Care of Patients with Arthritis and Total Joint Arthroplasty
Concepts in Arthritis and Total Joint Arthroplasty
Priority Concepts:
Mobility: The ability to move freely and easily, which can be significantly impaired in individuals with arthritis.
Immunity: Understanding the role of immune responses in joint inflammation and potential infections associated with joint diseases.
Interrelated Concepts:
Pain: Chronic pain is a key symptom of arthritis, affecting quality of life.
Clotting: Increased risks of thromboembolic events in patients with limited mobility.
Infection: Potential complications following joint surgeries such as Total Joint Arthroplasty (TJA).
Inflammation: Central to the pathology of arthritis, particularly in autoimmune forms.
Mobility Concept Exemplar: Osteoarthritis
Definition:
Osteoarthritis (OA) is the most prevalent form of arthritis, characterized by the progressive degeneration of cartilage, resulting in bone-on-bone contact in one or more joints, ultimately leading to decreased mobility.
Pathophysiology:
The condition is driven by a complex interplay of mechanical stress and biological factors, where the enzymatic breakdown of the articular matrix leads to the formation of osteophytes (bone spurs).
Inflammatory cytokines like Interleukin-1 (IL-1) exacerbate the breakdown process of cartilage.
The disintegration of cartilage can lead to bone fragments floating in the joint, creating a pathological condition characterized by crepitus, or a grating sound during movement.
results in joint pain and stiffness can lead to decreased mobility and muscle atophy
Etiology of Osteoarthritis
Primary OA:
Often associated with aging and genetic predisposition.
Particularly prevalent in weight-bearing joints (such as hips and knees), shoulders, vertebral column, and hands are most commonly affected, because they are used most often or bear the mechanical stress of the body weight and many years of use
Secondary OA:
joint injury and obesity
repetitive stress to joints increases risk; heavy manual occupations like carpet laying, construction, and farming)
football players, runners, gymnasts, amateur athletes
metabolic disease (diabetes mellitus, Paget disease)
blood disorders (hemophilia, sickle cell disease)
Prevalence:
Affects a significant population, most notably those over 60 years of age, with an increased incidence in women, particularly after menopause.
It is recognized as the 5th most common cause of disability globally, impacting daily functioning and quality of life.
Osteoarthritis Health Promotion and Wellness
Nutrition: Emphasize a balanced diet along with adequate vitamins and minerals which can help maintain joint health and reduce inflammation. Foods rich in omega-3 fatty acids, antioxidants, and anti-inflammatory properties should be prioritized.
Injury Prevention: Encourage protective measures during activities or sports to prevent joint injuries.
Activity Management: Incorporate regular rest periods and ergonomic modifications at work to minimize strain on affected joints.
Physical Activity: Advocate for low-impact exercises, such as swimming or cycling, which can enhance mobility without exacerbating joint pain.
Joint Changes in Degenerative Joint Disease
Bone Hypertrophy: The presence of bone spurs that develop due to abnormal growth and repair processes in response to cartilage degradation.
Cartilage Loss: Progressive deterioration of cartilage in affected joints is a hallmark of OA.
Cartilage Debris: Identification of cartilage particles within joints can lead to increased inflammation and further joint dysfunction.
Interprofessional Collaborative Care
History Taking:
age (typical onset 60yrs), weight, injury, occupation, sports, family history
physical assessment/clinical manifestations
joint pain and stiffness
similar to RA in early disease
mostly seen in middle-aged or older woman
Clinical Manifestations:
Key symptoms include persistent joint pain and stiffness that typically lessen with rest but worsen with physical activity, particularly after periods of inactivity.
later pain may occur with slight motion or even when at rest
pt may report joint stiffness that usually lasts less than 30 mins after a period of inactivity
crepitus (a coarse grating sound caused by loosened bone and cartilage) may be felt or heard as the joint goes through a range of motion.
Heberden’s nodes (distal interphalangeal, DIP):

bouchard’s nodes (proximal interphalangeal, PIP)
joint effusions: excess joint fluid is common when the knees are inflamed
atrophy of skeletal muscle: the disease can discourage movement of the painful joints, which may result in contractures, muscle atrophy, and further pain
Assessment Methods
Psychosocial Assessment:
Recognizing that chronic pain can lead to psychological challenges, increasing risks for depression and anxiety, is essential for holistic care.
Laboratory Tests:
Blood tests like (ESR) and high-sensitivity C-reactive protein (hsCRP) as well as joint fluid aspiration can provide crucial diagnostic insights.
Imaging Techniques:
Use of X-rays can help visualize changes in bone structure, while MRI and CT studies provide more detailed images of soft tissue and cartilage degeneration.
Analysis of Osteoarthritis
Priority Problems:
Continuous chronic pain attributed to inflammation and joint degeneration leading to significant discomfort and lifestyle limitations.
