Lecture 34: Glycogen
Glycogen Metabolism and Glucose Homeostasis
Lecturer: Dr. Katrine Wallis
Email: Katrine.wallis@warwick.ac.uk
Office: D134
Glycogen Structure and Function
Definition: Glycogen is a highly branched polymer of glucose residues.
Functions:
Serves as a store of glucose to maintain blood glucose levels.
Provides energy generation in muscles.
Storage Details:
Stored as insoluble granules:
Liver: up to 10% w/w (~100g)
Skeletal Muscle: up to 2% w/w (~400g)
Note: Remember the structure of glycogen from earlier lectures.
Glycogen and its Metabolic Pathways
Glycogen Interconversion:
Key Molecules:
Glycogen → Glucose-1-P → Glucose-6-P → Glucose (export to blood)
Supports various metabolic pathways:
Glycolysis
Pentose phosphate pathway
Enzymes involved:
Glycogen phosphorylase
Phosphoglucomutase
Glucose-6-phosphatase
Importance of Glycogen
Need for Glycogen:
Fatty acids cannot be metabolized anaerobically.
Blood glucose must be maintained for brain function.
Animals cannot convert fatty acids to glucose.
Glyoxylate Pathway
Applicability: Only occurs in plants and bacteria.
Process: Converts citrate to acetyl-CoA, producing glucose and fatty acids.
Glycogen Structure
Glycogen Molecular Structure:
Consists of:
a-1,6 linkages (branching)
a-1,4 linkages (linear).
Note: Two types of glycosidic bonds exist within glycogen.
Glycogen Breakdown Process
Phosphorolytic Cleavage:
Catalyzed by glycogen phosphorylase, producing glucose-1-phosphate and glycogen with reduced residues.
Saves one ATP molecule in energy expenditure.
Glycogen Degradation Steps
Cleavage Process:
Cleaves a-1,4 glycosidic bonds until four residues are left.
Transferase moves three residues to the other branch.
α-1,6 glycosidase removes the branch point residue.
Glycogen Synthesis
Energy Requirement:
Glycogen synthesis requires energy input through coupling to UTP cleavage.
Key Enzymes:
UDP-glucose pyrophosphorylase for synthesis.
Glycogen Primer and Branching
Glycogen Primer:
Composed of four or more α-1,4 linked glucose residues attached to tyrosine in glycogenin.
Branching Enzyme:
Attaches a-1,6 linkages to the glycogen molecule.
Andersen's Disease: Caused by lack of branching enzyme, leading to liver failure.
Glycogen-Storage Diseases
Types of Diseases:
I. Von Gierke Disease - Glucose 6-phosphatase deficiency.
II. Pompe Disease - Lysosomal a-1,4-glucosidase deficiency.
III. Cori Disease - Debranching enzyme deficiency.
IV. Andersen Disease - Branching enzyme deficiency.
V. McArdle Disease - Phosphorylase deficiency.
VI. Hers Disease - Liver phosphorylase deficiency.
VII. Phosphofructokinase deficiency.
VIII. Phosphorylase kinase deficiency.
Note: Most are inherited as autosomal recessives; Type VIII is sex-linked.
Glycogen Metabolism Regulation
Metabolism Control: Glycogen phosphorylase and glycogen synthase are reciprocally regulated by hormones.
Hormonal Regulation:
Breakdown: Glucagon and adrenaline.
Synthesis: Insulin.
Enzyme Regulation:
Phosphorylation and allosteric effectors regulate enzyme activity based on energy states.
Reciprocal Regulation Mechanism
Regulatory States:
Activity switch: Same signal can have opposite effects on glycogen synthesis and breakdown.
Protein kinase A is fundamental in this signaling pathway.
Phosphorylase States
Phosphorylase Forms:
Exists as relaxed (active) and tense (inactive) state.
Regulation by phosphorylation shifts conformational states regulating activity.
Energy Status Regulation
Muscle Phosphorylase:
Allosterically activated by AMP, inhibited by ATP and G-6-P based on available energy status.
Allosteric Regulation in Liver
Phosphorylase Regulation:
Glucose shifts the equilibrium to inactive T state, preventing further glycogen breakdown when glucose is sufficient.
Isozymes of Phosphorylase
Isozymes Defined:
Multiple forms of the same enzyme with different structures and regulation mechanisms.
Glucose Homeostasis
Organ Coordination:
Maintains glucose levels through organ collaboration to regulate blood glucose concentrations based on energy needs.
Pancreatic Regulation of Blood Glucose
Hormone Actions:
High blood glucose triggers insulin secretion (promotes glucose uptake).
Low blood glucose triggers glucagon secretion (promotes glycogen breakdown).
Mobilization of Glucose During Fasting
Hormonal Responses:
Glucagon and epinephrine released during stress, exercise mobilizing glucose for energy via glycogenolysis and gluconeogenesis.
The Cori Cycle
Process:
Involves conversion of lactate back to glucose in the liver from pyruvate generated in muscles.
Importance of the Cori Cycle
Application: Critical in high-energy demanding situations like marathon running where muscles rely on aerobic respiration more than on the Cori cycle for ATP.
Summary of Glycogen Metabolism
Key Points:
Hormonal regulation is crucial for maintaining glucose levels.
Glycogen synthesis and breakdown are reciprocally regulated to prevent futile cycling.
Understanding these pathways is essential for grasping energy metabolism in the body.