The Development of T Lymphocytes
7-1: The Development of T Cells in the Thymus
Thymus as a Primary Lymphoid Organ:
The thymus is involved in T cell development but not in direct immune response to infection.
Composed mainly of thymocytes (immature T cells), macrophages, dendritic cells, and thymic stroma (epithelial cells).
7-2: Thymocyte Lineage Commitment
Cell Migration:
Progenitor stem cells enter the thymus before rearranging T-cell receptor (TCR) genes.
Stem cells begin acquiring receptors, adhesion molecules, and signaling molecules like IL-7.
Thymocyte Characteristics:
Double-negative (DN) thymocytes lack markers CD4, CD8, CD3, and TCR but have T cell characteristics.
7-3: Lineage Differentiation of T Cells
Thymocyte Progenitor Cell:
Double Negative thymocytes initiate TCR gene rearrangement, entering a "race" to become αβ or γδ T cells.
If the γ or δ genes rearrange first, a γδ T cell forms; if the β gene rearranges first, it may form an αβ T cell.
Double Positive (DP) Thymocytes:
Express both CD4 and CD8 after successful β chain rearrangement, forming a pre-TCR that anchors the β chain.
Apoptosis of Non-Functional TCR:
Cells with non-functional TCRs undergo apoptosis, and macrophages clear dead cells.
7-4: Gene Rearrangement in DN Thymocytes
Rearrangement Process:
Genes for β, γ, and δ chains undergo rearrangement in DN thymocytes.
Pre-T Cell Receptor (pre-TCR):
Successful β-chain rearrangement leads to pre-TCR formation, which signals the cell to proliferate and mature.
7-5: α-Chain Gene Rearrangement in Pre-T Cells
Rearrangement Characteristics:
The α-chain gene rearrangement occurs only in pre-T cells and lacks a D segment, favoring successful adjustments.
When the α-chain is rearranged, the δ locus is excised, preventing the development of γδ T cells.
7-6: Developmental Stages of T Cells Marked by Gene Expression Changes
Early Development:
T cell development is marked by checkpoints such as pre-TCR formation and α-chain gene rearrangement.
7-7: Positive Selection of T Cells in the Thymus
Positive Selection:
Thymocytes recognizing self-MHC undergo positive selection influenced by thymic epithelial cells presenting self-peptide-MHC complexes.
Selection Measurement:
Effective binding leads thymocytes to survive; weak or absent binding results in apoptosis.
7-8: Impact of Thymic Specific Proteasome on Positive Selection
Peptides Importance:
Thymic specific proteasome generates peptides that influence positive selection, ensuring T cells that moderate self-reactivity survive.
7-9: Continuing α-Chain Rearrangement
Receptor Editing:
Defines the adaptation process by which T cells alter their α-chains to enhance binding to self-MHC, occurring during positive selection.
7-10: CD4 vs CD8 T Cell Precursor Decision
Selection Mechanism:
Interaction of TCR with peptide-MHC determines the lineage commitment to either CD4 or CD8 T cell.
DP thymocytes lose expression of one co-receptor (CD4 or CD8), becoming single-positive T cells.
7-11: Negative Selection of Self-Reactive T Cells
Neglect in Thymus:
Thymic dendritic cells and macrophages present self-antigens.
Strongly binding TCRs signal apoptotic pathways to eliminate potentially autoreactive T cells.
7-12: Role of AIRE in Negative Selection
Expression of Tissue-Specific Proteins:
The autoimmune regulator (AIRE) drives expression of non-thymic proteins, ensuring elimination of self-reactive T cells.
Pathology Involvement:
AIRE deficiency leads to autoimmune diseases such as autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy (APECED), reflecting increased self-reactive T cell survival.
7-13: Regulatory CD4 T Cells and Lineage
Regulatory Function:
Regulatory T cells (Tregs) maintain tolerance against self-antigens, providing additional control beyond negative selection.
Mechanism of selection for Tregs remains unclear.
7-14: Post-Thymic T Cell Differentiation in Secondary Lymphoid Tissue
After the Thymus:
T cells circulate between blood, lymph, and secondary lymphoid organs, remaining long-lived.
Upon antigen recognition, T cells differentiate into various effector subtypes (e.g., TH1 and TH2).
Chapter Overview and Therapy Implications
Therapeutic Relevance:
Bone marrow transplants require HLA compatibility to avoid graft versus host disease (GVHD).
Organ rejection occurs when the recipient immune system targets transplanted tissue due to HLA mismatch.
Immunotherapy seeks to utilize T cell capabilities for tumor eradication.