PA1-_Antihypertensive_therapy
Diuretics Overview
Definition: Diuretics are medications that increase renal sodium excretion, primarily used to manage hypertension through their natriuretic effect.
Key Classes of Diuretics:
Thiazides: First choice for hypertension.
Loop Diuretics: Less effective for hypertension and may increase the risk of hyponatremia.
Potassium-Sparing Diuretics: Used mainly in combination therapy to prevent hypokalemia.
General Advice on Diuretics
Use the lowest effective dosage to minimize adverse effects and protect renal function.
High salt intake can reduce the efficacy of diuretics.
Thiazide Diuretics
Indications:
Arterial hypertension (first-line therapy).
Sequential nephron blockage (when loop diuretics are ineffective).
Edema related to cardiac or renal issues.
Urolithiasis due to hypercalcuria.
Nephrogenic diabetes insipidus.
Examples:
Hydrochlorothiazide (thiazide).
Indapamide, Metolazone, Chlorthalidone, Xipamid (thiazide-like).
Mechanism of Action
Blocks Na+/Cl- symporter in the distal convoluted tubule.
Results in increased excretion of water, sodium, chloride, and potassium.
The effect stabilizes after about 3 weeks.
Effects of Thiazides
Reduces blood volume and lower blood pressure.
Long-term effects include reduction of sodium in vascular smooth muscle cells, leading to lower contractility and peripheral vascular resistance.
Serum changes: ↓Na, ↓K, ↑Ca. Higher doses beyond therapeutic range yield no additional effects.
Adverse Effects
Electrolyte Imbalances:
Hypokalemia, hyponatremia, and hypercalcemia.
Metabolic Effects:
Hyperglycemia (due to reduced insulin sensitivity).
Elevated lipid levels and hyperuricemia (risk of gout).
Interactions:
Can increase the effects of loop diuretics.
Efficacy is affected by GFR < 50 ml/min.
Loop Diuretics
Examples: Furosemide, Torasemide, Bumetanide, Piretanid.
Indications:
Requires intensive diuresis (e.g., peripheral and pulmonary edema).
Hypertension when thiazides are contraindicated.
Renal failure scenarios, volume overload, and forced diuresis for toxins.
Mechanism of Action
Inhibits Na+/K+/2Cl- cotransporter in the ascending limb of the loop of Henle.
Reduces osmotic pressure in the medulla, achieving diuresis even in poor renal function patients.
Effects of Loop Diuretics
Rapid and strong; the most potent diuretics available.
Serum changes: ↓Na+, ↓K+, ↓Ca+.
Adverse Effects
Electrolyte imbalances: hypokalemia, hyponatremia, hypermagnesemia.
Ototoxicity risk at high doses, especially with renal impairment.
Contraindications include electrolyte imbalances, hypovolemia, and sulfonamide allergy.
Potassium-Sparing Diuretics
Types:
Na+ Channel Blockers (e.g., Amiloride, Triamterene).
Aldosterone Receptor Antagonists (e.g., Spironolactone, Eplerenone).
Indications:
Treating hyperaldosteronism or hypokalemia caused by thiazides or loop diuretics.
Mechanism of Action
Both types prevent sodium reabsorption and potassium secretion in distal tubules and collecting ducts.
Adverse Effects
Hyperkalemia (rare, often with ACE inhibitors or renal dysfunction).
Specific side effects of Spironolactone: gynecomastia, impotence, amenorrhea.
Summary of Diuretic Use
Thiazides for arterial hypertension; cautious use below GFR <50 ml/min.
Loop diuretics for heart failure and situations of poor renal function; avoid evening doses to prevent nocturia.
Potassium-sparing diuretics added for patients developing hypokalemia under therapy.
Monitor renal function closely as all diuretics can cause electrolyte imbalances and potentially reduce renal function if not managed properly.