Study Notes on Rheumatology
Rheumatology Study Notes
Definition
Rheumatism or rheumatic disorders are conditions causing chronic, often intermittent pain affecting the joints or connective tissue.
Rheumatism does not specify any specific disorder but encompasses at least 200 different conditions, including arthritis.
Rheumatological Diseases
Raynaud Syndrome
Rheumatoid Arthritis
Systemic Lupus Erythematosus
Scleroderma / Systemic Sclerosis
Dermatomyositis / Polymyositis
Sjögren Syndrome
Wegener Granulomatosis
Giant Cell Arteritis
Raynaud's Phenomenon
Definition: Raynaud's phenomenon can be defined as a health condition in which the blood vessels in an individual's body narrow in response to cold and stress, leading to transient malperfusion of fingers, toes, nose, and ears.
Symptoms: Affected areas turn pale and numb when exposed to cold.
Causes: Primarily caused by cold temperatures.
Sequence of Color Change:
Initially, the skin becomes white due to a lack of blood supply.
The blue color appears due to the absence of oxygen.
Once the blood supply returns to normal, the skin then gets a red color.
Rheumatoid Arthritis (RA)
Definition: Rheumatoid arthritis (RA) is an autoimmune disease that causes chronic inflammation of the joints.
RA can also cause inflammation around the joints, as well as in other organs, meaning it is referred to as a systemic illness and is sometimes called rheumatoid disease.
It is characterized as a chronic illness, lasting for years, with potential for long periods without symptoms.
Progression: RA is typically progressive, with the potential to cause joint destruction and functional disability.
Joint Structure in RA
Healthy Joint Components:
Muscle
Bursa
Bone
Synovial membrane
Synovial fluid
Joint capsule
RA Characteristics:
Bone erosion
Swollen inflamed synovial membrane
Thinned cartilage leading to bone ends rubbing together.
Symptoms of RA
Symptoms fluctuate depending on tissue inflammation levels:
Active disease period: fatigue, loss of energy, appetite loss, low-grade fever, muscle and joint aches, and stiffness that is symmetrical.
Stiffness is most notable in the mornings and after inactivity.
During disease flares, joints often become red, swollen, painful, tender due to synovium inflammation, leading to excessive synovial fluid production (synovitis).
Deformities in RA
Radial Deviation of the hand at the wrist.
Ulnar Deviation of fingers at MCP joints.
Dorsal Subluxation of the ulnar head caused by interruption of the radioulnar ligament.
Late Stage: Includes “Boutonniere” deformity of the thumb, “Swan-neck” deformity of fingers.
Diagnosis of RA
Diagnostic Tools:
X-Ray
MRI
ESR (Erythrocyte Sedimentation Rate)
C-reactive protein
Total blood count
Rheumatoid factor (IgM autoantibodies against IgG): positive in 70% of RA patients but also in 5% of healthy individuals, hence used for prognostic rather than diagnostic purposes.
Antinuclear antibody testing.
Criteria for Diagnosis of RA
American College Of Rheumatology Revised Criteria: Four of the following criteria must be present for at least six weeks:
Morning stiffness
Arthritis of three or more joint areas
Arthritis of hand joints
Symmetric arthritis
Rheumatoid nodules
Serum rheumatoid factor
Radiographic changes
Treatment of RA
First-line drugs: aspirin and corticosteroids to reduce pain and inflammation.
Second-line drugs (DMARDs): gold, methotrexate, hydroxychloroquine promote disease remission and prevent joint destruction, though they are not anti-inflammatory.
If therapy fails, biologics like etanercept or infliximab may be considered as they exhibit specific inflammatory mediation.
Physiotherapy for RA
Regular physiotherapy is mandated for RA patients aiming to quickly restore mobility to affected joints and prevent complications:
Active and passive movement therapy
Cold application during acute episodes; heat application otherwise
Massage
Balneotherapy (use of thermal spas)
Systemic Lupus Erythematosus (SLE)
Definition: SLE is an autoimmune disease characterized by periods of flares and remission.
Symptoms of SLE
Symptoms vary greatly among individuals:
Skin ulcers
Arthritis, including joint pain, stiffness, and swelling
Inflammation of the heart and lungs (serositis)
Kidney issues, which can present with swelling or high blood pressure.
