physio exam 3
3/17/25
-muscles and their movements
All animal movements depend on muscle contractions
Smooth muscles
Control the digestive system and other organs
Skeletal muscles/striated muscles
Control movement of the body in relation to the environment
Cardiac muscles
Heart muscles that have properties of skeletal and smooth muscles
-muscle fibers
Muscles composed of many individual fibers
Area where neurons communicate with muscle fiber?
Neuromuscular junction
Synapse
-neuromuscular junction
The neurotransmitter released in the junction is acetylcholine
Acetylcholine binds to ionotropic receptors
When acetylcholine binds to the ionotropic receptors, it causes ligand-gated sodium channels to open
-Antagonistic muscles
What message is acetylcholine sending to muscles?
Contract
Movement requires alternating contraction of opposing sets of muscles
Antagonistic muscles
Flexor muscle
Flexes or raises an appendage
Extensor muscle
Extends an appendage or straightens it
-fast and slow muscles
Skeletal muscle types range from:
Fast twitch
Fibers produce fast contractions
Rapid fatigue
Slow twitch
Less vigorous contraction
No fatigue
-fast and slow muscles
Slow twitch fibers
Aerobic
Require oxygen during movement
Can work without fatigue
Fast twitch fibers
Anaerobic
Breaks down glucose without use of oxygen
Overuse of anaerobic metabolism results in fatigue
-the cerebral cortex
The primary motor cortex
Precentral gyrus located in frontal lobe
Important for complex movements
Writing
Speaking
-muscle control from the primary motor cortex
Axons from primary motor cortex
Connect to brainstem and spinal cord
Generate impulses that control muscles
-initiating movement
Specific areas of primary motor cortex control specific areas of opposite side of body
Contralateral control
Primary motor cortex
Active during movement
Controls outcome
Rather than specific muscle
-posterior parietal cortex
Monitors body position in environment
Damage to this area
Difficulty coordinating visual stimuli with movement
Stimulation posterior parietal cortex
Report intention to move
Increased stim
Report movement despite none
-supplementary motor cortex
Organizes rapid sequence of movements in a specific order
Especially according to internal preprogrammed plan
Inhibitory if necessary
Active seconds before movement
-premotor cortex
Active when motor sequences activated by external events
Receives information about a target
The pen I want to pick up is in front of me on the desk
Integrates information about body position and posture
My hands are currently on keyboard
Organizes direction of movement in space
How will I get from the keyboard to the pen
-prefrontal cortex
Active seconds before movement
Important for considering probable outcomes of movement
Damage?
Disorganized movements
-mirror neurons
Active during both preparation of a movement and while watching someone else perform same/similar movement
First reported in premotor cortex, later in humans
May be involved in social behaviors
Empathy
Likely develop their properties through learning and are not likely the cause of imitation
Not definitive
-the cerebellum
Brain structure often associated with balance and coordination
More neurons in cerebellum than in all other brain areas combined
Damage to cerebellum causes trouble with rapid movements requiring aim/timing
-the cerebellum: more than just a subcortical structure
Somatosensory function
Active when using haptic perception
Feeling an object to determine identity
Active when object is moved across skin
Strong response to violations in touch expectation
Critical for aspects of attention
Ability to shift attention and attend to visual stimuli
3/19/25
-the basal ganglia
Group of large subcortical structures in forebrain
Important for initiating spontaneous movements
Compromises the following structures
Caudate nucleus
Putamen
Globus pallidus
Substania nigra
What are the four parts of the basal ganglia, substantia nigra sends dopamine signaling to caudate and putamen
Direct pathway =
Responsible for making a movement
Indirect pathway
Inhibits inappropriate or competing movements
-Parkinson’s disease
Movement disorder characterized by
Muscle tremors
Rigidity
Slow movements
Difficulty initiating physical and mental activity
Associated with an impairment in initiating spontaneous movement in absence of stimuli to guide action
Symptoms also include
Depression
Memory and reasoning deficits
Loss of olfaction
Other cognitive deficits
Studies show genetic link
Small factor
Environmental influences
Exposure to toxins
Traumatic head injury
-immediate cause of Parkinson’s
Death of neurons, especially in substantia nigra
Gradual and progressive
Substantia nigra usually sends dopamine-releasing axons to caudate nucleus and putamen
