Mucosal LO3
Commensal Bacteria and Microbiota
Definition of Commensal Microbes / Microbiota:
Microbiota refers to the microbes living in and on our bodies.
Interdependence of intestinal microbiota and the host is highly significant and manifests in various ways.
Effect of Microbiota on Health:
Influences nutrient uptake and drug metabolism.
Alters risk of cancer and impacts cognitive function.
Studies show microbiota transplant from healthy to unhealthy individuals can treat diseases such as Crohn's disease.
Highlights correlation between microbiota and various body systems.
Bodily Systems and Commensal Bacteria
Importance of Gut Microbes:
Essential for vitamin synthesis and dietary fiber digestion.
Maintains gut epithelial integrity and stimulates immune responses that produce mucus and tight junctions.
Regulation of inflammatory responses.
Variety of Microbes:
A diverse range of microbes exist in and on our bodies, varying by anatomical site (e.g., skin vs. gut).
Predominantly bacterial species, but also includes other types of microbes.
Predominant Gut Bacterial Species:
Vermiculitis and Bacteroidetes are identified as the most predominant in the gut.
Factors Affecting Microbiota Composition
Birth Method:
Vaginal delivery vs. cesarean section affects microbiota composition.
Studies indicate inoculation of cesarean-delivered babies with vaginal secretions to favorably change microbiota.
Age and Microbiota Diversity:
Aging influences types and diversity of gut microbes.
Genetics:
Genetic predisposition can affect microbiota composition.
Geography and Environment:
Living conditions (rural vs. urban) and environmental exposure influence microbiota.
Antibiotic Use:
Antibiotics target not just pathogenic bacteria but also beneficial commensals, causing an imbalance.
Diet:
Diet plays a crucial role in shaping gut microbiota through the nutrients available to microbes.
Interrelation between age, diet, and microbiota diversity.
High fiber diet supports beneficial microbes; low fiber diet may promote inflammatory microbiota.
Immune Modulation by Commensal Microbes
Stimulation of Immune Responses:
Commensal microbes stimulate Paneth cells to secrete antimicrobial substances.
Goblet cells produce mucin glycoproteins, contributing to mucus formation, which creates an anti-inflammatory environment.
Cellular Mechanism:
Macrophages and dendritic cells sample commensal antigens, presenting them to lymphocytes in mesenteric lymph nodes.
T Cell Differentiation:
Dendritic cells equipped with TGF-beta and retinoic acid promote differentiation into T regulatory cells (Tregs) and Th17 cells.
Dendritic cells influence T cell homing back to the gut.
Short Chain Fatty Acids (SCFAs):
Produced by gut bacteria from dietary fiber and serve as energy sources for colonic epithelium.
SCFAs promote Treg cell proliferation and enhance immune responses against pathogens.
SCFAs also function as ligands for GPR43, influencing further immune modulation.
NF-kappa B Pathway Regulation
Significance of NF-kappa B:
NF-kappa B is a crucial transcription factor within immune responses.
Regulates genes associated with both innate and adaptive immunity.
Pathway Details:
Mechanism: When pathogens bind to Toll-like receptors, they induce the production of IRAK1, which degrades I kappa B, releasing NF-kappa B to enter the nucleus and promote inflammatory cytokine transcription.
Commensals can modulate this pathway in various ways to prevent excessive inflammation.
Dietary Impact on Microbiota and Immune System
Good vs. Bad Diets:
High-fiber, healthy diets foster anti-inflammatory microbiota.
Western diets (high fat, low fiber) promote inflammation and dysbiosis, resulting in negative health outcomes.
Concluding Concepts:
The interaction between commensal bacteria and the immune system is significant for the body’s health and disease prevention.
Maintaining a diverse and well-functioning microbiota is crucial.
Mucosal immunity is characterized by a tolerance to non-harmful antigens while responding appropriately to pathogens.
Protective epithelial barriers play a vital role in establishing an anti-inflammatory environment.
T cell and B cell responses are adapted to promote tolerance and neutralization of antigens, respectively.
Understanding these relationships helps elucidate the role of commensal microbes in health and disease management.