Neurological issues like headaches or seizures
Characteristic butterfly-shaped (malar) rash across the nose and cheeks, or a scaly, disk-shaped rash.
Risk Factors for SLE
Influences include:
Sunlight
Vitamin D deficiency
Smoking
Certain infections
The precise cause remains unclear.
SLICC Classification Criteria for SLE
Require at least 4 criteria (1 clinical, 1 laboratory) OR biopsy-proven lupus nephritis with positive ANA or Anti-DNA:
Clinical Criteria: include cutaneous lupus, oral/nasal ulcers, arthritis, serositis, renal, neurologic, and hematologic conditions.
Immunologic Criteria: positivity for ANA, Anti-DNA, Anti-Sm, antiphospholipid antibodies, and others.
Treatment of SLE
Involves preventing flares and minimizing severity:
Corticosteroids
Immunosuppressive agents.
Prognosis for SLE
Considered incurable but highly treatable; many individuals can expect a normal lifespan.
Scleroderma and Systemic Sclerosis
Progresses from localized scleroderma (skin only) to systemic sclerosis affecting skin and internal organs
Characterized by collagen accumulation causing organ infarctions.
Symptoms of Scleroderma
Reduced joint mobility with contractures
Distally tapering fingers: “Madonna fingers”
Rigidity of facial muscles (reduced facial mobility)
Small mouth opening with radial creases
CREST Syndrome
Limited scleroderma symptoms referred to as CREST:
Calci-Des deposits in skin
Raynaud’s phenomenon
Esophageal dysfunction
Sclerodactyly
Telangiectasias
Diagnosis of Scleroderma
Identified through presence of:
Anti-centromere antibodies (70% with limited cutaneous scleroderma)
Anti-Scl antibodies (40% with diffuse cutaneous scleroderma).
Treatment of Systemic Sclerosis
Treatment remains challenging: specific organ-focused therapies including but not limited to:
Renal crisis management with ACE inhibitors
Oesophageal involvement with PPIs
Pulmonary hypertension treated with calcium channel blockers or prostacyclins and possibly lung transplant in severe cases.
Dermatomyositis and Polymyositis
Conditions affecting proximal muscles, characterized by:
Muscle weakness, particularly during movements like standing or arm lifting.
Common symptoms also include muscular pain and fever.
Approximately 10% occur in association with malignancies (paraneoplasia).
Sjögren’s Syndrome
A chronic disorder leading to insufficient moisture production in certain glands.
Symptoms of Sjögren’s Syndrome
Keratoconjunctivitis sicca (dry eyes)
Symptoms include: gritty sensation, burning, redness.
Xerostomia (dry mouth)
Symptoms include: chewing and swallowing difficulties, decreased taste, speaking difficulties.
Enlarged parotid glands, excessive fatigue, muscle and joint aches.
Diagnosis of Sjögren’s Syndrome
Diagnosis relies on:
Dry eyes and mouth assessment (e.g., Schirmer test).
Laboratory tests identifying autoantibodies in blood (anti-SSA or anti-SSB).
Biopsy of the inner lip to identify salivary gland inflammation.
Treatment of Sjögren’s Syndrome
No cure, but treatment aims to reduce discomfort and harmful effects of dryness; tailored to patient symptoms.
Vasculitis
An inflammatory vascular disease instigated by immunological reactions, impacting various organs. Diagnosed based on clinical, pathological, anatomical, and immunological findings.
Wegener Granulomatosis
A specific type of vasculitis characterized by granulomas, often starting in the upper respiratory tract before affecting systemic areas (pulmorenal syndrome).
Symptoms may include chronic sinusitis, oral/nasal ulcers, hemoptysis, arthralgia, and signs of renal failure.
Granulomas
Defined as a structure formed during inflammation, a collection of immune cells (macrophages), formed by the immune system to wall off substances perceived as foreign.
Giant Cell Arteritis
Common vasculitis in older women, presenting mainly as temporal arteritis.
Symptoms include:
Throbbing headaches in temples
Eye pain with a risk of blindness from central artery involvement
Palpable painful hardened temporal artery.
The cause remains unknown, treatment primarily involves glucocorticoids.