Loss of dopamine= less stimulation of motor cortex
Slower onset of movements
-treatment of Parkinson’s
L-dopa
Primary treatment for Parkinson’s
Precursors to dopamine
Easily crosses blood-brain barrier
Often ineffective and especially for those in late stages of disease
Does not prevent continued loss of neurons
Enters other brain cells, producing unpleasant side effects
-use of fetal tissue in research
Fetal tissue
Cells fertilized for at least 8 weeks
Before that, embryonic tissue
Tissue commonly derived from elective abortions
Embryonic tissues
Tissues commonly leftover from in vitro fertilization treatments
3/24/25
-Huntington’s disease
-rhythms of waking and sleeping
Early psychologists view of biological rhythms
Cycles of wakefulness and sleep dependent upon external stimuli
Curt Richter (1922)
Body generates cycles of activity and inactivity
-endogenous circadian rhythms
Circannual rhythms
All animals produce endogenous circadian rhythms
Internal mechanisms operate on approximately 24-hour cycle
Sleep cycle
Frequency of eating and drinking
Secretion of hormones
Urination
Sensitivity to drugs
Body temperature
-setting and resetting the biological clock
Why do we have a biological clock?
Keeps bodily processes in phase with outside world
Rhythm determined using both internal and external cues
Resetting our circadian rhythms is sometimes necessary
Zeitgeber
German meaning “time giver”
Stimulus that rests circadian rhythm
Most powerful zeitgeber
Sunlight
Time of meals
Exercise
Any kind of arousal
Using zeitgebers other than sunlight
Depression
Irritability
Impaired job performance
-jet lag
Disruption of circadian rhythm due to time zones
-morning and evening people
Cycles can differ between people
Lead to different patterns of wakefulness and alertness
Change as a function of age
Young children are morning people
Adolescents are often night people
As an adult
Partially depends on genetics
-mechanisms of the biological clock
Mechanisms of circadian rhythms
The suprachiasmatic nucleus (SCN)
Melatonin levels
-the suprachiasmatic nucleus
Main control center of circadian rhythms
Located above (supra) optic chiasm
Part of the hypothalamus
Damage to SCN
Erratic body rhythms
Not synchronized to environmental light/dark patterns
-the suprachiasmatic nucleus (SCN) and the circadian rhythm
Circadian rhythms genetically controlled
Unlearned
Single cell extracted from SCN and raised in tissue culture
Continues to produce action potential in a rhythmic pattern
SCN transfer experiments
-the suprachiasmatic nucleus (SCN) and the Retinohypothalamic Path
Light resets SCN via a small branch of optic nerve
Retinohypothalamic path
Travels directly from the retina to the SCN
Retinohypothalamic path
Special population of ganglion cells
Have their own photopigment called melanopsin
Cells respond directly to light
Do not require any input from rods or cones
-melatonin
SCN regulates waking and sleeping by controlling activity levels in other areas of the brain
Pineal gland
Endocrine gland located posterior (behind) to the thalamus
Melatonin
Hormone released by pineal
Increases sleepiness
Secretion begins 2-3 hours before bedtime
Feeds back to reset biological clock through its effects on receptors in SCN
Melatonin taken in afternoon
Phase-advance internal clock
Help you fall asleep earlier
-sleep is special
Coma
Low level of brain activity
Little to no response to external stimulus
Unresponsive wakefulness syndrome
Alternated between sleep and moderate arousal
Open eyes
Thrash
No awareness of surroundings or purposeful behavior
Minimally conscious state
Occasional brief purposeful actions
Respond to command
Track movement with eyes
-stages of sleep: EEG
Electroencephalogram (EEG)
Led to discovery of sleep stages
Polysomnography
Combination of EEG and eye-movement records
Stage 1 sleep
Beta waves
Present when one is awake and alert
Alpha waves
Present when one begins a state of relaxation
Stage 1 sleep
EEG waves short, choppy, rapid
Neurons out of phase with one another
Brain activity begins to decrease
Stage 2 sleep
Sleep spindles
12-14 Hz waves during a burst that lasts a half a second
K-complex
Sharp wave associated with temporary inhibition of neuronal firing
Shown to occur in response to:
Low level sounds in environment
Touches to skin
Slow wave sleep: stage 3 and 4
EEG recording of slow, large amplitude wave
Slowing of heart rate, breathing rate, and brain activity
Highly synchronized neuronal activity
-paradoxical or REM sleep
Rapid eye movement (REM) sleep
Paradoxical sleep
EEG waves irregular, low-voltage, fast
Postural muscles paralyzed
-brain mechanisms of wakefulness and arousal: reticular formation
Reticular formation
Extends from medulla to forebrain
Responsible for arousal
Damage to this area?