There is a potential for decreased mobility due to joint-associated pain, stiffness, and resultant muscle atrophy over time.
Managing Chronic Pain
Nonsurgical Management:
Drug Therapy:
Acetaminophen (Tylenol) is typically the first-line treatment for pain management.
standard ceiling dose is 4000 mg each day
pt are at risk for liver damage if taking more than 3000 mg daily, have alcoholism or liver diseases
older pt are at risk because of normal changes of aging such as slowed excretion of drug metabolites
topical
lidocaine 5% (lidoderm), buspirone hCL (Buspar), otc preparation
teach pt to apply the patch on clean, intact skin for 12 hrs each day (up to 3 patches can be applied to a painful joint), but watch for skin irritation.
lidocaine patch is contraindicated in pt taking class 1 antidysrhythmics
NSAID
Cox-2 nonselective (celecoxib)
before beginning drug therapy get baseline lab (CBC, CMP,)
don’t give to pt with hypertension, kidney disease, or cardiovascular disease (can cause MI, and hypertension)
GI side effects, bleeding, acute kidney injury with long-term use.
tkae with food
adverse reactions: dark, tarry stools; SOB, edema, frequent dyspepsia, hematemesis (bloody vomitus) and changes in urine output.
Hyaluronic Acid (lubricating synthetic joint fluid)
contraindicated in obesity, severe OA, over 65, previous injection
cyclobenzaprine HCL (flexeril)
Other pain management approaches:
Balancing periods of rest and exercise to prevent worsening of symptoms, utilizing thermal modalities (heat/cold therapy), maintaining weight (encourage weightloss)
positioning: when pt is in a supine position (recumbent), use of small pillow under the head or neck but avoid the use of other pillows.
integrative therapies (these supplements are not recommended)
glucosamine may decrease inflammation
chondroitin plays a role in strengthening cartilage
Surgical Management:
Total Joint Arthroplasty (TJA) or TJR is a common procedure employed when conservative measures fail.
surgical creation of a functional synovial joint using implants.
most often to manage the pain of OA and improve mobility, RA, congenital anomalies, trauma, and osteonecrosis.
Contraindications for TJA:
active infection
advanced osteoporosis
progressive inflammation
diabetes/hypertension
preoperative TJA/TJR
patient education
multidisciplinary care
equipment
avoid procedure
lab testing
medication reconciliation
blood needs (cell saver, continuous reinfusion)
antiseptic bath
postoperative care: hip arthroplasty
prevention complications
hip dislocation-abduction devices with straps; be sure to loosen the straps every 2 hrs and check pt’s skin for irritation or breakdown
knee-positioning : operative leg needs to be fully supported with pillows to prevent slipping of the leg into adducted position which can lead to dislocation
VTE( DVT/PE)
older pt have increased risk for VTE because of age and decreased circulation before surgery. obese pt, pt who currently smokes, and those with history of VTE.
P (pharmacology), A (ambulation), C (compression)
anticoagulants: heparin, LMWH (enoxaparin, dalteparin), factor Xa inhibitors( SC fondaparinux, PO rivaroxaban)
infection (surgical incisions and vital signs-—increased redness, excessive/foul-smelling drainage, temp) s/s can be seen has early has 2 to 3 days after surgery
VS should be monitored every 4 hrs for first 24 hours and every 8-12 hrs
older pt may experience altered mental state, especially delirium
Anemia
dressings (excessive bleeding)
monitor labs (H&H)
neurovascular compromise
manage pain
short-term PCA or IV push morphine or hydromorphone, oral oxycodone
NSAIDS (ketorolac, celecoxib)
NMDA receptor antagonists( ketamine)
Gabapentinoids (gabapentin, pregabalin)
continuous peripheral nerve block (bupivacaine, ropivacaine)
Improving mobility
pt with OA often requires interprofessional efforts
physical therapy to teach weigh-bearing restrictions
occupational therapy
ambulatory aids may be needed for assistance with ADLs (walkers, crutches, cane)
care coordination and transition management
home care management: arranging to live on one floor with accessibility to all rooms is often the best solution. lowering counters, getting rid of throw rugs etc
self-management education: learning how to protect joints most importantly, weight reduction, eating well-balanced meals.
home care resources: providing patient safety information to prevent falls
Evaluation: reflecting
evaluate the care of the pt with OA on the basis of the identified priority problems. the expected outcome is that he or she
achieves pain control to a pain intensity level of 2 to 3 on a scale of 0 to 10 or at a level that is acceptable to the patient
does not experience complications associated with total joint arthroplasty ( if performed)
moves and functions in his or her own environment independently with or without assistive devices
Rheumatoid Arthritis (RA)
chronic, progressive, systemic inflammatory autoimmune disease process that affects primarily the synovial joints.
systemic means this disease can affect any or all parts of the body while affecting many joints
transformed autoantibodies (rheumatoid factors) that attack healthy tissues, especially synovium, are formed, causing inflammation
synovium thickens and becomes hyperemic, fluid accumulates in the joint space and the pannus forms
pannus is a vascular granulation tissue composed of inflammatory cells; it erodes articular cartilage and eventually destroys bone.
