chapter 66 burns quiz 7
This chapter reviews types of burns, their physiologic consequences, essential assessments when caring for clients with burns, and the principles and techniques of burn management.
Gerontologic Considerations
Increased Risk for Older Adults:
Aging or disease may reduce sensory perceptions of touch, vision, smell, or hearing, increase risk for peripheral neuropathy, reduce mobility, or cause cognitive changes.
Assessment must evaluate:
Functional ability levels
Home maintenance and safety.
Risk-Reduction Teaching:
Water temperature settings should be no higher than 110°F.
Working smoke detectors with scheduled battery changes every 6 months (may sync with changing clocks for daylight savings time).
Accessible working fire extinguishers in the home.
Older adults may face serious complications due to diminished renal, cardiac, and respiratory functions.
Client teaching should focus on reducing the risk of flame and scald burns in home settings.
Burn Statistics
Approximately 1 million people in the United States seek treatment for burn injuries, most of which are minor (Rice & Orgill, 2021).
The incidence of burns is decreasing, but around 30,000 individuals with major burns require hospitalization (American Burn Association, 2024).
The mortality rate from burn injuries is increased by concurrent medical problems and the client's age.
The American Burn Association (2024) estimates that 3,800 people die annually from burns.
The risk of acquiring a burn injury peaks among children and adults over 60 years of age.
Common causes of thermal burns at home include:
Flames and scalding from steam or hot liquids, often secondary to smoking, alcohol ingestion, or flammable substances. (Schaefer & Tannan, 2020).
Burn Injuries
Definition:
A burn is a traumatic injury to the skin and underlying tissues caused by heat, chemicals, or electricity.
Electrical burns are often the most severe due to deep tissue damage caused by current flow through the body.
Pathophysiology and Etiology
Causes of Cell Damage:
The immediate cause is heat, with severity dependent on:
Temperature of the heat source
Duration of contact
Thickness of the tissue exposed
Location of the burn.
Burns in sensitive areas (e.g., perineal area) are at higher risk for infection, and facial, neck, or chest burns may compromise ventilation.
Burns affecting hands or joints can impact mobility and dexterity.
Effects of Burns:
Thermal injuries cause protein coagulation in cells.
Chemical burns result in liquefying tissue due to the reactions of strong acids, bases, or organic compounds with cells.
Electric burns create higher levels of heat at entry/exit points, complicating damage assessment.
Inflammatory Processes
Initial burn injuries extend further due to inflammatory responses affecting deeper tissue layers.
Protease enzymes and oxidants can cause additional injury, and neutrophils consume oxygen leading to tissue hypoxia.
Injured capillaries can thrombose, leading to ischemia and necrosis of tissues.
Neuroendocrine Changes
Serious burns induce neuroendocrine changes within the first 24 hours:
Hormonal Releases:
Adrenocorticotropic hormone and antidiuretic hormone are released in response to stress.
Glucocorticoids and aldosterone release lead to hyperglycemia and sodium retention, causing peripheral edema and oliguria.
Hypermetabolic State:
Increased oxygen and nutritional demands are crucial for recovery due to accelerated tissue catabolism.
Shifts in Fluids and Electrolytes
Fluid Dynamics:
Fluid shifts and electrolyte deficits occur post-burn, with fluid moving toward the burn area and causing edema.
Some fluids become trapped, leading to intravascular fluid deficits and decreased blood pressure.
If unmonitored, these changes can proceed to irreversible shock.
Cellular, Chemical, and Concurrent Injuries
Common Complications:
Anemia, hemoconcentration, and renal failure due to myoglobin and hemoglobin overload.
Increased gastric acidity and risks for gastric ulcers.
Inhalation injuries, concurrent medical problems, and advanced age can heighten mortality rates.
Cardiac dysrhythmias and CNS complications are frequent in electrical burn victims.
Burn Assessment
Assessment Components:
Determination of burn depth, zones of injury, and estimation of total body surface area (TBSA) burned.
Depth of Burn Injury
Classification of burn depth includes:
Superficial (First Degree):
Involves epidermis; red, painful, and heals < 5 days.
Superficial Partial Thickness (Second Degree):
Epidermis/demis, blistering, heals within 14 days.
Deep Partial Thickness (Second Degree):
Involves deeper dermis, takes > 3 weeks to heal, may require grafting.
Full Thickness (Third Degree):
Destroys all skin layers; may need skin graft.
Fourth Degree:
Extends to structures: fat, fascia, muscle, bone; painless and severely damaging.
Zones of Burn Injury
Zone of Coagulation:
Most severe injury, deepest area.
Zone of Stasis:
Intermediate burn area; potential for tissue survival.
Zone of Hyperemia:
Least injury, minimally affected epidermis and dermis.
Extent of Burn Injury
Rule of Nines:
Initial method for estimating TBSA involved with burns.
Lund and Browder Method:
More accurate than the rule of nines for TBSA calculations.
Medical Management
First Aid to Hospital Care:
The outcome relies heavily on the quality of initial first aid and later treatment at hospital or burn center.
Key complications: inhalation injury, hypovolemic shock, and infection.
Referral Criteria:
Partial/full thickness >10% TBSA
Burns involving face, hands, feet, genitalia, perineum, or major joints.
Chemical and electrical burns, as well as inhalation injuries and burns connected with other trauma.
Initial First Aid
Safety First:
Smother flames with a blanket if clothing is on fire and transport to a hospital.
Oxygen Administration and IV Fluids:
Administer oxygen and initiate IV therapy during transport.
Acute Care
Assessment upon Arrival:
Assess extent of injury and monitor for additional trauma (fractures, head injury, lacerations).
Ventilation Maintenance:
Initiate fluid resuscitation promptly.
Compromised ventilation can arise from various causes during first 12-24 hours post-burn (swelling, chemical inhalation, respiratory failure).
Administer humidified oxygen if airway edema or soot indicates potential injury.
Fluid Resuscitation
Goals:
Restore intravascular volume, prevent ischemia, and maintain organ function.
Calculate volume based on burn severity and initial interventions.
Wound Management
Examine Output and Infection:
Use personal protective equipment during wound care and monitor bacteria for infection.
**Cleansing and Antimicrobial Application: **
Use open vs. closed methods based on patient needs and severity of burns.
Antimicrobial Therapy
Common Antimicrobials and Their Uses:
Silver Sulfadiazine (Silvadene):
Broad-spectrum coverage for partial-thickness burns.
Mafenide Acetate (Sulfamylon):
Used for deep burns, penetrates eschar with potential metabolic side effects.
Skin Management:
Options include topical dressings, gauzes, and skin substitutes.
Surgical Management
Debridement Practices:
Remove necrotic tissue effectively while managing pain.
Skin Substitutes and Grafting
Techniques:
Autografts utilize the patient's skin; xenografts use animal skin while allografts use human donor sources.
Client Management and Education
Nutrition:
Clients may require increased calories (4,000-5,000/day) and protein support (2.0-2.5 g/kg).
Fluid Management:
Monitoring I&O is essential post-burn due to fluid loss.
Conclusion
Holistic Care Approach:
Incorporate physical, emotional, and nutritional support tailored to individual needs throughout recovery.