enzymes 1
Enzymology
Professor: Prof. Dr. Esam Abd El-Mohsen Afifi
Enzymes are crucial biological catalysts, referred to as the "tools of life."
Enzyme Nomenclature
A. Recommended Names
Enzymes generally have names ending in "-ase" derived from substrates (e.g., glucosidase, urease) or actions performed (e.g., lactate dehydrogenase).
Some enzymes retain trivial names (e.g., trypsin, pepsin) without relation to substrate or action.
B. Systematic Names
Developed by the International Union of Biochemistry and Molecular Biology (IUBMB), enzymes are classified into six major classes.
Example: Hexokinase is systematically named ATP: D-hexose 6-phosphotransferase (E.C. 2.7.1.1).
Major Classes of Enzymes
Oxidoreductases
Involve oxidation-reduction reactions.
Example: Lactate dehydrogenase facilitating the conversion of lactate to pyruvate.
Transferases
Transfer functional groups (C-, N-, P-containing).
Example: Serine hydroxy methyl transferase.
Hydrolases
Cleave bonds with water addition.
Example: Urease catalyzes urea to ammonia and carbon dioxide.
Lyases
Catalyze bond cleavage without hydrolysis or oxidation.
Example: Pyruvate decarboxylase.
Isomerases
Catalyze isomerization (geometric and optical).
Example: Methylmalonyl CoA isomerization.
Ligases
Form bonds between C and O, S, or N atoms with ATP hydrolysis.
Example: Pyruvate carboxylase.
Properties of Enzymes
Enzymes are:
Protein catalysts, except for ribozymes (catalytic RNA).
Not consumed or permanently changed by the reaction.
Highly efficient (up to 10^8 times faster than non-catalyzed reactions).
Specific for substrates and reactions, contributing to selective catalysis.
Active Sites
The active site is a 3D region made up of amino acid side chains that provides specificity for substrate binding.
Notable models:
Lock and Key Model
Induced Fit Model
Catalytic Efficiency
Enzymes can convert 100-1,000 substrate molecules to product per second, defined as the turnover number (kcat).
Cofactors
Cofactors are non-protein molecules aiding enzyme reactions.
Organic Cofactors: Non-metal (e.g., coenzymes like NAD+).
Inorganic Cofactors: Metal ions (e.g., Zn2+, Fe2+).
Holoenzymes
Holoenzymes consist of an apoenzyme and its associated cofactor (inorganic or organic).
Coenzyme: Transiently associated organic molecules.
Prosthetic Group: Permanently bound organic molecules.
Cell Locations
Enzymes may be compartmentalized to optimize conditions for their respective reactions while isolating substrates/products from competing reactions.
Mechanism of Action
1. Thermodynamic Changes
Enzymes lower the activation energy barrier, enhancing the reaction velocity.
2. Active Site Processes
The active site stabilizes the transition state and provides catalytic groups, facilitating substrate conversion.
Factors Affecting Enzyme Activity
Temperature
Reaction velocity peaks at optimal temperature (35-40°C for human enzymes).
pH
Optimal pH varies by enzyme (e.g., pepsin at pH 2).
Substrate Concentration
Reaction velocity increases until saturation (Vmax).
Kinetics can be first-order or zero-order based on substrate concentration relative to Km.
Michaelis-Menten Kinetics
Describes the reaction rate based on substrate concentration.
Michaelis-Menten Equation:
Vo = (Vmax[S]) / (Km + [S]).
Km reflects the enzyme's affinity towards its substrate alluding to reaction rates across three substrate concentration conditions.
Enzyme Inhibition
Competitive Inhibition
Inhibitor resembles substrate; affects Km but not Vmax.
Noncompetitive Inhibition
Inhibitor binds separately; affects Vmax, not Km.
Uncompetitive Inhibition
Inhibitor binds to ES complex; lowers both Km and Vmax.
Clinical Application of Enzymes
1. Diagnosis via Plasma Enzyme Activity
Functional enzymes: Secreted by living cells (e.g., liver).
Non-functional enzymes: Released during cell turnover; increased levels indicate tissue damage (e.g., ALT in liver damage).
2. Isoenzymes
Different physical properties due to genetic variance; help localize tissue damage.
Key Enzymes in Myocardial Infarction
Troponin T/I and CK-MB markers critical for diagnosing myocardial infarction (MI).
Timing of elevation after MI: Troponin T/I appears after 4-6 hours; CK-MB within 4-8 hours.
High-Sensitivity Cardiac Troponin Test
Detects low troponin levels to expedite heart attack diagnosis and rule out coronary artery disease.
Q&A on Enzymology
Discusses various concepts including enzyme inhibitors, effects of pH and substrate concentration, and importance of structural properties in enzyme functionality.