RA is characterized by natural remission and exacerbations (flare-ups)
inflammation outside the joints( blood vessels vasculitis)
etiology and genetic risks
combination of environmental and genetic factors
females reproductive hormones influence the development of RA (young to middle-age women)
infectious organism like Epstein-Barr virus
physical and emotional stress linked to exacerbations
interprofessional collaborative care: RA
assessment
acute/severe
slow/progressive onset
more common in winter months
Physical assessment/clinical manifestation
early-joint inflammation, swelling, pain, fatigue, generalized weakness
anorexia (weight loss about 2-3 lbs or 1 kg)
persistent low-grade fever
paresthesias
joints may be slightly reddened, warm, stiff, swollen, and tender or painful, particularly on palpation (caused by synovitis)
bilateral and symmetric (both wrists)
Late
joints become progressively inflamed and quite painful
frequent morning stiffness
palpation the joints feel soft and look puffy because of synovitis and effusions
muscle atrophy
decreased ROM
RA late systemic complications
weight loss, fever, and extreme fatigue which are common in late disease exacerbations
exacerbations
subcutaneous nodules: soft, round, moveable, ulnar surface of the arm.
respiratory (pleurisy, pneumonitis, diffuse interstitial fibrosis, and pulmonary hypertension), cardiac complications (myocarditis, pericarditis)
vasculitis is inflammation of the blood vessels
periungual lesions- ischemic skin lesions that appear in groups as small, brownish spots (nail beds)
paresthesias caused by peripheral neuropathy usually in older ppl
associated syndromes
sjogren’s syndrome most common
dry eyes (keratoconjunctivitis sicca (KCS)
dry mouth (xerostomia)
dry vagina (in some cases)
felty’s syndrome
caplan’s syndrome
psychosocial assessment
body image changes
Laboratory
RF is not diagnostic for RA but there they will have a positive titer (greater than 1:80)
ESR greater than 20mm/hr
serum complement (C3 and C4)
serum immunoglobulins
HsCRP
Diagnostic
x-ray to visualize the joint changes and deformities
CT scans help determine the presence and degree of cervical spine involvement
arthrocentesis is an invasive diagnostic procedure used for pt with joint swelling caused by excess synovial fluid (effusion)
bone scan
planning and implementation: responding
managing chronic inflammation and pain
drug therapy
DMARD: given to slow the progression of the disease, for best results they should be started early in the disease process
hydroxychloroquine antimalarial drug. ( watch for retinal damage)
NSAIDs given with H2 blocker (famotidine) if no change in 6-8 wks it may be discontinued
BRMs: help reduce signals for the immune system to cause inflammation. prevents further deterioration
recommended every 3 to 6 months
pt is at high risk for developing impaired immunity and subsequent infection
don’t give to pt with a serious infection, TB, or MS because it can exacerbate their health problems
don’t take any live vaccines and avoid large crowds and ppl with respiratory infections
other
glucocorticoids: prednisone
given for fast-acting anti-inflammatory and immunosuppressive effects
chronic use can cause: DM, impaired or decreased immunity, fluid and electrolyte imbalances, hypertension, osteoporosis, glaucoma
Methotrexate (MTX): immunosuppressive agent, low once-in-a-week dose, slow-acting drug taking 4 to 6 wks to begin control joint inflammation
adverse effects: decreasing WBCs and platelets ( bone marrow suppression), elevation in liver enzyme or serum creatinine
risk for infection (avoid crowds and ill ppl)
folic acid and B vitamins is recommended
NO PREGNANCY, strict birth control
nonpharmacologic interventions
Adequate rest
proper positioning
ice and heat application
plasmapheresis (not common)
complementary and integrative therapies
Promotion of self-management
Management of fatigue
enhance body image
promoting mobility
promote independence
adaptive devices
combat fatigue
physical therapy
energy conservation, taking 1 to 2 naps each day.
pacing activities
allowing rest periods
setting priorities’
obtaining assistance when needed
enhancing self-esteem
communicating acceptance of the pt
promote coping strategies
avoid using the term “arthritis personality” because it represents a negative label
Management
home care management
doors must be wide enough to accommodate a wheelchair or walker
ramps needed, ADL on one floor, Handrails available in bathroom and halls
elevated chairs, toilet seats ( needed for older ppl with arthritis.
self-management education
assess coping strategies
discuss with HCP before getting any OTC home remedies
assess self-esteem
assess how RA affects their work life.