4. E. coli Shigella
Enterobacterales and Escherichia coli
Presenter: Tibor Pál
Affiliation: Department of Medical Microbiology and Immunology, University of Pécs, Hungary
Overview of Enterobacterales
Also known as "enteric bacteria"
Found in the gastrointestinal tract, plants, and environment
Comprises of over 60 genera
Medically significant for its roles in:
Urinary tract infections (UTIs): 50-80%
Hospital-acquired pneumoniae: 20-25%
Bloodstream infections: 15-30%
Notable genera:
Escherichia (includes Shigella), Salmonella, Yersinia, Proteus, Morganella, Klebsiella, Enterobacter, Citrobacter, Serratia
Characteristics of Enterobacterales
Primarily composed of Gram-negative rods
Possess LPS (lipopolysaccharides) - endotoxin that can cause shock
Multi-resistance tendencies
Structure includes:
Outer membrane
Cell membrane
Cytoplasm
Antigens:
O antigen (LPS) is always present (except Yersinia pestis)
K antigen (capsule) presence varies by species/strain
H antigen (flagellum) presence varies by species/strain
Escherichia coli (and Shigella)
E. coli is a standard member of the gut flora
Antigenic variations: O:(K+/-):(H+/-)
Pathogenic potential varies significantly across strains
Shigella is genetically close to E. coli - traditionally classified as a separate genus due to specific pathogenicity traits
Types of E. coli Infections
E. coli can be categorized as:
Apathogens: Normal intestinal flora
Extraintestinal Pathogenic (ExPEC) strains:
Involved in enteric infections, causing diarrhea and other systemic infections
Uropathogens (UPEC) responsible for UTIs
Neonatal Meningitis (NMEC)
Strains related to diarrhea-causing pathotypes include:
Enteropathogenic E. coli (EPEC)
Enterohaemorrhagic E. coli (EHEC)
Enterotoxigenic E. coli (ETEC)
Enteroaggregative E. coli (EAEC)
Enteroinvasive E. coli (EIEC)
ExPEC Virulence Factors
Include:
Siderophores: Iron-chelating compounds
Cytotoxins
Fimbriae: Various types (type 1, P, S, M) aiding adhesion
Surface proteins: Include ibeA, traT, afa/dra
Toxins: Include cytotoxic distending toxin (CDT) and cytotoxic necrotising factor (CNF)
Capsule (K1, K5, ...): Functions to evade phagocytosis
Pathogenesis of Urinary Tract Infections (UTIs)
Community-acquired UTIs: 70-80% caused by E. coli
Predominantly affects women due to anatomical proximity and shorter urethra
Infections usually ascend from the colon to the urethra and bladder
Cystitis Mechanism:
Type 1 fimbriae adhere to uroplakin (mannose) leading to cell invasion,
Inflammation causes cell detachment which can be detectable in urine
Pyelonephritis involves specific adhesins (e.g., P fimbriae) and toxins leading to kidney infections
Asymptomatic Bacteriuria
Strains may lose virulence factors but can form biofilms, prompting treatment when seen in children and pregnancy
Bloodstream Infections
Derived from pyelonephritis or gut bacteria through mucosal barrier injuries
Increased risk in neonates and patients with intestinal perforations or other infections
Neonatal Meningitis (NMEC)
Major cause of neonatal meningitis associated with K1 capsular antigen
Mechanism includes blood-brain barrier transcytosis, leading to infection and inflammation in the brain
Diarrhea-causing Pathotypes of E. coli
Types include:
EPEC: Causes watery diarrhea; adhesion disrupts absorption
EHEC: Shiga toxin-producing, associated with severe complications like HUS
ETEC: Common in developing countries; causes watery diarrhea through enterotoxins
EAEC: Chronic diarrhea in children; linked to malabsorption
EIEC/Shigella: Causes dysentery, severe inflammation, and ulceration
Summary of Pathogenic Characteristics and Treatment Approaches
EPEC: Affects primarily children under 3; management involves rehydration and sometimes antibiotics.
EHEC: Low infectious dose, foodborne transmission; management focuses on supportive care.
ETEC: Human reservoirs, often food-related; supportive measures are key, with limited antibiotic use.
EAEC: Not well understood epidemiology; treatments may include rehydration.
EIEC/Shigella: More severe; treatment may include antibiotics and electrolytes.
Further Readings and Contact Information
Reference: Murray, P., Rosenthal, K., Pfaller, M. (eds.): Medical Microbiology, 8th ed. 2015 Elsevier
Contact: pal.tibor2@pte.hu