Infections of the Skin

Types of Skin Lesions

  • Macules: Flat lesions with a change in skin color.
  • Papules: Raised, solid lesions less than 5 mm in diameter.
  • Plaques: Flat, elevated lesions larger than 5 mm in diameter (plateau-like).
  • Nodules: Rounded, raised lesions greater than 5 mm in diameter.
  • Urticaria (Wheals/Hives): Annular (ring-like) papules or plaques, pinkish in color.
  • Vesicles: Circumscribed, fluid-filled lesions less than 5 mm in diameter.
  • Bullae: Circumscribed, fluid-filled lesions greater than 5 mm in diameter.
  • Pustules: Circumscribed lesions filled with exudate.
  • Purpura: Skin lesions caused by bleeding into the skin.
    • Petechiae: Purpura less than 3 mm in diameter.
    • Ecchymosis: Purpura greater than 3 mm in diameter.
  • Ulcer: Crater-like lesion involving epidermis and dermis.
  • Eschar: Necrotic ulcer covered by a blackened scab.

Bacterial Skin Infections

Staphylococci: Staphylococcus aureus

  • Important Properties:
    • Common human pathogen.
    • Gram-positive cocci, arranged individually, in pairs, short chains, or grape-like clusters.
    • Found on skin and in nasopharynx.
    • Culture: Gray to golden yellow colonies (best at 20°C–25°C).
    • Coagulase-positive (differentiates from other Staphylococcus species).
    • Catalase-positive (differentiates from Streptococci).
    • Produces enzymes and toxins, responsible for invasiveness and pathogenicity.
  • Mode of Transmission:
    • Direct contact with purulent lesions
    • Hands of healthcare workers
    • Fomites (bed linens, contaminated clothing)
  • Clinical Findings:
    • Folliculitis: Pyogenic infection involving hair follicle; localized painful inflammation; heals rapidly after pus drainage.
    • Furuncle (Boil): Extension of folliculitis; larger, painful nodules with necrotic tissue.
    • Carbuncle: Coalescence of furuncles extending into subcutaneous tissue with multiple sinus tracts.
    • Sty (Hordeolum): Folliculitis at the base of eyelids.
    • Impetigo: Common in young children; involves face and limbs; begins as a flattened red spot (macule) that becomes a pus-filled vesicle, ruptures, and forms a honey-colored crust; caused by S. aureus and S. pyogenes.
    • Staphylococcal Scalded Skin Syndrome (Ritter’s disease): Primarily in newborns and young children; sudden perioral erythema spreading across the body within two days; positive Nikolsky sign (skin displacement with slight pressure); bullae and cutaneous blister formation followed by desquamation; exfoliativetoxin-mediated; no scarring.
  • Laboratory Diagnosis:
    • Microscopic examination: Gram-stained specimen (gram-positive cocci).
    • Culture: Gray to golden yellow colonies.
    • Catalase-positive and coagulase-positive.
  • Treatment and Prevention:
    • Beta-lactam antibiotics (e.g., penicillin; however, resistance is common).
    • Oxacillin is effective against S. aureus.
    • Incision and drainage of localized infections.

Staphylococcus epidermidis

  • Part of normal skin flora.
  • Associated with stitch abscesses, UTIs, and endocarditis.
  • Causes infections in individuals with prosthetic devices.

Streptococci: Streptococcus pyogenes

  • Gram-positive cocci arranged in pairs or chains.
  • Group A Streptococci based on Lancefield classification.
  • Beta-hemolytic (complete hemolysis of blood).
  • Major virulence factor: M protein (anti-phagocytic).
  • Produces enzymes and toxins responsible for pathogenesis.
  • Some strains are encapsulated, protecting them from phagocytosis and potentially causing severe systemic infections.
  • Mode of Transmission:
    • Direct contact with infected persons.
    • Fomites.
  • Clinical Findings:
    • Pyoderma (Impetigo): Localized purulent skin infection involving face and extremities; vesicles progress to pustules, rupture, and form honey-colored crusts; regional lymph node enlargement may occur without systemic infection.
    • Erysipelas (St. Anthony’s Fire): Follows respiratory tract or skin infection; localized raised areas with pain, erythema, and warmth; distinct from normal skin; lymphadenopathy and systemic manifestations are present.
    • Cellulitis: Involves skin and subcutaneous tissue; less demarcated borders than erysipelas; local inflammation with systemic signs.
    • Necrotizing Fasciitis (Flesh-Eating Disease/Streptococcal Gangrene): Deep subcutaneous tissue infection; starts as cellulitis, becomes bullous and gangrenous, spreads to fascia, muscle, and fat; may become systemic and cause multi-organ failure and death.
  • Complications:
    • Acute glomerulonephritis (more commonly with skin infections).
    • Rheumatic fever (usually associated with throat infection).
  • Laboratory Diagnosis:
    • Microscopy: Gram stain shows gram-positive cocci in pairs and chains, associated with leukocytes.
    • Culture: Positive beta-hemolysis on blood agar.
    • Bacitracin test: (+) zone of inhibition of growth around the Bacitracin disc.
  • Treatment and Prevention:
    • Penicillin is the drug of choice.
    • Macrolides (erythromycin, azithromycin) or cephalosporins for penicillin-allergic patients.
    • Drainage of pus and thorough debridement of infected tissues.

Pseudomonas aeruginosa

  • Gram-negative bacilli arranged in pairs and encapsulated.
  • Produces water-soluble pigments (e.g., pyocyanin – blue).
  • Opportunistic pathogen; common cause of nosocomial infections; resistant to many antibiotics.
  • Virulence factors: adhesins (flagella, pili, LPS, alginate), toxins (exotoxin A, pigments), and enzymes.
  • Mode of Transmission:
    • Colonization of previously injured skin.
  • Clinical Findings:
    • Colonization of burn wounds with blue-green pus and sweet, grape-like odor.
    • Folliculitis.
    • Secondary infections in acne and nail infections (from contaminated water).
    • Osteochondritis (inflammation of bone and cartilage) following penetrating injury.
  • Laboratory Diagnosis:
    • Gram stain: gram-negative bacilli arranged individually or in pairs.
    • Culture: flat colonies with green pigmentation and sweet, grape-like odor.
    • Oxidase test: positive.
  • Treatment and Prevention:
    • Culture and sensitivity testing.
    • Preventing contamination of sterile hospital equipment and cross-contamination of patients.

Clostridium perfringens

  • Gram-positive bacilli, anaerobic, rarely produce endospores.
  • Produces four lethal toxins (alpha, beta, iota, epsilon); alpha toxin is a lecithinase causing lysis of erythrocytes, platelets, and leukocytes, as well as massive hemolysis, bleeding, and tissue destruction.
  • Widely distributed in nature, associated with soil and water contaminated with feces.
    • Often seen with soil and water.
  • Mode of Transmission:
    • Colonization of skin following physical trauma or surgery.
  • Clinical Findings:
    • Soft tissue infections: cellulitis, suppurative myositis, and myonecrosis (gas gangrene).
    • Gas gangrene: life-threatening infection following physical trauma or surgery; massive tissue necrosis with gas formation, shock, renal failure, and death within two days of onset.
  • Laboratory Diagnosis:
    • Microscopic detection of gram-positive bacilli in pairs.
    • Growth in culture under anaerobic condition.
  • Treatment and Prevention:
    • Surgical wound debridement.
    • High-dose penicillin therapy.

Bacillus anthracis

  • Gram-positive bacilli arranged individually, in pairs, or long serpentine chains (