations:
Only about 50% sensitive (may miss some MIs).
Non-MI conditions can mimic MIs.
Benefits:
Highly specific (if STEMI is present, it's likely accurate).
Quick identification compared to lab tests.
Can identify other complications.
I See All Leads
Inferior (II, III, aVF).
Septal (V1, V2).
Anterior (V3, V4).
Lateral (I, aVL, V5, V6).
Reciprocal Changes
ST depression in some leads when ST elevation is present in others.
Not present in septal or anterior MIs.
Lateral STEMI often has inferior reciprocal changes.
Inferior STEMI often has lateral reciprocal changes.
Posterior involvement:
May see T-wave inversion in V1-V3.
Consider with inferior STEMI.
Checking for Right Ventricular Involvement
Move V4 lead to the right side of the chest (V4R).
Elevation in V4R indicates right ventricular infarction.
Hold nitroglycerin if right ventricular involvement is present.
Posterior Involvement
Move V5 and V6 leads to beneath the scapula (V8 and V9).
Elevation there suggests posterior involvement.
15-Lead ECG
Combine standard 12-lead with V4R, V8, and V9.
Fluid Administration
In general, avoid fluid boluses in STEMI patients unless hypotensive.
Inferior MI Considerations
About 50% of MIs are inferior.
Posterior and/or right ventricular involvement is common.
Hold nitro if right ventricular involvement is present.
Pathological Q Waves
Downward deflection at the start of the QRS complex.
Indicates past infarction (dead tissue).
Pathological if more than one small box wide or more than one-third the height of the R wave.
ST segment elevation with pathological Q waves indicates ongoing injury and death.
Location of Q waves indicates the area of tissue death.