RBC Blood Transfusions
RBC REACTIONS - Dr. Francina Towne
READING ASSIGNMENT
Textbook: Basic Immunology: Functions & Disorders of the Immune System, Abbas & Lichtman
Pages to Read: 215-216
Figures to Preview: Figure 10-15 and the accompanying PowerPoint presentation.
LEARNING OBJECTIVES
Define and Discuss Agglutination: Understand the significance and mechanism behind agglutination.
Describe the ABO Blood Groups: Understand the characteristics of ABO blood groups and the Bombay blood type.
Compare and Contrast Histocompatibility: Assess differences between the ABO and Rh blood groups.
Blood Typing Results: Learn how to interpret and predict blood typing results.
Cross-Match Test Importance: Explain the process and significance of cross-matching blood before transfusions.
Understanding Recipients Serum and PRBCs: Define recipient's serum and packed red blood cells (PRBCs). Identify plasma contents.
Matching Donor Plasma: Discuss procedures for matching donor plasma to the recipient.
Transfusion Reactions: Compare different types of transfusion reactions that may occur.
Hemolytic Anemia Mechanism: Understand the mechanisms involved in hemolytic anemia.
RhoGAM Shot: Describe the RhoGAM shot and its applications in clinical practice.
AGGLUTINATION
Definition: Agglutination refers to the process where clumps (agglutinates) form when particulate antigens (e.g., whole cells) combine with corresponding antibodies.
Importance: It serves as evidence of an antigen-antibody reaction, resulting in a positive diagnostic test.
Hemagglutination: A specific type of agglutination test where the clumping of red blood cells (RBCs) indicates a positive result. It is primarily used for detecting A, B, and Rhesus (Rh) antigens.
BLOOD TRANSFUSION
Overview: Blood transfusion is a common clinical transplantation procedure.
Statistics: Approximately 25% of the population will require a blood transfusion at some stage in their life.
Importance of Matching Blood Types: Correct blood type matching is crucial to prevent adverse transfusion reactions.
STRUCTURES OF THE ABO BLOOD GROUP
Antigens Present on RBCs:
O Antigen
A Antigen (GalNAc)
B Antigen (Gal)
Structures include Fucose and Galactose residues.
ABO GENES
Genetic Traits:
A and B alleles are dominant over the O allele.
A and B alleles are codominant.
O allele is recessive.
Genotype Notation: Black text denotes genotype, and red denotes blood type.
ANTIGENS AND ANTIBODIES IN BLOOD GROUPS
Track presence of antigens vs antibodies in serum:
Difference between Plasma and Serum: Plasma includes clotting factors, while serum does not.
BLOOD DONORS AND RECIPIENTS
ABO Blood Group Matching:
Recipients with type A blood have anti-B antibodies and so forth.
Compatibility is necessary to prevent immune reactions.
ANTI-A AND ANTI-B ANTIBODIES IN TYPE O BLOOD
Individuals with type O blood develop anti-A and anti-B antibodies through environmental exposure and not inheritance.
RH (RHESUS) BLOOD GROUP SYSTEM
Rh+ Definition: Presence of the “D” antigen indicates Rh positive status.
Rh- Population: 15% may carry a deletion or alteration in the RhD allele, leading to Rh negative status.
Inheritance Patterns: Rh+ is dominant and Rh- recessive, following Mendelian inheritance principles.
BLOOD TYPING SUMMARY
Purpose: To determine which antigens are on the surface of the sample's cells.
Procedure & Example Card:
Utilize synthetic antibodies and toothpicks to identify antigens, NOT focusing on antibodies present in the serum sample.
INTERPRETING BLOOD TYPES
Agglutination Reactions:
Antiserum
Anti-A
Anti-B
Anti-Rh
Interpretation
Yes
A+
Yes
A antigen present; B antigen absent; Rh antigen present
Yes
A-
No
A antigen present; B antigen absent; Rh antigen absent
No
B+
Yes
B antigen present; A antigen absent; Rh antigen present
No
O-
No
A and B antigens absent; Rh antigen absent
EXCEPTIONS IN BLOOD TYPING
Non-identical Twins: Chimeric blood may occur when siblings share blood circulation.
Multiparous Women: May have retained cells from previous pregnancies (micro-chimerism).
Bombay Blood Group: Rare variants of blood types necessitate diligent cross-matching.
BOMBAY BLOOD GROUP
Characteristics:
Occurs in 4 people per million overall and in 1 per 10,000 in Mumbai.
Lacks fucose residue, appears as type O but has antibodies against A, B, and O.
Must receive blood only from Bombay blood group individuals.
CROSS-MATCH TEST
Purpose: To eliminate risk of unexpected transfusion reactions.
Procedure: Tests recipient's serum against donor RBCs, NOT against donor serum to avoid false matches.
TYPICAL BLOOD TRANSFUSIONS
Components Used:
Packed Red Blood Cells (PRBCs); a combination of whole blood and anticoagulant.
Whole blood is rarely used unless there’s massive loss.
Infection Screening: All blood products are screened for various infectious diseases (e.g., Hepatitis B & C, HIV, etc.).
TYPES OF BLOOD TRANSFUSION REACTIONS
Hemolytic Transfusion Reactions:
Type II hypersensitivity response.
Acute reactions may lead to severe symptoms (fever, chills, shock).
Treatment Protocols: Stop transfusion, maintain blood flow, treat shock symptoms.
Allergic Reactions:
Type I hypersensitivity response often related to plasma proteins.
Symptoms include wheezing and urticaria.
Treatment: Antihistamines.
Anaphylactic Reactions:
Severe allergic response; can be life-threatening.
Examples: Patients deficient in IgA must avoid IgA-containing products.
Treatment Procedures: Stop transfusion, administer rescue medications like adrenaline.
Febrile Non-Hemolytic Transfusion Reaction:
Most common type II hypersensitivity reaction.
Symptoms include fever and chills without hemolysis.
General Care: Treat with fever reducers, monitor closely.
UNIVERSAL RBCs AND STEM CELL RESEARCH
Research into producing universal red blood cells is ongoing due to declining blood donations.
Challenges: Though methods using stem cells show promise, mass production remains complicated.
NEW ROLES OF RBCs IN IMMUNE RESPONSE
Recent studies suggest RBCs play active roles in innate immune activation, contradicting old beliefs of them being immunologically inert.
Findings: RBCs express TLR9 and can bind DNA from pathogens, leading to inflammation and anemia.
HEMOLYTIC DISEASE OF THE NEWBORN (HDNB)
Mechanism in Rh- Mother and Rh+ Child: Antibodies formed during the first pregnancy attack fetal RBCs in subsequent pregnancies if the mother is Rh-.
RhoGAM Usage: Administered during pregnancy (28 weeks) and post-birth to prevent sensitization and subsequent fetal anemia.
Effects of RhoGAM: Prevents antibody response by binding to any Rh+ fetal cells that may enter the maternal circulation.