knee
what three muscle groups demand increases with running speed
soleus
quadriceps
hip complex
what plane of movement does an ACL strain most often occur in
the sagittal plane
what is the main protective muscle group for the ACL
the hamstrings
patella instability diagnostic tests
ottawa knee rules
sweep test
apprehension test - gliding patella laterally
medial joint palpation (MPFL tenderness)
joint glides and compressions
what are the muscles that oppose ACL loading
hamstrings
soleus
gluteus Medius
what are the muscles likely to increase ACL load
quadriceps
gastrocnemius
are previous mild knee strains a risk factor for ACL rupture
in animal studies it found that there was a
decreased ACL strength and knee joint remodelling
altered movement control
what cartilage affects happen post ACL
cartilage homeostasis is affected
increase in non healthy cartilage growth
cartilage hypertrophy (swelling) = increased water content
cartilage doesn’t recover as well
neuro-biomechanical adaptations following ACL injury , ACLR
kinematics
increased knee extension and external tibial rotation
kinetics
decreased external knee flexion moments
increase and decreased knee adduction moments
neuromuscular
weakness
atrophy
decreased vasti activation
increased co-contraction
decreased quad force control
do ACLR patients underload TFJ
ACL reheb
Phase 1 - recovery from surgery
Goals to
PWB-FWB
Get rid of swelling
Get to 100 deg knee flexion
Get quad strength to 4+/5
5/5 hamstring strength
Phase 2 - strength and neuromuscular control - 2-12 weeks
Goals to
Have no swelling
Full knee hyperextension
Knee flexion 130 degrees
Full squat
Good balalnce and control
Unrestricted walking
Phase 3 - running, agility and landings
Goals to
Full ROM
Full strength
Jogging
Running
Agility
Sport specific skills
Phase 4 return to sport
what is patellofemoral pain
anterior or retropatellar pain in the absence of other pathology
List PFP typical symptoms
typically non-traumatic
diffuse ache in anterior knee
pain exacerbated by loading activities like squatting, stairs, run
ache with prolonged sitting
crepitus (popping, cracking)
giving way
swelling
locking
what is the structural contributors to the pain
it is unclear ?
what are the potential pathways that link to PFP
elevated PFJ stress
psychological factors
pain adaptations
what are the two main causes of elevated PFJ stress
decreased PFJ contact area
caused by patella malalignment / maltracking
increased PFJ reaction forces
caused by altered joint kinetics / kinematics / muscle tightness
what anatomical changes can lead to PFP
patella
unusual shapes more likely to develop PFP
trochlear
lateral wall deficient
trochlear shallow or areas of convexity
what direction of patellar translation is common amongst people with PFP
lateral
what are the passive soft tissue restraints to patellar tracking
lateral retinaculum - tightness
ITB - tightness
medial weakness
explain the quadricep muscle relationship to patella maltracking
the VL has a great CSA and larger velocity producing properties than the VMO and can often result in a greater pull on the patella laterally by overpowering the VMO. this can alter PF rotation and tilt which may alter the contact area of the patellar within the trochlear which can result in increasing PFJ stress
how does vast neuromotor control relate to PFP
those with PFP can have VMO delayed activation leading to increase PFJ stress
how does an increased hip internal rotation relate to PFP
leads to
increased tightness of ITB
increased lateral quadriceps moment
changed orientation of femoral trochlea
what can cause increased hip internal rotation
glute medius dysfunction
femoral anteversion
what is hamstrings and gastrocs relationship to PFP
decreased length of hamstrings and gastrocs increases the knee flexion moment and increases PFJ reaction force and PFP
what are the psychosocial features of PFP
anxiety, depression, catastrophising and fear of movement is increased in people with PFP
altered pain processing

management of PFP
education
passive interventions
active rehabilitation
psychosocial factors
what is important to educate with PFP
the contributing factors of PFP
the management options
the importance of engaging with rehab
to manage expectations
weight loss advice
advice for activity modifications
what is involved with active rehab for PFP
vasti retraining
quadriceps and gluteal strengthening
movement pattern re-training
what are specific exercises to do
VMO retraining
learn how to contract VMO
use palpation
attempt to contract the VMO before the rest of the quads
quarter squat
about 40 degrees knee flexion
focus on glutes
ensure correct alignment
step downs
emphasis control both VMO and proximal
may use a mirror
what passive rehab can be done
tailored patellar taping
foot orthoses
massage / dry needling of tight lateral structures
PFJ mobilisation of hypomobile joint
Skye is a fourteen-year-old who dislocated her right patella five days ago whilst playing softball. She has been partial weight-bearing with crutches since the injury.
5a) List some of the contributing factors, and describe how these factors may contribute to, patello-femoral joint dysfunction. You may use a flow chart to illustrate your answer.
- Tight lateral structures pulling on the patella such as ITB, lateral retinaculum
- Abnormal patella positioning such as baja or alta meaning it does not sit in the trochlea optimally
- Increased Q angle / valgus alignment leading to more lateral patella tracking
- Trochlear dysplasia / shallow groove leading to reduced bony stability
- Weak VMO leading to poor medial stabilization
- Ligament laxity and hypermobility
Structural factors (↑Q-angle, trochlear dysplasia, patella alta)
↓
Reduced bony stability
↓
Patella prone to lateral displacement
↓
Muscle imbalance (weak VMO, tight lateral structures)
↓
Dynamic maltracking
↓
Pain, swelling, quadriceps inhibition
↓
Patellofemoral joint dysfunction
6 marks
5b) Describe two (2) exercises that may be appropriate to address her problems
i) at 5 days post-injury
isometric quad strengthening
straight leg raises
sidelying hip abduction
isometric hamstring stretches
ankle pumps
2. Mr C is day 1 post right (R) ACL reconstruction (gracilis / semitendinosis). He is
resting in bed with a Zimmer splint on his right knee and has had his drain tube
removed. The surgeon's protocol indicates that he may full weight bear, without the
splint (if he has good quads control) and to discharge him as soon as possible.
a) Outline your objective assessment and expected findings for this patient.
- Moderate pain and swelling
• Limited knee flexion, near full passive extension achievable
• Reduced quadriceps activation (difficulty with SLR)
• Hamstring donor site soreness
• Mobilising with crutches, WBAT if quads control present
(9 marks)
b) List your criteria for discharge?
- Safe independent mobility with crutches and weight bearing as tolerates
- To have good quad control so be able to perform a straight leg raise without any quad lag
- Patient education on home exercise program, precautions, wound care
- Pain and swelling controlled
(4 marks)
c) Outline 6 potential rehabilitation issues/complications following discharge,
and discuss your principles of management for two (2) of these.
- Reduced knee ROM
o Knee ROM exercises: such as
§ Passive knee extension and flexion
§ Heal slides
§ Patellar mobilizations
- Reduced quad strength
o Early isometric quad activation
o Straight leg raises
- Hamstring weakness from the donor site
- Swelling
- Pain
- Impaired mobility