Prolonged bouts of sleep
-brain mechanisms of wakefulness and arousal: Pontomesencephalon
Pons and mesencephalon
Part of reticular formation
Axons extend to hypothalamus, thalamus, and basal forebrain
Release acetylcholine and glutamate
Produce excitatory effects to widespread areas of cortex
Stimulation of pontomesencephalon
Awakens sleeping individuals
Increases alertness in those already awake
-brain mechanisms of wakefulness and arousal: locus coeruleus
Small area within pons
Usually quiescent
Fires when exposed to emotionally relevant information
Releases norepinephrine throughout cortex
Wide ranging effect
Enhanced attention and memory for important info
-brain mechanisms of wakefulness and arousal: hypothalamus
Contains neurons that release histamine
Produce widespread excitatory effects
Fire consistently when alert
Fire at a lower rate when sleepy
Cease firing when in non-REM or REM sleep
Antihistamines produce sleepiness
Orexin/hypocretin
Released by neurons extending from hypothalamus to basal forebrain
Necessary for maintaining arousal
Orexin knock-out mice
Alternate between waking and sleeping, even during wheel running
-brain mechanisms of wakefulness and arousal: GABA and Acetylcholine
Basal forebrain
Involved both in sleep and wakefulness
Some axons release acetylcholine
Excitatory and increases arousal
Released during wakefulness and REM sleep
Release sharpens attention
Some axons release GABA
Inhibitory neurotransmitter
Essential for sleep
-sleep and the inhibition of brain activity
GABA important for
Decreasing temperature and metabolic rate
Decreasing stimulation of neurons
-sleep as a local phenomenon
Sleep can be localized within brain
Sleepwalkers
Awake in one part of brain and asleep in others
Most extreme version of sleep localization in brain
Dolphins
Lucid dreaming
Dreaming but aware of being asleep and dreaming
-brain functions in REM sleep
Results from rather invasive sleep studies
During REM sleep
Activity increases in pons and limbic system
Activity decreases in primary visual cortex, motor cortex, and dorsolateral prefrontal cortex
Pons send messages to spinal cord
Inhibits motor neurons that control large muscles
Prevents motor movement during REM sleep
REM regulated by serotonin and acetylcholine
Drugs that stimulate acetylcholine receptors quickly move people to REM
Serotonin interrupts REM
-narcolepsy
Sleep disorder characterized by frequent periods of sleepiness
Gradual/sudden attack of sleepiness
Occasional cataplexy
Muscle weakness triggered by strong emotions
Sleep paralysis
Hypnagogic hallucinations
Dreamlike experiences during sleep onset
Difficult to distinguish from reality
Runs in families
Primary treatment
Stimulant drugs
-sleepwalking
Runs in families
Mostly in stages 3-4 of sleep
Not dangerous to wake sleepwalker
-sleep and memory
Important role in enhancing learning memory
Performance on newly learned task better the next day if adequately rested
Patterns of activity hippocampus during learning were like those shown during sleep
Suggests brain replays daily experiences during sleep
-amounts of REM sleep
Humans spend one-third of their life asleep
About one-fifth is spent in REM
Species vary in amount of sleep time spent in REM
Most common in birds and mammals
Percentage of REM sleep is positively correlated with the total amount of sleep in most animals
Amond humans, those who get the most sleep have the highest percentage of REM
-functions of REM sleep
Brain may discard useless connections
learned motor skills may be consolidated
Disruption of sleep early in night
Verbal learning disruptions
Disruption of sleep later in night
Motor learning disruptions
3/31/25
-sex and hormones
Prenatal sexual differentiation begins with sex chromosomes
Female mammal
XX
Male mammal
XY
During early prenatal development
Mullerian ducts
Precursors to female’s oviducts, uterus, and upper vagina
Wolffian duct
Precursors to other male reproductive organs
Vans deferens and seminal vesicles
Undifferentiated gonads
-the SRY gene
Gene on male Y chromosome responsible for masculinization
Primitive gonads into testes
Sperm-producing organs
Developing testes produce androgens
Increase testes growth
Mullerian inhibiting hormone (MIH)
Females= SRY gene
Gonads develop into ovaries
Egg producing organs
-hormones in males and females
Androgens and estrogens
Classes of hormones
Not hormones themselves
Most famous androgen
Testosterone
Most famous estrogen
Estradiol
-steroid hormones
Exert effects in three ways
Binding to membrane receptors like neurotransmitters
Entering cells
Can pass through cell membrane
Activate proteins in cytoplasm
Bind to chromosomes in nucleus
Activate or inactivate certain genes
-different effects of sex hormones
Organizational effects
Occur at sensitive of periods
Before birth and at puberty in humans
Determine whether brain and body will develop male or female characteristics
Activating effects
Occur any time of life
Temporarily activate a particular
Pregnancy
Menstruation
Some cognitive effects
-sexual differentiation
Human sensitive period for genital formation
Occurs during first trimester of pregnancy
Depends mostly on level of testosterone
Conversion to DHT responsible for external genitalia formation
-organizing of sex hormones: estrogens
Estradiol and other estrogens
Not vital for early reproductive development
Surge of estradiol necessary for secondary sex characteristics at puberty
Absence of sex hormones
Leads to female-looking external genitalia
Independent of sex chromosomes
XX individuals is lacking estradiol during early life
Lower than average sexual interest in adulthood
-sex differences in childhood behavior
Childhood toy behavior
Boys
Cars, trains, balls and guns, and play roughly
Girls
Dolls and tea sets, and play cooperatively
Preferences tend to be consistent over time
Socialization or general preferences of each sex?
Baby monkeys show similar preferences
-sex differences in childhood behavior: prenatal hormones
Girls exposed to high testosterone in utero
Slightly elevated preference for typical boys’ toys
Boys born to women with high phthalate levels (inhibit T production)
Elevated interest in typical girls’ toys
-actinvational effects of hormones
The menstrual cycle
Periodic variation in hormones and fertility over approx. 28. Days
Hypothalamus and pituitary gland interact with ovaries to produce cycle
-Females: FSH
After end of a menstrual period
Anterior pituitary releases follicle-stimulating hormone (FSH)
Promotes growth of follicle in ovary
Follicle nurtures ovum and produces estradiol
Towards middle of cycle
Follicle builds up receptors to FSH
Follicle produces increasing amounts of estradiol
-Females: FSH and LH
Increased estradiol causes anterior pituitary to increase release of FSH and luteinizing hormone (LH)
FSH and LH cause follicle to release an ovum
Remnants of follicle release hormone progesterone
Prepares uterus for implantation of a fertilized ovum
Inhibits further release of LH
-Females: pregnancy
Estradiol and progesterone levels increase gradually throughout pregnancy
No pregnancy
Levels decline
Uterine lining is cast off (menstruation)
-Females: birth control pills
Interfere with usual feedback cycle between ovaries and pituitary gland
Combination-pill contains estrogen and progesterone
Prevents surge of FSH and LH that would release and ovum
Thickens mucus of cervix
More difficult for sperm to reach egg
Prevents egg from implanting in uterus
No female birth control that prevents STI
-gender identity and gender differentiated behaviors
Gender identity
How we identify and what we call ourselves
All nurture, rather than nature
Current advice
Biological factors, especially prenatal hormones, also play role
Sex differences
Biological differences
Gender differences
Differences that result from people’s thoughts about themselves as male or female
-individuals who are intersexed
Anatomies intermediate between phenotypically male or female
Causes
Woman released two ova, each fertilized by a different sperm
Ova united instead of becoming twins; rare
Difference in sex chromosomes; atypical hormone patterns before birth
-genital ambiguity
1 in about 5,000 has genital ambiguity to make sex assignment in US
-chromosomal differences
turner syndrome
Lack or damage of second X chromosome
Variety of medical problems
Low thyroid hormone, heart defects, diabetes
Will not menstruate or develop female secondary sex characteristics without hormone replacement therapy
Klinefelter Syndrome (XXY)
Results in
Sterility
Small testes
Greater than average height (for males)
Poor coordination
Weak muscle tone
“Supermale syndrome” (XYY)
Criminal gene?
Associated with
Increased height
Increased risk of learning problems
Average IQ
Acne
-androgen insensitivity syndrome
Androgen insensitivity or testicular feminization
Individuals with XY chromosome pattern
Production of androgens normal
Lack androgen receptor that enables activation of genes
Female phenotypic presentation
Smaller than average penis or genital appearance of female
Individuals tend to identify as women
-discrepancies of sexual appearance
5-alpha reductase syndromme
Genetic males can’t convert testosterone to dihydrotestoerone (DHT)
Feminized genitalia at birth
Penis develops during adolescence in puberty
Many endorse male gender identity and heterosexual attractions
-mental rotation
Robust sex difference (male advantage)
Test factors that exacerbate male advantage
Time vs untimed
2D vs 3D
Cultural factors
Socioeconomic status
Gender equity in participant country of origin
-navigational tasks
Magnitude of sex difference is smaller
Evidence from animal studies suggests that sex difference can be erased through training
-task of verbal memory
Verbal fluency
Producing words from a particular semantic category
Name all the types of flowers you can in 30 seconds
Verbal memory
Recalling words from previously presented lists
Recognizing words previously exposed to among a list of distractor words
-memory for object location
Female advantage
Identifying new objects in an array after delay
Identifying moved objects in an array after delay
Not all studies agree on female advantage on the tasks
Evidence that familiarity with objects is important
-the case for a hormonal influence
Evidence from CAH
Women with CAH have better spatial performance than women without CAH
Women with fraternal male twins
Better spatial performance than controls
Exposure to prenatal testosterone likely increases female spatial performance
-sexual orientation
Homosexual behavior occurs in many animal species
Most men discover sexual orientation early
Process slower for women
Feminine-type behaviors during childhood and adolescence correlate strongly with homosexuality in males
Masculine-type behavior in girls a poor predictor of sexual orientation
-genetics
One contributing factor in homosexuality
Research studies of twins
If one twin is homosexual, probability that other twin is as well is high (monozygotic twins)
No single gene has been identified
-prenatal influences
Sexual orientation not related to adult hormone levels
Homosexual men and women tend to have normal hormone levels for their biological sex
-prenatal stress
Mother's immune system may exert prenatal effects
Mother may react to protein in a son and alter subsequent sons’ development
Prenatal exposure to stress and alcohol may play a role
Stress releases endorphins
Antagonizes effects of testosterone on hypothalamus
-brain anatomy
Differences in brain anatomy between homosexuals and heterosexuals exist
On average, the homosexual brain is shifted towards the opposite sex in some ways
Several reported differences have no link to sexuality
May relate to behavior differences